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Test your basic knowledge |
MCAT Biology Circulatory System
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Subjects
:
mcat
,
health-sciences
Instructions:
Answer 50 questions in 15 minutes.
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study here
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Match each statement with the correct term.
Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.
This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. As low as pressure gets btw heart beats in arteries
diastolic blood pressure
Rh blood group
cardiac output (L/min)
hemostasis
2. What is the most important plasma protein in the body? Why?
Na leak channels
albumin b/c it provides the bulk of oncotic pressure in blood vessels - preventing edema
Functional syncytium
nutrients
3. Produced during cell metabolism and diffuses through the endothelial cells into the blood stream - where it is picked up by the liver and converted to forms that can be excreted (all other wastes are picked up by the kidneys)
Waste
when person that is Rh - is exposed to blood that is Rh+
Temperature or metabolic rate
CNS decreases vagal signal and sympathetic input increases
4. Because the veins have essentially 0 pressure - these valves ensure one - way flow - skeletal muscle contraction encourages flow through veins
Peripheral resistance
arteries
pulmonary circulation
Valves of the venous system
5. Amount of blood pumped w/ each systolic contraction
oncotic pressure
primary bicarbonate generated from CO2.
stroke volume
T- tubules
6. Maximize entry of Ca into the cell by allowing entry of Ca extracellular environment; leads to contraction of actin - myosin fibers
SA node
WBC
T- tubules
Hepatic portal vein
7. Gap junctions in the cardiac muscle - where depolarization is communicated directly btw cytoplasm of neighboring cardiac cells
Intercalated discs
megakaryocytes
ventricles
Hemolytic disease of a newborn
8. Occurs when increased cardiac output is needed; the postganglionic nerve directly innervates the heart - releasing norepinephrine - increasing heart rate and force of contraction
Sympathetic regulation of heart
eosinophil
ventricles
bone marrow
9. Flow of blood from the heart to the lungs - pumped by the right side of the heart
pulmonary circulation
local autoregulation
basophil
AB+ since no antibodies are made to any blood type
10. Phagocytose bacteria resulting in pus; amoeboid motility and chemotaxis
nutrients
neutrophil
atrioventricular valves
High since the concentration of plasma proteins has increased due to movement of water
11. When do Rh antibodies develop?
Platelet fxn
when person that is Rh - is exposed to blood that is Rh+
arteries
Blood plasma
12. Number of systole contractions per unit time
heart rate
valves
Sympathetic regulation of heart
Vagal Signal
13. Transportation of blood though the body and exchange of material btw blood and tissues
Granulocytes
fibrinogen
AV node
Fxn of circulatory system
14. Key proteins for the function of the immune system that are produced and released by B- cells
nutrients - wastes - and WBC
Vagal Signal
Immunoglobulins (antibodies)
oncotic pressure
15. Excessive bleeding that results from defective proteins
Rh blood group
ABO blood group
hemophilia
Sickle cell anemia
16. 2 chambers of the heart
atria and ventricles
Bundle of His
CNS decreases vagal signal and sympathetic input increases
increased hydrostatic pressure in the capillaries - which increases the fluid that leaks out of the capillaries into the interstitum
17. Confirmation of hemoglobin with O2 bound - where affinity is high 1. pH 2. pCO2 3.
Relaxed
bone marrow
neutrophil
adrenergic tone
18. Muscular pump that forces blood through series of branching vessels
Right atrium
Tense
It is the same - otherwise it would lead to fluid backup
heart
19. The difference in pressure divided blood flow; controlled by the sympathetic nervous system generating adrenergic tone
albumin b/c it provides the bulk of oncotic pressure in blood vessels - preventing edema
Peripheral resistance
Erythrocytes
adrenergic tone
20. Filling of the ventricles by squeezing of the atria - marks the beginning of the 'dub' sound
Primary transportation fo CO2 in the blood
Diastole
to transport O2 to tissues and CO2 to the lungs
atria
21. Response by CNS when blood pressure is too low
Thrombus
CNS decreases vagal signal and sympathetic input increases
urea
B cells and T cells
22. Adequate circulation - but O2 supply is reduced (no build up waste products or loss of nutrients)
Hepatic portal vein
T- tubules
hypoxia
hemostasis
23. Highest blood pressure that occurs during ventricular contraction
AB+ since no antibodies are made to any blood type
systolic blood pressure
bicuspid (mitral) valve
Lipoproteins
24. Have single layer endothelial cells w/ spaces in between cells called intercellular cleft
Immunoglobulins (antibodies)
Valves of the venous system
oncotic pressure
Capillaries
25. Breakdown product of the hemogloblin heme group
albumin
bilirubin
Immunoglobulins (antibodies)
