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Test your basic knowledge |
MCAT Biology Circulatory System
Start Test
Study First
Subjects
:
mcat
,
health-sciences
Instructions:
Answer 50 questions in 15 minutes.
If you are not ready to take this test, you can
study here
.
Match each statement with the correct term.
Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.
This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. Opposing friction force to flow - which increases with decreased radius; determined by degree of contraction of arterial smooth muscle
nutrients
resistance
albumin b/c it provides the bulk of oncotic pressure in blood vessels - preventing edema
Repolarization of nodes
2. Heart rate *stroke volume= (units)
chylomicrons
tricuspid valve
Valves of the venous system
cardiac output (L/min)
3. Open when threshold is reached causing membrane potential to increase/depolarize; operate slower than Na channels
Ca channels
bilirubin
chylomicrons
Inflammation
4. Response by CNS when blood pressure is too low
B cells and T cells
Valves of the venous system
CNS decreases vagal signal and sympathetic input increases
hepatic portal system and hypothalamic - hypophosial portal system
5. Monocyte that phagocytoses debris and microorganisms - has amoeboid motility - and displays chemotaxis
macrophage
Ischemia
nutrients - wastes - and WBC
Systole
6. Caused by closure of Ca channels and opening of K channels
Perfusion
2 components of antigens
Repolarization of nodes
Right atrium
7. At position 6 - missense mutation substitutes valine for glutamate. valine is hydrophobic - where glutamate was charged. It is an autosomal recessive disease where RBCs accumulated in small vessels - heterozygote for (blank) shows resistance to malar
Platelet fxn
bicuspid (mitral) valve
Sickle cell anemia
Thrombus
8. Response by CNS when blood pressure is too high
increase vagal signal and inhibits sympathetic input
nutrients
Fxn of circulatory system
Temperature or metabolic rate
9. 1. depolarization caused by fast Na channels - where action potential through intercalated discs reaches threshold potential - opening Na channels 2. initial depolarization with Na channels closing and k channels opening - but Ca channels also open 3
5 phases of cardiac muscle cell contraction
Functional syncytium
resistance
urea
10. Large particles consisting of fats - cholesterol - and carrier proteins; transport lipids through the blood stream
nutrients - wastes - and WBC
Hemolytic disease of a newborn
Lipoproteins
Repolarization of nodes
11. ABO blood group and Rh blood group
CNS decreases vagal signal and sympathetic input increases
2 components of antigens
heart
oncotic pressure
12. Produced during cell metabolism and diffuses through the endothelial cells into the blood stream - where it is picked up by the liver and converted to forms that can be excreted (all other wastes are picked up by the kidneys)
Slow Ca channels
Glucose
Waste
It has the most Na leak channels - allowing to reach threshold potential first; all other nodes leak - but rate at as quick of a rate
13. Receptors in the carotid arteries and aortic arch that notify CNS if blood pressure is high or low
ventricles
Baroreceptors
stroke volume
local autoregulation
14. Have single layer endothelial cells w/ spaces in between cells called intercellular cleft
Slow Ca channels
Capillaries
atria and ventricles
local autoregulation
15. Highest blood pressure that occurs during ventricular contraction
Diastole is longer
systolic blood pressure
ventricles
Waste
16. Bone marrow cells that give rise to RBC and platelets
Lipoproteins
megakaryocytes
WBC
Arterial pressure=ventricular pressure
17. Stretching to greater degree of heart muscle causes more forceful contraction; stretching increase occur by increasing fluid volume
Secondary transportation of CO2 in the blood
Frank - Starling Effect
when person that is Rh - is exposed to blood that is Rh+
Diastole
18. Protein in RBC that transport O2 though the blood since O2 is too hydrophobic in plasma; protein has 4 subunits that change confirmation cooperatively depending on the concentration of O2
Hemoglobin
B cells and T cells
SA node
arteries
19. Why is the SA node the primary pacemaker?
CNS decreases vagal signal and sympathetic input increases
It has the most Na leak channels - allowing to reach threshold potential first; all other nodes leak - but rate at as quick of a rate
heart
fibrin
20. Valves between the ventricle and the atria to prevent back flow
atrioventricular valves
Diastole is longer
venous return
capillaries
21. Where do all components of the blood develop from?
systolic blood pressure
bone marrow
primary bicarbonate generated from CO2.
systemic arterial blood pressure
22. Occurs when increased cardiac output is needed; the postganglionic nerve directly innervates the heart - releasing norepinephrine - increasing heart rate and force of contraction
glycolysis. RBC have no ETC - FA oxidation - or TCA cycle
Slow Ca channels
adipocytes
Sympathetic regulation of heart
23. Flow of blood from the heart to the lungs - pumped by the right side of the heart
pulmonary circulation
Hemolytic disease of a newborn
primary bicarbonate generated from CO2.
Rh blood group
24. Universal donor
1. increase total blood volume by retaining more H2O 2. Contraction of large veins - propelling blood toward the heart
Fast Na channels
bicuspid (mitral) valve
O- since there are no surface antigens for antibodies to bind to...
