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Test your basic knowledge |
MCAT Biology Circulatory System
Start Test
Study First
Subjects
:
mcat
,
health-sciences
Instructions:
Answer 50 questions in 15 minutes.
If you are not ready to take this test, you can
study here
.
Match each statement with the correct term.
Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.
This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. Is cardiac output the same or different btw the two ventricles?
chylomicrons
high osmolarity of tissues
increase vagal signal and inhibits sympathetic input
It is the same - otherwise it would lead to fluid backup
2. What is the direct cause of edema?
heart
increased hydrostatic pressure in the capillaries - which increases the fluid that leaks out of the capillaries into the interstitum
Blood plasma
coronary sinus
3. Where blood passes through 2 sets of capillaries before returning to the heart; Evolved as direct transport routes
Portal systems
valves
It is the same - otherwise it would lead to fluid backup
Blood plasma
4. 3 substances that can diffuse through intercellular cleft
atrioventricular valves
nutrients - wastes - and WBC
coronary sinus
AB+ since no antibodies are made to any blood type
5. Pool of deoxygenated blood at low pressure - which collects blood from coronary veins - Only deoxygenated blood to not enter the right atrium via the vena cava
coronary sinus
oncotic pressure
Sickle cell anemia
venous blood pressure
6. Allow Na to leak across membrane - causing cell potential to get closer to threshold potential; allow threshold to be reached for Ca channels to open let Ca into the cell
Na leak channels
Capillaries
Internodal tract
O- since there are no surface antigens for antibodies to bind to...
7. 2 ways to increase venous return
cardiac output (L/min)
1. increase total blood volume by retaining more H2O 2. Contraction of large veins - propelling blood toward the heart
stroke volume
Primary transportation fo CO2 in the blood
8. 2 portal systems to know
High since the concentration of plasma proteins has increased due to movement of water
B cells and T cells
Pulmonary and aortic semilunar valves
hepatic portal system and hypothalamic - hypophosial portal system
9. When the valve of a vein fails and back flow occurs; blood not being moved toward the heart
varicose veins
local autoregulation
WBC
Hepatic portal vein
10. CO2 is soluble in H2O - and thus some is dissolved and carried to lungs and tissues in plasma - O2 is not soluble in plasma at all
Third transportation of CO2 in the blood
bicuspid (mitral) valve
CNS decreases vagal signal and sympathetic input increases
Ohm's law
11. Amount of blood pumped w/ each systolic contraction
T- tubules
Sympathetic regulation of heart
stroke volume
resistance
12. Force per unit area exerted by blood on walls of arteries
albumin
Relaxed
systemic arterial blood pressure
increase vagal signal and inhibits sympathetic input
13. Why is the SA node the primary pacemaker?
bilirubin
It has the most Na leak channels - allowing to reach threshold potential first; all other nodes leak - but rate at as quick of a rate
Frank - Starling Effect
nutrients - wastes - and WBC
14. Response by CNS when blood pressure is too low
Slow Ca channels
CNS decreases vagal signal and sympathetic input increases
heart rate
WBC
15. Vessels that carry blood away from the heart at high pressure
Diastole is longer
Erythropoetin
systemic circulation
arteries
16. At position 6 - missense mutation substitutes valine for glutamate. valine is hydrophobic - where glutamate was charged. It is an autosomal recessive disease where RBCs accumulated in small vessels - heterozygote for (blank) shows resistance to malar
Hemoglobin
eosinophil
Sickle cell anemia
systemic arterial blood pressure
17. Gap junctions in the cardiac muscle - where depolarization is communicated directly btw cytoplasm of neighboring cardiac cells
Intercalated discs
Hemolytic disease of a newborn
chylomicrons
Rh blood group
18. Metabolic waste product in breakdown of amino acids
adrenergic tone
Ohm's law
Coronary arteries
urea
19. Destroy parasites and are involved in allergic rxns
eosinophil
cardiac output (L/min)
systemic arterial blood pressure
T- tubules
20. The difference in pressure divided blood flow; controlled by the sympathetic nervous system generating adrenergic tone
venous blood pressure
Peripheral resistance
Coronary veins
Right atrium
21. Where are RBCs broken down?
Spleen and liver
fibrinogen
primary bicarbonate generated from CO2.
