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Test your basic knowledge |
MCAT Biology Circulatory System
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Subjects
:
mcat
,
health-sciences
Instructions:
Answer 50 questions in 15 minutes.
If you are not ready to take this test, you can
study here
.
Match each statement with the correct term.
Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.
This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. Vessels where deoxygenated blood from coronary sinus continue to flow into heart
systemic arterial blood pressure
systolic blood pressure
Coronary veins
Blood plasma
2. Fat storage cells of the body
veins
Inflammation
Pulmonary and aortic semilunar valves
adipocytes
3. Connects the two capillary beds of the intestine and the liver
Hepatic portal vein
fibrinogen
arteries
heart
4. Essentially 0 mmHg - which results b/c of branching of vessels dissipating pressure to overcome resistance
Coronary veins
Spleen and liver
venous blood pressure
hemostasis
5. Occurs when increased cardiac output is needed; the postganglionic nerve directly innervates the heart - releasing norepinephrine - increasing heart rate and force of contraction
varicose veins
increased hydrostatic pressure in the capillaries - which increases the fluid that leaks out of the capillaries into the interstitum
veins
Sympathetic regulation of heart
6. The principle sugar in blood that maintains a relatively constant concentration for adequate nutrition
1. increase total blood volume by retaining more H2O 2. Contraction of large veins - propelling blood toward the heart
Glucose
pulmonary circulation
Diastole
7. When the valve of a vein fails and back flow occurs; blood not being moved toward the heart
AB+ since no antibodies are made to any blood type
varicose veins
valves
Secondary transportation of CO2 in the blood
8. Protein that maintains oncotic pressure in capillaries
Glucose
venous blood pressure
albumin
bicuspid (mitral) valve
9. Because the veins have essentially 0 pressure - these valves ensure one - way flow - skeletal muscle contraction encourages flow through veins
2 components of antigens
Valves of the venous system
Sickle cell anemia
cardiac output (L/min)
10. Filling of the ventricles by squeezing of the atria - marks the beginning of the 'dub' sound
Sympathetic regulation of heart
Functional syncytium
Diastole
Lipoproteins
11. Transportation of blood though the body and exchange of material btw blood and tissues
Thrombus
Fxn of circulatory system
Peripheral resistance
Diastole is longer
12. Number of systole contractions per unit time
Peripheral resistance
fibrin
heart rate
Tense
13. Key proteins for the function of the immune system that are produced and released by B- cells
Immunoglobulins (antibodies)
nutrients - wastes - and WBC
varicose veins
Arterial pressure=ventricular pressure
14. Which is longer - diastole or systole?
Ischemia
Peripheral resistance
Na leak channels
Diastole is longer
15. Confirmation of hemoglobin with O2 bound - where affinity is high 1. pH 2. pCO2 3.
Relaxed
resistance
diastolic blood pressure
bone marrow
16. Receives deoxygenated blood from systemic circulation (superior and inferior vena cava)
Right atrium
Spleen and liver
Fxn of circulatory system
bicuspid (mitral) valve
17. 3 substances that can diffuse through intercellular cleft
macrophage
Right atrium
albumin
nutrients - wastes - and WBC
18. Flow of blood from the heart to the lungs - pumped by the right side of the heart
Slow Ca channels
pulmonary circulation
albumin
AV node
19. Amount of blood pumped w/ each systolic contraction
capillaries
Coronary arteries
stroke volume
cardiac output (L/min)
20. Control of by ANS of rate of contraction through the Vagus nerve. Postganglionic release in SA node of ACH inhibits depolarization
