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Test your basic knowledge |
MCAT Biology Circulatory System
Start Test
Study First
Subjects
:
mcat
,
health-sciences
Instructions:
Answer 50 questions in 15 minutes.
If you are not ready to take this test, you can
study here
.
Match each statement with the correct term.
Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.
This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. Adequate circulation - but O2 supply is reduced (no build up waste products or loss of nutrients)
AV node
nutrients - wastes - and WBC
when person that is Rh - is exposed to blood that is Rh+
hypoxia
2. Rh factor that follows dominant pattern (Rh+ in heterozygote)
stroke volume
Granulocytes
Lipoproteins
Rh blood group
3. Aggregate at site of damage to a blood vessel and form a platelet plug to stop bleeding
Granulocytes
Platelet fxn
Coronary arteries
basophil
4. 2 lymphocytes
It is the same - otherwise it would lead to fluid backup
B cells and T cells
Platelet fxn
neutrophil
5. Request by tissues to increase blood flow - where build up of metabolic waste causes arterioles to dialate
local autoregulation
tricuspid valve
atria
Relaxed
6. Pass through the capillaries in order to patrol the tissue for invading organisms; only macrophages and neutrophils can squeeze through cleft
Temperature or metabolic rate
Capillaries
WBC
Ohm's law
7. Bone marrow cells that give rise to RBC and platelets
nutrients
Perfusion
megakaryocytes
atrioventricular valves
8. Open when threshold is reached causing membrane potential to increase/depolarize; operate slower than Na channels
AV node
varicose veins
Hepatic portal vein
Ca channels
9. Protein in RBC that transport O2 though the blood since O2 is too hydrophobic in plasma; protein has 4 subunits that change confirmation cooperatively depending on the concentration of O2
Hemoglobin
Ohm's law
B cells and T cells
Secondary transportation of CO2 in the blood
10. Blood clot or scab circulating in bloodstream
Capillaries
Thrombus
fats
Diastole
11. When do Rh antibodies develop?
when person that is Rh - is exposed to blood that is Rh+
fibrin
Secondary transportation of CO2 in the blood
coronary sinus
12. Valves between the ventricle and the atria to prevent back flow
systemic circulation
increase vagal signal and inhibits sympathetic input
atrioventricular valves
coronary sinus
13. Contraction of the ventricles - where pressure increases rapidly - causing AV valves to close - Marks the beginning of the 'lub' sound
Systole
oncotic pressure
to transport O2 to tissues and CO2 to the lungs
albumin b/c it provides the bulk of oncotic pressure in blood vessels - preventing edema
14. Caused by closure of Ca channels and opening of K channels
Temperature or metabolic rate
Repolarization of nodes
Ohm's law
urea
15. Receptors in the carotid arteries and aortic arch that notify CNS if blood pressure is high or low
Third transportation of CO2 in the blood
heart rate
Bundle of His
Baroreceptors
16. Osmotic pressure in capillaries due to plasma proteins
atria and ventricles
to transport O2 to tissues and CO2 to the lungs
oncotic pressure
bone marrow
17. Pool of deoxygenated blood at low pressure - which collects blood from coronary veins - Only deoxygenated blood to not enter the right atrium via the vena cava
Baroreceptors
coronary sinus
high osmolarity of tissues
heart rate
18. Flow of blood through a tissue
Ohm's law
Diastole
Perfusion
hepatic portal system and hypothalamic - hypophosial portal system
19. Receives deoxygenated blood from systemic circulation (superior and inferior vena cava)
Thrombus
pulse pressure
Right atrium
oncotic pressure
20. Path where impulse travels from SA to AV node
Internodal tract
atria
eosinophil
Temperature or metabolic rate
21. Opposing friction force to flow - which increases with decreased radius; determined by degree of contraction of arterial smooth muscle
resistance
Ischemia
Diastole
tricuspid valve
22. The principle sugar in blood that maintains a relatively constant concentration for adequate nutrition
albumin
Glucose
varicose veins
Immunoglobulins (antibodies)
23. Heart rate *stroke volume= (units)
Vagal Signal
cardiac output (L/min)
Sickle cell anemia
Hepatic portal vein
24. The difference in pressure divided blood flow; controlled by the sympathetic nervous system generating adrenergic tone
Diastole is longer
Temperature or metabolic rate
Baroreceptors
Peripheral resistance
25. Occurs when increased cardiac output is needed; the postganglionic nerve directly innervates the heart - releasing norepinephrine - increasing heart rate and force of contraction
Right atrium
Frank - Starling Effect
Sympathetic regulation of heart
B cells and T cells
26. Connected to SA node via internodal tract - and passes signal to Common bundle of His to contract ventricles
Blood plasma
AV node
glycolysis. RBC have no ETC - FA oxidation - or TCA cycle
coronary sinus
27. Voltage - gated channels that stay open longer than Na channels and open later responsible for the plateau phase of cardiac muscle contraction
