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Test your basic knowledge |
MCAT Biology Circulatory System
Start Test
Study First
Subjects
:
mcat
,
health-sciences
Instructions:
Answer 50 questions in 15 minutes.
If you are not ready to take this test, you can
study here
.
Match each statement with the correct term.
Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.
This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. Pool of deoxygenated blood at low pressure - which collects blood from coronary veins - Only deoxygenated blood to not enter the right atrium via the vena cava
heart rate
pulse pressure
coronary sinus
increased hydrostatic pressure in the capillaries - which increases the fluid that leaks out of the capillaries into the interstitum
2. 1. depolarization caused by fast Na channels - where action potential through intercalated discs reaches threshold potential - opening Na channels 2. initial depolarization with Na channels closing and k channels opening - but Ca channels also open 3
5 phases of cardiac muscle cell contraction
pulmonary circulation
Na leak channels
Fxn of circulatory system
3. Key proteins for the function of the immune system that are produced and released by B- cells
Blood plasma
Immunoglobulins (antibodies)
resistance
atrioventricular valves
4. Have single layer endothelial cells w/ spaces in between cells called intercellular cleft
Capillaries
Waste
macrophage
Erythropoetin
5. Pass through the capillaries in order to patrol the tissue for invading organisms; only macrophages and neutrophils can squeeze through cleft
basophil
ABO blood group
Primary transportation fo CO2 in the blood
WBC
6. 2 chambers of the heart
Coronary arteries
atria and ventricles
megakaryocytes
Third transportation of CO2 in the blood
7. Stretching to greater degree of heart muscle causes more forceful contraction; stretching increase occur by increasing fluid volume
Immunoglobulins (antibodies)
Frank - Starling Effect
oncotic pressure
adipocytes
8. Essentially 0 mmHg - which results b/c of branching of vessels dissipating pressure to overcome resistance
pulmonary circulation
venous blood pressure
albumin
fibrin
9. CO2 is soluble in H2O - and thus some is dissolved and carried to lungs and tissues in plasma - O2 is not soluble in plasma at all
Third transportation of CO2 in the blood
oncotic pressure
glycolysis. RBC have no ETC - FA oxidation - or TCA cycle
Waste
10. What causes tendency of water flow out of blood?
high osmolarity of tissues
Peripheral resistance
Thrombus
pulmonary circulation
11. Valves between the large arteries and the ventricles
stroke volume
Frank - Starling Effect
It has the most Na leak channels - allowing to reach threshold potential first; all other nodes leak - but rate at as quick of a rate
Pulmonary and aortic semilunar valves
12. Crosses septum and connects to Purkinje fibers to allow coordinated contraction of ventricles. Key is that is slows transmission across septum to allow ventricles to fully fill before contraction
local autoregulation
arteries
resistance
Bundle of His
13. Protein in RBC that transport O2 though the blood since O2 is too hydrophobic in plasma; protein has 4 subunits that change confirmation cooperatively depending on the concentration of O2
Capillaries
Bundle of His
serum
Hemoglobin
14. Capillaries dilate - increasing the cleft size - which allows more H2O to move through to tissues
Inflammation
Functional syncytium
bicuspid (mitral) valve
Primary transportation fo CO2 in the blood
15. Purpose of erythrocytes?
Secondary transportation of CO2 in the blood
to transport O2 to tissues and CO2 to the lungs
Intercalated discs
Valves of the venous system
16. Inadequate blood flow - resulting in tissue damage due to shortage of O2 and nutrients - and increase of metabolic waste
Immunoglobulins (antibodies)
increase vagal signal and inhibits sympathetic input
Ischemia
Fast Na channels
17. Blood clot or scab circulating in bloodstream
Arterial pressure=ventricular pressure
chylomicrons
Thrombus
Slow Ca channels
18. At position 6 - missense mutation substitutes valine for glutamate. valine is hydrophobic - where glutamate was charged. It is an autosomal recessive disease where RBCs accumulated in small vessels - heterozygote for (blank) shows resistance to malar
venous blood pressure
Sickle cell anemia
Thrombus
glycolysis. RBC have no ETC - FA oxidation - or TCA cycle
19. Opposing friction force to flow - which increases with decreased radius; determined by degree of contraction of arterial smooth muscle
systemic circulation
Coronary veins
resistance
Granulocytes
20. Connected to SA node via internodal tract - and passes signal to Common bundle of His to contract ventricles
systolic blood pressure
AV node
Thrombus
Ohm's law
21. Reservoirs where blood collects from veins
atria and ventricles
tricuspid valve
Glucose
atria
22. AV valve between right atrium and right ventricle
coronary sinus
Internodal tract
tricuspid valve
veins
23. Transportation of blood though the body and exchange of material btw blood and tissues
Erythrocytes
bicuspid (mitral) valve
cardiac output (L/min)
Fxn of circulatory system
24. Highest blood pressure that occurs during ventricular contraction
fibrin
It is the same - otherwise it would lead to fluid backup
systolic blood pressure
Systole
25. Gap junctions in the cardiac muscle - where depolarization is communicated directly btw cytoplasm of neighboring cardiac cells
Intercalated discs
high osmolarity of tissues
atrioventricular valves
Third transportation of CO2 in the blood
26. Receives deoxygenated blood from systemic circulation (superior and inferior vena cava)
fibrin
Right atrium
Hepatic portal vein
oncotic pressure
27. Universal donor
Coronary arteries
bilirubin
pulmonary circulation
O- since there are no surface antigens for antibodies to bind to...
