Test your basic knowledge |

First Aid: Reproductive

Instructions:
  • Answer 50 questions in 15 minutes.
  • If you are not ready to take this test, you can study here.
  • Match each statement with the correct term.
  • Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.

This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. Where does fertilization most commonly occur?






2. Which ligament is the derivative of the gubernaculum and travels through the inguinal canal






3. What is the genetic material in the secondary oocyte?






4. Which androgen is responsible for differentiation of epididymis - vas deferens - seminal vesicles - internal genitalia (except prostate)






5. Which cells secrete beta hCG






6. histologic subtype of fibrocystic with increased acini and intralobular fibrosis






7. Which gynecologic tumors have the worst prognosis?






8. <0.5 L of amniotic fluid






9. What cellular structure is the acrosome derived from?






10. Ecsematous patches on nipple and/or vulva - suggests underlying carcinoma

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11. histologic type of fibrocystic with fluid filled - blue dome and ductal dilation






12. Breast path - diseeases of the lobules






13. reinke crystals - androgen producing - gynecomastia in men - precocious puberty in boys - golden brown color






14. in males - are mature teratomas malignant? What is the case for females






15. What complications are associated with oligohydramnios

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16. Red velvety plaques - usually involving the glans - similar to Bowen's






17. hyperandrogenism due to deranged steroid synthesis by theca cells - increased LH leading to anovulation






18. histo: simple columnar epithelium






19. < 21 day cycle






20. Where is androstenedione made?






21. testicular masses that can be transilluminated






22. gray - soliarty - crusty plaque - usually on the shaft of the penis or on the scrotum - peak incidence in 5th decade of life - can progress to invasive SCC in < 10% of cases

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23. Atypical cells in epithelial hyperplasia






24. histo: simple cuboidal epithelium






25. defect in androgen receptor resulting in normal appearing female - rudimentry vagina - no uterus or uterine tubes






26. Connects cervix to side wall of pelvis - contains uterine vessels






27. Multiple papular lesions on the penis - affects younger age group - usually does not become invasive






28. A leimyoma is overgrowth of what cell






29. What are risk factors for placenta acreta






30. When does endometiral carcinoma usually occur






31. What is HELLP syndrome






32. What are common causes of hyperestrogenism

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33. What are the treatments for BPH






34. bundles of spindle shaped fibroblasts - pulling sensation in the groin






35. tumor that fills ductal lumen - arises from ductal hyperplasia - early malignancy without BM penetration






36. What are the functions of oxytocin - maybe






37. What hormones regulate sperm creation?






38. What common valvular abnormality is common in Turner's






39. What are the associated risk factors for malignant breast tumors






40. What estrogen does the ovary secrete






41. Where is the enlargement found in BPH






42. dx with increased testosterone and increased LH






43. What pathologic states cause increases in hCG






44. Which androgen is responsible for libido






45. malignancy in ovaries - testis - sacrococcygeal area of young children - yellow friable solid masses with 50% having schiller - duval bodies






46. dx with decreased testosterone - increased LH






47. tumor with firm fibrous - 'rock hard' mass with sharp margins and small glandular duct like cells - worst prognosis and most common






48. Which androgen is responsible for the deepening of the voice






49. How does endometriosis cause infertility






50. How does endometrial hyperplasia manifest clinically