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Test your basic knowledge |
MCAT Biology Circulatory System
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Subjects
:
mcat
,
health-sciences
Instructions:
Answer 50 questions in 15 minutes.
If you are not ready to take this test, you can
study here
.
Match each statement with the correct term.
Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.
This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. Amount of blood pumped w/ each systolic contraction
stroke volume
Sympathetic regulation of heart
5 phases of cardiac muscle cell contraction
serum
2. The principle sugar in blood that maintains a relatively constant concentration for adequate nutrition
Glucose
2 components of antigens
serum
It has the most Na leak channels - allowing to reach threshold potential first; all other nodes leak - but rate at as quick of a rate
3. What is the direct cause of edema?
1. increase total blood volume by retaining more H2O 2. Contraction of large veins - propelling blood toward the heart
ABO blood group
stroke volume
increased hydrostatic pressure in the capillaries - which increases the fluid that leaks out of the capillaries into the interstitum
4. At the end of the capillary - is the osmotic pressure high or low?
High since the concentration of plasma proteins has increased due to movement of water
hemostasis
systemic circulation
heart rate
5. Pump blood out of the heart at high pressures into arteries
ventricles
oncotic pressure
Temperature or metabolic rate
Sympathetic regulation of heart
6. Excessive bleeding that results from defective proteins
hemophilia
varicose veins
bilirubin
Waste
7. Because the veins have essentially 0 pressure - these valves ensure one - way flow - skeletal muscle contraction encourages flow through veins
Systole
Erythropoetin
1. increase total blood volume by retaining more H2O 2. Contraction of large veins - propelling blood toward the heart
Valves of the venous system
8. Stretching to greater degree of heart muscle causes more forceful contraction; stretching increase occur by increasing fluid volume
Granulocytes
Erythrocytes
Frank - Starling Effect
adipocytes
9. Large particles consisting of fats - cholesterol - and carrier proteins; transport lipids through the blood stream
Secondary transportation of CO2 in the blood
systolic blood pressure
Immunoglobulins (antibodies)
Lipoproteins
10. Metabolic waste product in breakdown of amino acids
macrophage
Functional syncytium
urea
arteries
11. Opposing friction force to flow - which increases with decreased radius; determined by degree of contraction of arterial smooth muscle
High since the concentration of plasma proteins has increased due to movement of water
local autoregulation
resistance
Relaxed
12. Ensure the one - way flow through the circulatory system
valves
Capillaries
Hepatic portal vein
albumin
13. Where blood passes through 2 sets of capillaries before returning to the heart; Evolved as direct transport routes
Hemoglobin
Diastole is longer
cardiac output (L/min)
Portal systems
14. Buffer in blood. Keeps pH around 7.4
WBC
local autoregulation
primary bicarbonate generated from CO2.
adrenergic tone
15. Pool of deoxygenated blood at low pressure - which collects blood from coronary veins - Only deoxygenated blood to not enter the right atrium via the vena cava
Ischemia
adrenergic tone
coronary sinus
chylomicrons
16. As low as pressure gets btw heart beats in arteries
Vagal Signal
diastolic blood pressure
systolic blood pressure
arteries
17. Valves between the ventricle and the atria to prevent back flow
Ohm's law
atrioventricular valves
Na leak channels
High since the concentration of plasma proteins has increased due to movement of water
18. Vessels where deoxygenated blood from coronary sinus continue to flow into heart
High since the concentration of plasma proteins has increased due to movement of water
hemostasis
Functional syncytium
Coronary veins
19. 1. depolarization caused by fast Na channels - where action potential through intercalated discs reaches threshold potential - opening Na channels 2. initial depolarization with Na channels closing and k channels opening - but Ca channels also open 3
local autoregulation
albumin
5 phases of cardiac muscle cell contraction
varicose veins
20. Breakdown product of the hemogloblin heme group
eosinophil
It is the same - otherwise it would lead to fluid backup
bilirubin
hemophilia
21. Caused by closure of Ca channels and opening of K channels
Waste
adipocytes
chylomicrons
Repolarization of nodes
22. Lipoproteins that enter lacteal vessels of lymphatic system in the intestinal wall
Granulocytes
macrophage
chylomicrons
veins
23. Reservoirs where blood collects from veins
atria
Hepatic portal vein
Erythropoetin
pulmonary circulation
24. The difference btw systolic and diastolic blood pressures
Slow Ca channels
pulse pressure
systemic arterial blood pressure
T- tubules
25. Receptors in the carotid arteries and aortic arch that notify CNS if blood pressure is high or low
primary bicarbonate generated from CO2.
