SUBJECTS
|
BROWSE
|
CAREER CENTER
|
POPULAR
|
JOIN
|
LOGIN
Business Skills
|
Soft Skills
|
Basic Literacy
|
Certifications
About
|
Help
|
Privacy
|
Terms
|
Email
Search
Test your basic knowledge |
Cardiac
Start Test
Study First
Subject
:
health-sciences
Instructions:
Answer 50 questions in 15 minutes.
If you are not ready to take this test, you can
study here
.
Match each statement with the correct term.
Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.
This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. What valves are most commonly involved in chronic rheumatic heart disease?
Mitral mitral+aortic
Myocarditis
Kawasaki disease
Decreased forward perfusion pulmonary congestion
2. What coronary arterysupplies the lateral wall of the LV?
Heart transplant
3-8 wks
Circumflex
Endomyocardial fibrosis w/eosinophilic infiltrate and eosinophilia
3. Drug that vasodilates both arteries and veins but mostly veins. Used to decrease preload to heart.
Nitroglycerin
Wear and tear
Congested central veins
Concentric hypertrophy - can't oxygenate full wall - ischemic damage
4. What is the tx for VSD?
Large vegetations of S aureus
Myxoid degeneration
LA
Surgical closure small defects may close spontaneously
5. Which angina(s) show ST elevation on EKG? ST depression?
PGE
Prinzmetal stable and unstable
LA dilation
PDA
6. What cardiac disease is associated with tuberous sclerosis?
Inability to maintain systemic pressure w/lack of O2 to vital organs
Pancarditis
Rhabdomyoma
PDA
7. L- to - R shunt switching to R- to - L shunt.
Acute inflammation
Foci of chronic inflammation - reactive histiocytes with slender - wavy nuclei - giant cells - and fibrinoid material
Eisenmenger syndrome
IV drug users
8. In what pt population does S aureus commonly cause valvular disease?
Tetralogy of fallot
Early - blowing diastolic murmur bounding pulse - pulsating nail bed - and head bobbing
Sudden cardiac death
IV drug users
9. Pericarditis 6-8 wks post MI.
Dressler syndrome
Opening snap followed by diastolic rumble
Mitral mitral+aortic
Contraction band necrosis
10. In transposition of the great vessels - What is required for survival? How is this achieved?
Ischemia - htn - dilated cardiomyopathy - MI - restrictive cardiomyopathy
Shunt - PGE to maintain PDA until surgical repair can be performed
Coxsackie A or B
CHF
11. Which congenital heart defect is associated with maternal diabetes?
Red border granulation tissue
>70%
Transposition of the great vessels
Aortic stenosis
12. When is a post - MI pt at highest risk for an aneurysm? With what microscopic change is this complication associated?
Months out fibrosis
Janeway lesions
Aspirin and/or heparin - supplemental O2 - nitrates - beta blocker - ACE inhibitor - fibrinolysis or angioplasty
Bounding pulse
13. What are the clinical features of RHF?
Jugular venous distension - painful hepatosplenomegaly w/nutmeg liver - cardica cirrhosis - dependent pitting edema
Valve replacement
VSD
Right to left
14. How does restrictive cardiomyopathy present?
ST- segment depression
Pancarditis
Libman - Sacks endocarditis
Congestive heart failure
15. What does rupture of the IV septum cause?
Aspirin and/or heparin - supplemental O2 - nitrates - beta blocker - ACE inhibitor - fibrinolysis or angioplasty
Split S2 on auscultation
Shunt
Contraction band necrosis
16. What is the characteristic murmurr of mitral stenosis?
Opening snap followed by diastolic rumble
Day 1-7
Nonbacterial thrombotic endocarditis (marantic endocarditis)
4-6 hours - 24 hours - 72 hours
17. Dense layer of elastic and fibrotic tissue in the endocardium.
45%
Bicuspid aortic valve
Reperfusion injury
Endocardial fibroelastosis
18. When do neutrophils infiltrate the myocardium post MI?
Reperfusion injury
1-3 days
Large vegetations of S aureus
Chest pain <20 min brought on by exertion or emotional stress
19. How do ACE inhibitors tx MI?
Valve replacement AFTER the onset of complications
Paradoxical emboli
Decreases LV dilation by decreasing volume
Shunt
20. What is the most comon cause of aortic regurg? What are the other causes?
Isolated root dilation - valve damage (infective endocarditis) - aortic root dilation (syphilitic aneurysm or aortic dissection)
Wear and tear
2-3 weeks
Widens it diastolic pressure decreases due to regurgitation while systolic pressure increases due to increased stroke volume
21. Holosystolic blowing murmur that increases w/expiration?
Small vegetations along the line of closure
Mitral regurg
Circumflex
Blood vessels coming in from normal tissue
22. Is scar tissue or myocardium stronger?
Myocardium
Holosystolic blowing murmur
LAD
Concentric LV hypertophy
23. What type of shunt dose PDA cause?
Left -->right
Increased arterial resistence decreases shunting - allowing more blood to reach lungs
