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First Aid: Reproductive

Instructions:
  • Answer 50 questions in 15 minutes.
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  • Match each statement with the correct term.
  • Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.

This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. What are the four functions of estrogen






2. testicular atrophy - eunochoid body shape - tall - long extremities - gynecomastia - inactivated X chromosome - dysgenesis of seminiferous tubules - decreased inhibin - abnormal leydig cell function

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3. What does hCG do in the first trimester to maintain the corpus luteum






4. pain with or without bleeding - increased in hCG - sudden lower abdominal pain - mistaken for appendicitis






5. dx with decreased testosterone - increased LH






6. in males - are mature teratomas malignant? What is the case for females






7. Where does FSH work - what enzyme works there - what substrate comes in and what leaves






8. Wher does dysplasia and carcinoma in situ of the cervix usually begin






9. large - hyperchromatic syncytiotrophoblasts cells - increased freq theca leutin cysts - develops during pregnancy in mom or baby - marked by hCG






10. dx with increased testosterone and increased LH






11. attachment of palceta to lower uterine segment that may occlude internal os - painless bleeding in any trimester






12. common cause of recurrent miscarriage in 2nd trimester






13. What is the common presentation of metastasis in prostate cancer






14. What are the pathologic features of leiosarcoma






15. What is the lymphatic drainage the ovaries/testis






16. cystic swelling of chorionic villi and proliferation of chorionic epithelium that presents with abnormal vaginal bleeding






17. What are risk factors for abruptio placentae?






18. gray - soliarty - crusty plaque - usually on the shaft of the penis or on the scrotum - peak incidence in 5th decade of life - can progress to invasive SCC in < 10% of cases

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19. Which cells secrete beta hCG






20. Which system and nerve are responsible for emission






21. What is the number 1 risk factor for cervical dysplasia or carcinoma in situ






22. Which androgen is responsible for differentiation of epididymis - vas deferens - seminal vesicles - internal genitalia (except prostate)






23. What does estrogen to do prolaction






24. Which teratoma - mature or immature - is aggresively malignant






25. What is the genetic material in the ovum






26. increased fluid secondary to incomplete fustion with processus vaginalis






27. What is DHT responsible for in early development?






28. Which hydatidiform mole has the greater risk for malignancy






29. In what phase is meiosis II arrested






30. List the estrogens in order of decreasing potency






31. What happens to a leiomyoma in pregs and menopause and why






32. marked increased hCG - complete or partial






33. vaginal carcinoma affecting girls < 4 spindle shaped tumors cells that are desmin positive






34. increased AFP - schiller duvel bodies - yellow mucinous






35. What is a complication of invasive carcinoma






36. Is fertility compromised in double Y males?






37. What forms the blood testis barrier?






38. How does BPH present






39. Histological subtype of fibrocystic with increased number of epithelial cell lauers in terminal duct lobule - women over 30






40. Uterin fundus to labia majora






41. blood containing cyst from ovarian endometriosis that varies with menstrual cycle






42. What is the typical cell change in HPV infection






43. Why does the skin resemble an orange peel in inflammatory type of maligantn breast tumor






44. Which androgen is responsible for the deepening of the voice






45. What are the 3 androgens






46. What is the risk for carcinoma among patients with intraductal papilloma






47. When is follicular growth the fastest?






48. What is the genetic material in the primary oocyte?






49. What changes in the aorta are common in Turner's?






50. hyperandrogenism due to deranged steroid synthesis by theca cells - increased LH leading to anovulation







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