Rh blood group
resistance
Baroreceptors
26. First branches from the aorta that provide the heart's blood supply
CNS decreases vagal signal and sympathetic input increases
Coronary arteries
Right atrium
O- since there are no surface antigens for antibodies to bind to...
27. Allow Na to leak across membrane - causing cell potential to get closer to threshold potential; allow threshold to be reached for Ca channels to open let Ca into the cell
to transport O2 to tissues and CO2 to the lungs
nutrients
Bundle of His
Na leak channels
28. 2 portal systems to know
hepatic portal system and hypothalamic - hypophosial portal system
oncotic pressure
Coronary veins
bone marrow
29. 2 ways to increase venous return
bone marrow
Valves of the venous system
1. increase total blood volume by retaining more H2O 2. Contraction of large veins - propelling blood toward the heart
Functional syncytium
30. Inadequate blood flow - resulting in tissue damage due to shortage of O2 and nutrients - and increase of metabolic waste
Temperature or metabolic rate
Ischemia
1. increase total blood volume by retaining more H2O 2. Contraction of large veins - propelling blood toward the heart
Portal systems
31. Blood clot or scab circulating in bloodstream
High since the concentration of plasma proteins has increased due to movement of water
Thrombus
B cells and T cells
Hemolytic disease of a newborn
32. What is the most important plasma protein in the body? Why?
albumin b/c it provides the bulk of oncotic pressure in blood vessels - preventing edema
Granulocytes
Inflammation
Right atrium
33. 3 factors that dictate the affinity of hemoglobin for O2
atria and ventricles
bicuspid (mitral) valve
Temperature or metabolic rate
systolic blood pressure
34. Force per unit area exerted by blood on walls of arteries
systemic arterial blood pressure
venous return
serum
Diastole
35. Glycoproteins that are coded for by 3 alleles (A - B - i)
coronary sinus
cardiac output (L/min)
Pulmonary and aortic semilunar valves
ABO blood group
36. AV valve between left atrium and left ventricle
Lipoproteins
bicuspid (mitral) valve
Perfusion
1. increase total blood volume by retaining more H2O 2. Contraction of large veins - propelling blood toward the heart
37. Peptide hormone secreted from the kidneys to increase RBC production in bone marrow
eosinophil
2 components of antigens
Functional syncytium
Erythropoetin
38. Return of blood to the heart by the vena cava - where increased venous return causes increased stretching of the muscle (increases stroke volume)
Diastole
venous return
fibrinogen
Spleen and liver
39. Body's mechanism of preventing bleeding
Sickle cell anemia
hemostasis
Tense
Fast Na channels
40. Connects the two capillary beds of the intestine and the liver
Hemolytic disease of a newborn
hepatic portal system and hypothalamic - hypophosial portal system
Hepatic portal vein
albumin
41. When do Rh antibodies develop?
when person that is Rh - is exposed to blood that is Rh+
High since the concentration of plasma proteins has increased due to movement of water
diastolic blood pressure
hepatic portal system and hypothalamic - hypophosial portal system
42. Response by CNS when blood pressure is too high
Secondary transportation of CO2 in the blood
Hepatic portal vein
increase vagal signal and inhibits sympathetic input
hepatic portal system and hypothalamic - hypophosial portal system
43. When the valve of a vein fails and back flow occurs; blood not being moved toward the heart
local autoregulation
varicose veins
Repolarization of nodes
adrenergic tone
44. Gap junctions in the cardiac muscle - where depolarization is communicated directly btw cytoplasm of neighboring cardiac cells
Intercalated discs
systolic blood pressure
Primary transportation fo CO2 in the blood
fibrin
45. Filling of the ventricles by squeezing of the atria - marks the beginning of the 'dub' sound
Fxn of circulatory system
cardiac output (L/min)
Diastole
venous return
46. Muscular pump that forces blood through series of branching vessels
Spleen and liver
glycolysis. RBC have no ETC - FA oxidation - or TCA cycle
Ischemia
heart
47. Difference in pressure=blood flow (L/min)*resitance ^P=Q*R
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48. Where do all components of the blood develop from?
ventricles
bone marrow
venous return
Primary transportation fo CO2 in the blood
49. 20% transported stuck to hemoglobin; why increased pCO2 decreases affinity of O2
Secondary transportation of CO2 in the blood
Hepatic portal vein
heart
urea
50. Absorbed in the intestine and packaged in chylomicrons - which enter the lymphatic system - and dumped into the subclavian vein via the thoracic duct; the liver takes fats once in blood - converts them to another lipoprotein and sends them to adipocy
SA node
glycolysis. RBC have no ETC - FA oxidation - or TCA cycle
fats
Fxn of circulatory system
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