It is the same - otherwise it would lead to fluid backup
Vagal Signal
5 phases of cardiac muscle cell contraction
tricuspid valve
21. When do semilunar valves close?
Arterial pressure=ventricular pressure
Glucose
arteries
ABO blood group
22. 1. depolarization caused by fast Na channels - where action potential through intercalated discs reaches threshold potential - opening Na channels 2. initial depolarization with Na channels closing and k channels opening - but Ca channels also open 3
local autoregulation
Frank - Starling Effect
5 phases of cardiac muscle cell contraction
Hemoglobin
23. Caused by closure of Ca channels and opening of K channels
Intercalated discs
adipocytes
fibrin
Repolarization of nodes
24. Flow from the heart to the rest of the body; pumped by the left side of the heart
resistance
Immunoglobulins (antibodies)
bicuspid (mitral) valve
systemic circulation
25. Resting membrane potential of -90mV and have long duration action potentials
High since the concentration of plasma proteins has increased due to movement of water
Cardiac muscle cells
Erythropoetin
pulmonary circulation
26. Neural sympathetic input by postganglionic neuron of norepinephrine innervating arterial smooth muscle
venous return
adrenergic tone
heart rate
Coronary veins
27. Pool of deoxygenated blood at low pressure - which collects blood from coronary veins - Only deoxygenated blood to not enter the right atrium via the vena cava
1. increase total blood volume by retaining more H2O 2. Contraction of large veins - propelling blood toward the heart
It is the same - otherwise it would lead to fluid backup
coronary sinus
macrophage
28. Universal acceptor
It has the most Na leak channels - allowing to reach threshold potential first; all other nodes leak - but rate at as quick of a rate
AB+ since no antibodies are made to any blood type
Thrombus
Erythrocytes
29. ABO blood group and Rh blood group
Secondary transportation of CO2 in the blood
Frank - Starling Effect
Third transportation of CO2 in the blood
2 components of antigens
30. Vessels that carry blood back to the heart at low pressure
veins
Granulocytes
Hemoglobin
megakaryocytes
31. Stretching to greater degree of heart muscle causes more forceful contraction; stretching increase occur by increasing fluid volume
Frank - Starling Effect
Peripheral resistance
adipocytes
atria
32. Osmotic pressure in capillaries due to plasma proteins
Blood plasma
oncotic pressure
venous blood pressure
Third transportation of CO2 in the blood
33. Ensure the one - way flow through the circulatory system
ventricles
fats
valves
Spleen and liver
34. Contraction of the ventricles - where pressure increases rapidly - causing AV valves to close - Marks the beginning of the 'lub' sound
Systole
Rh blood group
fats
valves
35. Glycoproteins that are coded for by 3 alleles (A - B - i)
CNS decreases vagal signal and sympathetic input increases
Thrombus
ABO blood group
cardiac output (L/min)
36. 2 lymphocytes
venous blood pressure
CNS decreases vagal signal and sympathetic input increases
diastolic blood pressure
B cells and T cells
37. As low as pressure gets btw heart beats in arteries
diastolic blood pressure
Primary transportation fo CO2 in the blood
valves
Hepatic portal vein
38. Response by CNS when blood pressure is too high
hemophilia
bicuspid (mitral) valve
It has the most Na leak channels - allowing to reach threshold potential first; all other nodes leak - but rate at as quick of a rate
increase vagal signal and inhibits sympathetic input
39. Excessive bleeding that results from defective proteins
hemophilia
varicose veins
Inflammation
hemostasis
40. 73% of CO2 converted to carbonic acid by carbonic anhydrase - and carbonic acid is converted to bicarbonate - which acts a buffer
high osmolarity of tissues
High since the concentration of plasma proteins has increased due to movement of water
Inflammation
Primary transportation fo CO2 in the blood
41. Request by tissues to increase blood flow - where build up of metabolic waste causes arterioles to dialate
local autoregulation
Repolarization of nodes
Third transportation of CO2 in the blood
AV node
42. Protein in RBC that transport O2 though the blood since O2 is too hydrophobic in plasma; protein has 4 subunits that change confirmation cooperatively depending on the concentration of O2
AB+ since no antibodies are made to any blood type
systolic blood pressure
Hemoglobin
Na leak channels
43. Path where impulse travels from SA to AV node
T- tubules
increase vagal signal and inhibits sympathetic input
Internodal tract
atria
44. Absorbed in the intestine and packaged in chylomicrons - which enter the lymphatic system - and dumped into the subclavian vein via the thoracic duct; the liver takes fats once in blood - converts them to another lipoprotein and sends them to adipocy
Functional syncytium
fats
glycolysis. RBC have no ETC - FA oxidation - or TCA cycle
Fxn of circulatory system
45. Open when threshold is reached causing membrane potential to increase/depolarize; operate slower than Na channels
amino acids and glucose
Ca channels
Secondary transportation of CO2 in the blood
diastolic blood pressure
46. 3 factors that dictate the affinity of hemoglobin for O2
bone marrow
Temperature or metabolic rate
Erythrocytes
tricuspid valve
47. Purpose of erythrocytes?
Rh blood group
bilirubin
to transport O2 to tissues and CO2 to the lungs
Lipoproteins
48. Connected to SA node via internodal tract - and passes signal to Common bundle of His to contract ventricles
AV node
capillaries
pulse pressure
atria
49. Reservoirs where blood collects from veins
atria
Granulocytes
Rh blood group
urea
50. Buffer in blood. Keeps pH around 7.4
fibrin
primary bicarbonate generated from CO2.
fats
albumin b/c it provides the bulk of oncotic pressure in blood vessels - preventing edema
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