Test your basic knowledge |

Cardiology

Subject : health-sciences
Instructions:
  • Answer 50 questions in 15 minutes.
  • If you are not ready to take this test, you can study here.
  • Match each statement with the correct term.
  • Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.

This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. with what heart sounds do ASD usually present?






2. moncekberg






3. necrotizing granulomas in lung and upper airways - nectrotizing GN - small vessel vasculitis

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4. tearing chest pain radiation to the back - associated with marfan






5. decrease stretch in baroreceptors leads to what response?






6. EDV - ESV






7. Do dihydropyridine or non - dihyrdropyridine Ca channel blockers decrease contractility






8. What are the complications of atherosclerosis?






9. Which murmur is heard with mitral prolapse?






10. What does an isoelectric ST segment indicate?






11. PCWP > LV diastolic pressure






12. What are common causes of mitral regurg?






13. Equilibration of diastolic pressures in all 4 chambers - decreased CO from compression of heart by fluid in pericardium






14. Central chemoreceptors do not respond directly to which parameter?






15. What are tendinous xanthoma - atheromas - and corneal arcus signs of?






16. When do you find hemosiderin laden macrophages in the lungs?






17. absecnce of tricuspid valve - hypoplastic RV






18. What are aschoff bodies






19. When do you see extensive coagulative necrosis in an MI






20. What murmur is heard with aortic regurg?






21. In an EKG - What is the T wave?






22. Which sympathetic receptors raise MAP






23. In the evolution of an MI - when the risk for free wall rupture - tamponade - papillary muscle rupture - or interventricular septal rupture the hightest? Why?






24. On the cardiac cycle graph - on which corners do the opening and closing of the aortic and mitral valves occur?






25. Mitral stenosis is most often secondary to which condition?






26. What channels do the the pacemaker cells lack?






27. What is the danger of torsades to pointes?






28. dyspnea - fatigue - edema and rales - multiple causes






29. Why is there edema after burns or during infection






30. What does mitral prolapse predeispose to?






31. Fatal arrhythmia






32. Expiration causes an increase in which sided heart sounds






33. What are the complications from bacterial endocarditis?






34. What other sign is often present with congenital long QT syndrome - why?






35. In an acute MI - are there any visible changes via LM in the first 2-4 hours






36. What causes ankle - sacral edema - jugular venous distention






37. When does extracellular calcium enter the cardiac muscle cells during contraction?






38. What is association with fixed S2 splitting - does not increase with inspiration






39. Which area of the endocardium is especially vulnerable to infarction? Why?






40. When is the scar completely formed in an MI?






41. Which channel accounts for automaticity of the SA and AV nodes?






42. What happens in phase 2 of the cardiac ventricular action potential?






43. In the cardiac and vascular function curves - In what instance is the vascular curve shifted to the right?






44. How does a patient with Tet of fallot learn to improve symptoms?






45. How does digitatlis increase contractility?






46. serum marker for wegener's






47. retrosternal chest main with exertion - ST depression on ECG - likely due atherosclerosis






48. Why is contractility decreased in heart failure?






49. pulseless disease - granulomatous thickening of the aortic arch and/or proximal great vessels - elev ESR - asian females > 40

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50. Where are pacemaker cells?