Test your basic knowledge |

Cardiology

Subject : health-sciences
Instructions:
  • Answer 50 questions in 15 minutes.
  • If you are not ready to take this test, you can study here.
  • Match each statement with the correct term.
  • Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.

This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. pulmonary veins drain into right heart circulation (SVC - coronary sinus)






2. Left to right shunts are more common in babies or kids?






3. How do catecholamines increase contractility?






4. What is the cushing triad?






5. What is the result of not have fast sodium channels in pacemaker cells?






6. in the JVP - What is the c wave?






7. immune mediated transmural vasculitis with fibrinoid necrosis - small and medium vessels - renal and viscera - not pulm arteries - hep B seropos in 30% of pts






8. port wine stains on face - intracerebral AVM - siezures - early onset glaucoma - congenital






9. Churg Strauss - presentation and test






10. Why is contractility decreased in heart failure?






11. What murmur is heard with aortic regurg?






12. What do the carotid and aortic bodies respond to?






13. How does a patient with Tet of fallot learn to improve symptoms?






14. In terms of starling forces - why does heart failure cause edema?






15. Which bacteria can cause endocarditis from prosthetic valves?






16. What are the complications from bacterial endocarditis?






17. skin rash on buttocks and legs - arthralgia - intestinal hemorrhage - abdominal pain - melena. Follows URI - IgA immune complex - most common childhood systemic vasculitis






18. Right to left shunts are more common in babies or kids?






19. congenital heart defect in an infant with a diabetic mother?






20. no relation between p waves and QRS intervals - treatment and predisposing factor






21. Which valve is most commonly involved in bacterial endocarditis?






22. What kind of infarct show ST depression






23. dyspnea - fatigue - edema and rales - multiple causes






24. What causes the early cyanosis in Tet of Fallot?






25. Which valve is commonly involved in bacterial endocarditis from IV drug use and Which bacteria are most common?

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26. What causes tet of fallot?






27. In what disease states is blood viscosity increased?






28. absecnce of tricuspid valve - hypoplastic RV






29. What is the formula for EF?






30. What is the gold standard for dx of MI in the first 6 hours






31. lymphatic malignancy associated with persistant lymphadema - post radical mastectomy






32. disruption of the vasa vasorum of aorta - dilation of aorta and valve ring - tree bark appearance (calcifications on aortic root)






33. Given P = QR - what factors influence resistance?






34. In an EKG - What is the QT interval?






35. What is the early and late lesion in rheumatic heart disease






36. What does TAPVR stand for






37. Endothelial malignancy of the skin assocated with HHV-8 and HIV

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38. What causes aortic stenosis






39. highly lethal malignancy of the liver - associated with vinyl chloride - arsenic - and thorosrast exposure






40. What is the effect on the slope of phase 4 in pacemaker cells by catecholamines and






41. Fatal arrhythmia






42. wartiike - sterile vegetations occur on both sides of the valve - commonly causes mitral regurg. SLE causes it






43. What other syndrom is associated with infantile aortic coarctation






44. Which lab value indicates blood viscosity?






45. What is the progression of atherosclerosis?






46. In a lateral wall infarct - which artery is effected - and which leads show Q waves?






47. Irregularly irregular ECG - no p waves: dx and treatment






48. What do patients die early from in rheumatic heart disease?






49. When do you see extensive coagulative necrosis in an MI






50. dilated tortous veins due to chronically inc venous pressure - poor wound healing - varicose ulcers