serum
22. Body's mechanism of preventing bleeding
venous return
Vagal Signal
hemostasis
to transport O2 to tissues and CO2 to the lungs
23. Capillaries dilate - increasing the cleft size - which allows more H2O to move through to tissues
Portal systems
Inflammation
Bundle of His
glycolysis. RBC have no ETC - FA oxidation - or TCA cycle
24. 2 chambers of the heart
venous blood pressure
atria
CNS decreases vagal signal and sympathetic input increases
atria and ventricles
25. Bone marrow cells that give rise to RBC and platelets
megakaryocytes
albumin b/c it provides the bulk of oncotic pressure in blood vessels - preventing edema
Slow Ca channels
Erythrocytes
26. Region that initiates start of cardiac cycle - which acts as a pacemaker of the heart; has unstable resting potential due to Na leak channels
local autoregulation
ABO blood group
varicose veins
SA node
27. Response by CNS when blood pressure is too high
Blood plasma
Right atrium
Third transportation of CO2 in the blood
increase vagal signal and inhibits sympathetic input
28. Breakdown product of the hemogloblin heme group
bone marrow
atria and ventricles
bilirubin
fibrinogen
29. Lipoproteins that enter lacteal vessels of lymphatic system in the intestinal wall
Systole
atria
Immunoglobulins (antibodies)
chylomicrons
30. Adequate circulation - but O2 supply is reduced (no build up waste products or loss of nutrients)
hypoxia
Ischemia
bicuspid (mitral) valve
Valves of the venous system
31. Produced during cell metabolism and diffuses through the endothelial cells into the blood stream - where it is picked up by the liver and converted to forms that can be excreted (all other wastes are picked up by the kidneys)
Glucose
Waste
neutrophil
fibrin
32. AV valve between left atrium and left ventricle
Primary transportation fo CO2 in the blood
Intercalated discs
bicuspid (mitral) valve
increase vagal signal and inhibits sympathetic input
33. 3 factors that dictate the affinity of hemoglobin for O2
Temperature or metabolic rate
Lipoproteins
resistance
2 components of antigens
34. Blood clot or scab circulating in bloodstream
hepatic portal system and hypothalamic - hypophosial portal system
hypoxia
Thrombus
Functional syncytium
35. When do Rh antibodies develop?
when person that is Rh - is exposed to blood that is Rh+
albumin
capillaries
eosinophil
36. Confirmation of hemoglobin with O2 bound - where affinity is high 1. pH 2. pCO2 3.
Relaxed
Na leak channels
hemostasis
Erythropoetin
37. When do semilunar valves close?
oncotic pressure
Erythrocytes
Arterial pressure=ventricular pressure
nutrients - wastes - and WBC
38. ABO blood group and Rh blood group
Pulmonary and aortic semilunar valves
2 components of antigens
Hepatic portal vein
Platelet fxn
39. Crosses septum and connects to Purkinje fibers to allow coordinated contraction of ventricles. Key is that is slows transmission across septum to allow ventricles to fully fill before contraction
Third transportation of CO2 in the blood
Bundle of His
Waste
eosinophil
40. Pass through the capillaries in order to patrol the tissue for invading organisms; only macrophages and neutrophils can squeeze through cleft
Relaxed
WBC
Perfusion
High since the concentration of plasma proteins has increased due to movement of water
41. Store and release histamine and are involved in allergic rxns
basophil
Fxn of circulatory system
fats
Hemolytic disease of a newborn
42. Ensure the one - way flow through the circulatory system
Cardiac muscle cells
Arterial pressure=ventricular pressure
Rh blood group
valves
43. Absorbed in the intestine and packaged in chylomicrons - which enter the lymphatic system - and dumped into the subclavian vein via the thoracic duct; the liver takes fats once in blood - converts them to another lipoprotein and sends them to adipocy
Sympathetic regulation of heart
fats
pulmonary circulation
oncotic pressure
44. First branches from the aorta that provide the heart's blood supply
Pulmonary and aortic semilunar valves
increased hydrostatic pressure in the capillaries - which increases the fluid that leaks out of the capillaries into the interstitum
chylomicrons
Coronary arteries
45. Resting membrane potential of -90mV and have long duration action potentials
albumin b/c it provides the bulk of oncotic pressure in blood vessels - preventing edema
Lipoproteins
amino acids and glucose
Cardiac muscle cells
46. Protein in RBC that transport O2 though the blood since O2 is too hydrophobic in plasma; protein has 4 subunits that change confirmation cooperatively depending on the concentration of O2
High since the concentration of plasma proteins has increased due to movement of water
Right atrium
T- tubules
Hemoglobin
47. Key proteins for the function of the immune system that are produced and released by B- cells
Platelet fxn
Immunoglobulins (antibodies)
CNS decreases vagal signal and sympathetic input increases
primary bicarbonate generated from CO2.
48. Peptide hormone secreted from the kidneys to increase RBC production in bone marrow
nutrients
B cells and T cells
Lipoproteins
Erythropoetin
49. Excessive bleeding that results from defective proteins
albumin
bone marrow
heart
hemophilia
50. Valves between the ventricle and the atria to prevent back flow
albumin b/c it provides the bulk of oncotic pressure in blood vessels - preventing edema
resistance
SA node
atrioventricular valves