Sickle cell anemia
26. Absorbed by the GI tract and brought to the liver via the hepatic portal vein - where they are stored in the liver and enter the blood stream when needed
tricuspid valve
hemostasis
amino acids and glucose
Coronary veins
27. Glucose - amino acids - and fats
nutrients
Secondary transportation of CO2 in the blood
adrenergic tone
Hemolytic disease of a newborn
28. Ensure the one - way flow through the circulatory system
AV node
T- tubules
valves
CNS decreases vagal signal and sympathetic input increases
29. Protein in RBC that transport O2 though the blood since O2 is too hydrophobic in plasma; protein has 4 subunits that change confirmation cooperatively depending on the concentration of O2
albumin b/c it provides the bulk of oncotic pressure in blood vessels - preventing edema
Hemoglobin
Erythropoetin
increased hydrostatic pressure in the capillaries - which increases the fluid that leaks out of the capillaries into the interstitum
30. Inadequate blood flow - resulting in tissue damage due to shortage of O2 and nutrients - and increase of metabolic waste
Internodal tract
Coronary arteries
glycolysis. RBC have no ETC - FA oxidation - or TCA cycle
Ischemia
31. Valves between the large arteries and the ventricles
Glucose
amino acids and glucose
varicose veins
Pulmonary and aortic semilunar valves
32. Tissue which the cytoplasm of different cells communicate via gap junctions
Functional syncytium
Erythrocytes
Sickle cell anemia
increase vagal signal and inhibits sympathetic input
33. Return of blood to the heart by the vena cava - where increased venous return causes increased stretching of the muscle (increases stroke volume)
venous return
Fxn of circulatory system
Coronary veins
Thrombus
34. Purpose of erythrocytes?
hypoxia
Rh blood group
to transport O2 to tissues and CO2 to the lungs
Pulmonary and aortic semilunar valves
35. 55% of whole blood that is composed of electrolytes - lipoproteins - sugars - buffer - and metabolic waste
hemostasis
Blood plasma
Frank - Starling Effect
Ohm's law
36. Region that initiates start of cardiac cycle - which acts as a pacemaker of the heart; has unstable resting potential due to Na leak channels
atria
Baroreceptors
Relaxed
SA node
37. Stretching to greater degree of heart muscle causes more forceful contraction; stretching increase occur by increasing fluid volume
Hemolytic disease of a newborn
Secondary transportation of CO2 in the blood
Frank - Starling Effect
Peripheral resistance
38. Where are RBCs broken down?
Spleen and liver
Waste
Inflammation
stroke volume
39. Control of by ANS of rate of contraction through the Vagus nerve. Postganglionic release in SA node of ACH inhibits depolarization
urea
arteries
Systole
Vagal Signal
40. Voltage - gated channels that stay open longer than Na channels and open later responsible for the plateau phase of cardiac muscle contraction
Waste
Coronary veins
Slow Ca channels
nutrients - wastes - and WBC
41. Allow Na to leak across membrane - causing cell potential to get closer to threshold potential; allow threshold to be reached for Ca channels to open let Ca into the cell
resistance
varicose veins
venous blood pressure
Na leak channels
42. At position 6 - missense mutation substitutes valine for glutamate. valine is hydrophobic - where glutamate was charged. It is an autosomal recessive disease where RBCs accumulated in small vessels - heterozygote for (blank) shows resistance to malar
nutrients
Sickle cell anemia
Hemoglobin
heart rate
43. Hematocrit or RBC those compose 35-45% of the blood; cells are non - nucleated and have no organelles. Acquire ATP through glycolysis have biconcave shape to maximize surface area for binding O2
Erythrocytes
Baroreceptors
Right atrium
Hemoglobin
44. Vessels that carry blood back to the heart at low pressure
Erythropoetin
veins
Waste
Diastole
45. CO2 is soluble in H2O - and thus some is dissolved and carried to lungs and tissues in plasma - O2 is not soluble in plasma at all
Third transportation of CO2 in the blood
It has the most Na leak channels - allowing to reach threshold potential first; all other nodes leak - but rate at as quick of a rate
Glucose
bone marrow
46. Flow of blood through a tissue
cardiac output (L/min)
Granulocytes
Perfusion
atria and ventricles
47. Vessels that carry blood away from the heart at high pressure
Functional syncytium
hemophilia
arteries
chylomicrons
48. Receptors in the carotid arteries and aortic arch that notify CNS if blood pressure is high or low
Baroreceptors
Hepatic portal vein
adipocytes
diastolic blood pressure
49. 1. depolarization caused by fast Na channels - where action potential through intercalated discs reaches threshold potential - opening Na channels 2. initial depolarization with Na channels closing and k channels opening - but Ca channels also open 3
arteries
oncotic pressure
5 phases of cardiac muscle cell contraction
Primary transportation fo CO2 in the blood
50. Metabolic waste product in breakdown of amino acids
systemic arterial blood pressure
valves
veins
urea
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