Coexisting mitral stenosis and fusion of commisures exist
Infantile coarctation of the aorta PDA
24. What shunt does tetralogy of fallot produce?
2-4 hours - 24 hours - 7-10 days
Right -->left
Cardiac tamponade
Loss of LV fx
25. What effect does transposition of the great vessels have on the ventricles?
Hypertophy of RV atrophy of LV
Gelatinous - abundant ground substance
Infectious endocarditis
Rupture of plaque with w/thrombus and incomplete occlusion of coronary artery
26. Endomyocardial fibrosis w/eosinophilic infiltrate and eosinophilia.
Spontaneous
Large vegetations of S aureus
Large - destructive vegetations
Loeffler syndrome
27. What complications occur within 4 hrs post MI?
Cardiogenic shock - CHF - arrhythmia
Widens it diastolic pressure decreases due to regurgitation while systolic pressure increases due to increased stroke volume
Wear and tear
1 day: coag necr 1 wk: inflammation (neutrophils and macrophages) 1 mo: scar
28. How do you tx prinzmetal angina?
IV drug users
NG or Ca channel blocker
Months out fibrosis
Endomyocardial fibrosis w/eosinophilic infiltrate and eosinophilia
29. What is the characteristic finding on CXR in tetralogy of fallot?
1) damaged endocardial surface develops thrombotic vegetations 2) transient bacteremia leads to trapping of bacteria in the vegetations
Loss of fx
Bicuspid aortic valve
Boot shaped heart
30. How does squating decrease hypoxemia in tetralogy of fallot?
Eisenmenger syndrome
Aspirin and/or heparin - supplemental O2 - nitrates - beta blocker - ACE inhibitor - fibrinolysis or angioplasty
Hemosiderin laden macrophages
Increased arterial resistence decreases shunting - allowing more blood to reach lungs
31. What causes heart failure cells?
Restrictive cardiomyopathy
Cyanosis - RV hypertrophy - polycythemia - clubbing
Positive blood cultures anemia of chronic disease
Rupture of capillaries leading to intraalveolar hemorrhage - sx of LHF
32. What type of tumor is a rhabdomyoma?
Harmartoma
Aspirin and/or heparin - supplemental O2 - nitrates - beta blocker - ACE inhibitor - fibrinolysis or angioplasty
Small - sterile fibrin deposits randomly arranged on closure of valve leaflets
4-7 days macrophage infiltration
33. What are the clinical features of endocarditis? What causes each feature?
Fever: due to bacteremia murmur: due to vegetations janeway lesions - osler nodes - and splinter hemorrhages: due to embolization of septic vegetations anemia of chronic disease: due to chronic inflammation
Circumflex
ACE inhibitor
Tuberous sclerosis
34. What is the rate of congenital heart defects?
1%
Chronic rheumatic heart disease
Fibrosis and dystrophic calcification
Reversible
35. Hypertension in upper extremities - hypotension in lower extremities - notching of ribs on CXR.
Adult coarctation of the aorta
Mitral regurg
Transesophageal echo
Fever: due to bacteremia murmur: due to vegetations janeway lesions - osler nodes - and splinter hemorrhages: due to embolization of septic vegetations anemia of chronic disease: due to chronic inflammation
36. What does nonbacterial thrombotic endocarditis cause?
Mitral regurg
Regurg vs stenosis
SLE
Friction rub and chest pain
37. What effect does squatting have on the murmur of mitral valve prolapse? Why?
Opening snap followed by diastolic rumble
Louder - increased systemic resistence decreases LV emptying
Split S2 on auscultation
Turner syndrome
38. How does stable angina present?
Shunt
Chest pain <20 min brought on by exertion or emotional stress
Pericardial effusion due to pericardial involvement
Valve replacement
39. What is the effect of acute vs chronic rheumatic disease off the mitral valve?
Fetal alcohol syndrome
Regurg vs stenosis
S aureus
Red border granulation tissue
40. Opening snap followed by diastolic rumble.
VSD
Amyloidosis - sarcoidosis - hemochromatosis - and Loeffler syndrome
Intercostal arteries enlarged due to collateral circulation
Mitral stenosis
41. What heart sound manifest with an ASD?
Split S2 on auscultation
VSD
Erythematous nontender lesions on palms and soles.
Sudden cardiac death
42. What is the most common cause of sudden cardiac death? What are less common causes of sudden cardiac death?
Left -->right
Acute ischemia - mitral valve prolapse - cardiomyopathy - cocaine abuse
CK- MB
Transesophageal echo
43. What type of shunt does transposition of the great vessels cause?
Thickening of chrodae tendinae and cusps - mitral stenosis
Nonbacterial thrombotic endocarditis (marantic endocarditis)
LHF - left - to - right shunt - chronic lung disease (cor pulmonale)
R-->L
44. What is the only Jones criteria that doesn't resolve with time?
PGE
PDA
Evidence of prior group A beta - hemolytic strep plus major and minor criteria
Pancarditis
45. What is typically the mechanims of sudden cardiac death?
Chronic ischemic heart disease
Ventricular arrhythmia
Amyloidosis - sarcoidosis - hemochromatosis - and Loeffler syndrome
VSD
46. When do CK- MB levels rise - peak - and return to normal?
Concentric LV hypertophy
Fibrinous pericarditis
4-6 hours - 24 hours - 72 hours
Concentric hypertrophy - can't oxygenate full wall - ischemic damage
47. What is the most common primary cardiac tumor in adults? Is it malignant or benign?
4-7 days
Myxoma - benign
Contraction band necrosis - reperfusion injury
Maternal diabetes
48. Which coronary artery supplies the anterior wall and anterior septum?
Opening snap followed by diastolic rumble
LAD
Low voltage EKG w/diminished QRS amplitude
Shunt
49. What two things cause coronary artery vasospasm?
Nonbacterial thrombotic endocarditis (marantic endocarditis)
Heart transplant
Osler nodes (ouch - ouch Osler)
Prinzmetal angina - cocaine
50. What causes prinzmetal angina?
SLE
RHF
Shunt - PGE to maintain PDA until surgical repair can be performed
Coronary artery vasospasm