28. Where blood passes through 2 sets of capillaries before returning to the heart; Evolved as direct transport routes
Portal systems
ABO blood group
Third transportation of CO2 in the blood
tricuspid valve
29. Which is longer - diastole or systole?
Diastole is longer
macrophage
Functional syncytium
systolic blood pressure
30. Voltage - gated channels that open quickly; open at threshold potential
Hepatic portal vein
Fast Na channels
AV node
ventricles
31. When the valve of a vein fails and back flow occurs; blood not being moved toward the heart
Ca channels
fibrinogen
varicose veins
Hepatic portal vein
32. Response by CNS when blood pressure is too high
coronary sinus
Tense
increase vagal signal and inhibits sympathetic input
T- tubules
33. First branches from the aorta that provide the heart's blood supply
fats
resistance
bone marrow
Coronary arteries
34. Number of systole contractions per unit time
Cardiac muscle cells
heart rate
Right atrium
systemic arterial blood pressure
35. Monocyte that phagocytoses debris and microorganisms - has amoeboid motility - and displays chemotaxis
macrophage
varicose veins
T- tubules
Spleen and liver
36. Absorbed in the intestine and packaged in chylomicrons - which enter the lymphatic system - and dumped into the subclavian vein via the thoracic duct; the liver takes fats once in blood - converts them to another lipoprotein and sends them to adipocy
atria and ventricles
atria
fats
Arterial pressure=ventricular pressure
37. Excessive bleeding that results from defective proteins
1. increase total blood volume by retaining more H2O 2. Contraction of large veins - propelling blood toward the heart
fats
hemophilia
Hemoglobin
38. Body's mechanism of preventing bleeding
veins
hemostasis
Waste
amino acids and glucose
39. Valves between the ventricle and the atria to prevent back flow
atrioventricular valves
Systole
coronary sinus
Immunoglobulins (antibodies)
40. Region that initiates start of cardiac cycle - which acts as a pacemaker of the heart; has unstable resting potential due to Na leak channels
Third transportation of CO2 in the blood
resistance
SA node
glycolysis. RBC have no ETC - FA oxidation - or TCA cycle
41. When do semilunar valves close?
Waste
Arterial pressure=ventricular pressure
Intercalated discs
Temperature or metabolic rate
42. Mother has Rh - blood with Rh+ antibodies that attack the babies Rh+ blood
serum
Hemolytic disease of a newborn
atria and ventricles
heart
43. When do Rh antibodies develop?
Baroreceptors
heart
stroke volume
when person that is Rh - is exposed to blood that is Rh+
44. Hematocrit or RBC those compose 35-45% of the blood; cells are non - nucleated and have no organelles. Acquire ATP through glycolysis have biconcave shape to maximize surface area for binding O2
oncotic pressure
Erythrocytes
cardiac output (L/min)
Primary transportation fo CO2 in the blood
45. Buffer in blood. Keeps pH around 7.4
albumin
Frank - Starling Effect
fibrin
primary bicarbonate generated from CO2.
46. Connects the two capillary beds of the intestine and the liver
Hepatic portal vein
Capillaries
Frank - Starling Effect
Cardiac muscle cells
47. Osmotic pressure in capillaries due to plasma proteins
Glucose
Ischemia
It has the most Na leak channels - allowing to reach threshold potential first; all other nodes leak - but rate at as quick of a rate
oncotic pressure
48. Occurs when increased cardiac output is needed; the postganglionic nerve directly innervates the heart - releasing norepinephrine - increasing heart rate and force of contraction
Sympathetic regulation of heart
hemostasis
Ischemia
bilirubin
49. Store and release histamine and are involved in allergic rxns
Hemolytic disease of a newborn
stroke volume
basophil
Bundle of His
50. Return of blood to the heart by the vena cava - where increased venous return causes increased stretching of the muscle (increases stroke volume)
valves
venous return
AB+ since no antibodies are made to any blood type
WBC