systolic blood pressure
coronary sinus
Functional syncytium
Slow Ca channels
28. Which is longer - diastole or systole?
amino acids and glucose
heart
Lipoproteins
Diastole is longer
29. Neural sympathetic input by postganglionic neuron of norepinephrine innervating arterial smooth muscle
adrenergic tone
oncotic pressure
venous return
Coronary veins
30. Crosses septum and connects to Purkinje fibers to allow coordinated contraction of ventricles. Key is that is slows transmission across septum to allow ventricles to fully fill before contraction
Platelet fxn
Baroreceptors
Bundle of His
5 phases of cardiac muscle cell contraction
31. Mother has Rh - blood with Rh+ antibodies that attack the babies Rh+ blood
fats
Systole
stroke volume
Hemolytic disease of a newborn
32. Why is the SA node the primary pacemaker?
It has the most Na leak channels - allowing to reach threshold potential first; all other nodes leak - but rate at as quick of a rate
Diastole is longer
tricuspid valve
AB+ since no antibodies are made to any blood type
33. Maximize entry of Ca into the cell by allowing entry of Ca extracellular environment; leads to contraction of actin - myosin fibers
Hepatic portal vein
5 phases of cardiac muscle cell contraction
T- tubules
atria
34. Absorbed by the GI tract and brought to the liver via the hepatic portal vein - where they are stored in the liver and enter the blood stream when needed
urea
Waste
stroke volume
amino acids and glucose
35. Phagocytose bacteria resulting in pus; amoeboid motility and chemotaxis
neutrophil
Systole
tricuspid valve
Waste
36. Transportation of blood though the body and exchange of material btw blood and tissues
ventricles
Platelet fxn
Fxn of circulatory system
venous return
37. Inadequate blood flow - resulting in tissue damage due to shortage of O2 and nutrients - and increase of metabolic waste
neutrophil
Ischemia
glycolysis. RBC have no ETC - FA oxidation - or TCA cycle
Pulmonary and aortic semilunar valves
38. Muscular pump that forces blood through series of branching vessels
heart
capillaries
albumin b/c it provides the bulk of oncotic pressure in blood vessels - preventing edema
Systole
39. Ensure the one - way flow through the circulatory system
varicose veins
cardiac output (L/min)
valves
fibrin
40. Amount of blood pumped w/ each systolic contraction
SA node
Erythrocytes
stroke volume
albumin b/c it provides the bulk of oncotic pressure in blood vessels - preventing edema
41. AV valve between right atrium and right ventricle
Thrombus
tricuspid valve
Portal systems
pulse pressure
42. Precursor to fibrin - which is necessary for blood clotting
fibrinogen
Ohm's law
It is the same - otherwise it would lead to fluid backup
Blood plasma
43. 2 ways to increase venous return
T- tubules
atria
1. increase total blood volume by retaining more H2O 2. Contraction of large veins - propelling blood toward the heart
fibrin
44. Confirmation of hemoglobin with O2 bound - where affinity is high 1. pH 2. pCO2 3.
Right atrium
Perfusion
Coronary arteries
Relaxed
45. Key proteins for the function of the immune system that are produced and released by B- cells
fibrin
basophil
venous return
Immunoglobulins (antibodies)
46. Highest blood pressure that occurs during ventricular contraction
resistance
basophil
systolic blood pressure
Diastole
47. Buffer in blood. Keeps pH around 7.4
adipocytes
primary bicarbonate generated from CO2.
AB+ since no antibodies are made to any blood type
Internodal tract
48. Return of blood to the heart by the vena cava - where increased venous return causes increased stretching of the muscle (increases stroke volume)
bicuspid (mitral) valve
Lipoproteins
fibrinogen
venous return
49. 73% of CO2 converted to carbonic acid by carbonic anhydrase - and carbonic acid is converted to bicarbonate - which acts a buffer
Slow Ca channels
Systole
nutrients - wastes - and WBC
Primary transportation fo CO2 in the blood
50. Absorbed in the intestine and packaged in chylomicrons - which enter the lymphatic system - and dumped into the subclavian vein via the thoracic duct; the liver takes fats once in blood - converts them to another lipoprotein and sends them to adipocy
Valves of the venous system
Erythropoetin
fats
veins