Test your basic knowledge |

Cardiology

Subject : health-sciences
Instructions:
  • Answer 50 questions in 15 minutes.
  • If you are not ready to take this test, you can study here.
  • Match each statement with the correct term.
  • Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.

This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. Which class of drugs decreases afterload?






2. Does eccentric hypertrophy or concentric hypertrophy cause systolic disfunction






3. How does a patient with Tet of fallot learn to improve symptoms?






4. The cause of dyspnea on exertion?






5. which medications are used to maintain patency or close the ductus arteriosus?






6. Chronic mitral stenosis can lead to what changes in size of the LA






7. Mitral stenosis is most often secondary to which condition?






8. What does mitral prolapse predeispose to?






9. What causes the cushing reflex and why






10. tearing chest pain radiation to the back - associated with marfan






11. Which murmur do you hear in mitral stenosis?






12. What does prolonged QT predispose to?






13. Which class of drugs decrease preload






14. What causes the early cyanosis in Tet of Fallot?






15. What causes ankle - sacral edema - jugular venous distention






16. smaller vegetations - congenitally abnormal or diseased valves - sequela of dental procedures. Insidious onset






17. Which artery supplies the SA and AV nodes?






18. What is the machine like murmur? What is the heart pathology and the predisposing causes






19. Which kind of infarct show ST elevation - and/or pathologic Q waves






20. MAP is also known as






21. What happens with a decrease of extracellular Na






22. port wine stains on face - intracerebral AVM - siezures - early onset glaucoma - congenital






23. What is the S2 sound?






24. Where are pacemaker cells?






25. What is the difference between adult and infantile type aortic coarctation?






26. What is the time frame for arrhythmia risk in the evolution of MI






27. diaphoresis - N/V - severe retrosternal pain - pain in left arm/jaw - SOB - fatigue - adrenergic symptoms






28. What is the gold standard for dx of MI in the first 6 hours






29. In an anteroseptal infarct - which artery is effected - and which leads show Q waves?






30. In an anterior wall infarct - which artery is effected and which leads show Q waves






31. Wegener's presentation






32. What other sign is often present with congenital long QT syndrome - why?






33. Left to right shunts are more common in babies or kids?






34. EDV is also known as






35. The 7 complications of MI

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36. What masks atrial repolarization?






37. What is the effect on the slope of phase 4 in pacemaker cells by catecholamines and






38. Which murmur is heard with VSD?






39. coronary artery spasm - ST elevation






40. What are the diastolic heart sounds?






41. What causes hepatomegaly?






42. benign cap hemangioma of infancy - spont regresses






43. What is the result of not have fast sodium channels in pacemaker cells?






44. What is the characteristic pulse in aortic stenosis?






45. What are aschoff bodies






46. Which organ gets the largest share of systemic cardiac output






47. Rank the following by speed of conduction - av node - atria - purkinjee - ventricles






48. In terms of starling forces - why does heart failure cause edema?






49. In an anterolateral infarct - which artery is effected and which leads show Q waves






50. In an acute MI - are there any visible changes via LM in the first 2-4 hours