25. Filling of the ventricles by squeezing of the atria - marks the beginning of the 'dub' sound
Lipoproteins
Valves of the venous system
serum
Diastole
26. Gap junctions in the cardiac muscle - where depolarization is communicated directly btw cytoplasm of neighboring cardiac cells
Thrombus
Coronary arteries
Intercalated discs
Capillaries
27. Destroy parasites and are involved in allergic rxns
Fxn of circulatory system
eosinophil
Vagal Signal
Erythrocytes
28. CO2 is soluble in H2O - and thus some is dissolved and carried to lungs and tissues in plasma - O2 is not soluble in plasma at all
Capillaries
hemostasis
Ischemia
Third transportation of CO2 in the blood
29. Active form of fibrinogen - protein forms a mesh that holds platelet plug together to protect wound - ibrinogen is converted to (blank) by thrombin
fibrin
Cardiac muscle cells
systemic arterial blood pressure
albumin b/c it provides the bulk of oncotic pressure in blood vessels - preventing edema
30. Where blood passes through 2 sets of capillaries before returning to the heart; Evolved as direct transport routes
diastolic blood pressure
Portal systems
eosinophil
serum
31. Confirmation of hemoglobin with O2 bound - where affinity is high 1. pH 2. pCO2 3.
CNS decreases vagal signal and sympathetic input increases
Relaxed
Na leak channels
Vagal Signal
32. Control of by ANS of rate of contraction through the Vagus nerve. Postganglionic release in SA node of ACH inhibits depolarization
Vagal Signal
hepatic portal system and hypothalamic - hypophosial portal system
Ischemia
megakaryocytes
33. Precursor to fibrin - which is necessary for blood clotting
systemic circulation
AV node
Ca channels
fibrinogen
34. Universal acceptor
Capillaries
Perfusion
oncotic pressure
AB+ since no antibodies are made to any blood type
35. Inadequate blood flow - resulting in tissue damage due to shortage of O2 and nutrients - and increase of metabolic waste
SA node
pulse pressure
Ischemia
Thrombus
36. Maximize entry of Ca into the cell by allowing entry of Ca extracellular environment; leads to contraction of actin - myosin fibers
WBC
T- tubules
macrophage
Inflammation
37. What is the only process RBC use to generate ATP?
tricuspid valve
glycolysis. RBC have no ETC - FA oxidation - or TCA cycle
Spleen and liver
B cells and T cells
38. Osmotic pressure in capillaries due to plasma proteins
oncotic pressure
chylomicrons
basophil
bicuspid (mitral) valve
39. Flow of blood through a tissue
arteries
Perfusion
eosinophil
adipocytes
40. Purpose of erythrocytes?
basophil
to transport O2 to tissues and CO2 to the lungs
adrenergic tone
5 phases of cardiac muscle cell contraction
41. Force per unit area exerted by blood on walls of arteries
hemophilia
Frank - Starling Effect
It has the most Na leak channels - allowing to reach threshold potential first; all other nodes leak - but rate at as quick of a rate
systemic arterial blood pressure
42. Absorbed in the intestine and packaged in chylomicrons - which enter the lymphatic system - and dumped into the subclavian vein via the thoracic duct; the liver takes fats once in blood - converts them to another lipoprotein and sends them to adipocy
High since the concentration of plasma proteins has increased due to movement of water
1. increase total blood volume by retaining more H2O 2. Contraction of large veins - propelling blood toward the heart
fats
glycolysis. RBC have no ETC - FA oxidation - or TCA cycle
43. 2 ways to increase venous return
1. increase total blood volume by retaining more H2O 2. Contraction of large veins - propelling blood toward the heart
High since the concentration of plasma proteins has increased due to movement of water
Functional syncytium
Diastole
44. Vessels that carry blood back to the heart at low pressure
High since the concentration of plasma proteins has increased due to movement of water
tricuspid valve
veins
Systole
45. Crosses septum and connects to Purkinje fibers to allow coordinated contraction of ventricles. Key is that is slows transmission across septum to allow ventricles to fully fill before contraction
Internodal tract
nutrients - wastes - and WBC
Bundle of His
urea
46. Rh factor that follows dominant pattern (Rh+ in heterozygote)
Diastole is longer
Rh blood group
high osmolarity of tissues
venous blood pressure
47. Fat storage cells of the body
increase vagal signal and inhibits sympathetic input
Fxn of circulatory system
fats
adipocytes
48. Glycoproteins that are coded for by 3 alleles (A - B - i)
fibrinogen
ABO blood group
Erythrocytes
Sickle cell anemia
49. Return of blood to the heart by the vena cava - where increased venous return causes increased stretching of the muscle (increases stroke volume)
venous return
Spleen and liver
It is the same - otherwise it would lead to fluid backup
Granulocytes
50. When do semilunar valves close?
Systole
Arterial pressure=ventricular pressure
when person that is Rh - is exposed to blood that is Rh+
Granulocytes