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Test your basic knowledge |
MCAT Biology Circulatory System
Start Test
Study First
Subjects
:
mcat
,
health-sciences
Instructions:
Answer 50 questions in 15 minutes.
If you are not ready to take this test, you can
study here
.
Match each statement with the correct term.
Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.
This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. 73% of CO2 converted to carbonic acid by carbonic anhydrase - and carbonic acid is converted to bicarbonate - which acts a buffer
Primary transportation fo CO2 in the blood
nutrients
megakaryocytes
Inflammation
2. Essentially 0 mmHg - which results b/c of branching of vessels dissipating pressure to overcome resistance
Frank - Starling Effect
when person that is Rh - is exposed to blood that is Rh+
venous blood pressure
Baroreceptors
3. Where are RBCs broken down?
ventricles
atria
Spleen and liver
Blood plasma
4. Occurs when increased cardiac output is needed; the postganglionic nerve directly innervates the heart - releasing norepinephrine - increasing heart rate and force of contraction
SA node
varicose veins
Sympathetic regulation of heart
CNS decreases vagal signal and sympathetic input increases
5. Inadequate blood flow - resulting in tissue damage due to shortage of O2 and nutrients - and increase of metabolic waste
stroke volume
Ischemia
Sickle cell anemia
Fxn of circulatory system
6. Adequate circulation - but O2 supply is reduced (no build up waste products or loss of nutrients)
B cells and T cells
bilirubin
hypoxia
CNS decreases vagal signal and sympathetic input increases
7. Open when threshold is reached causing membrane potential to increase/depolarize; operate slower than Na channels
Ca channels
Pulmonary and aortic semilunar valves
diastolic blood pressure
glycolysis. RBC have no ETC - FA oxidation - or TCA cycle
8. Flow of blood through a tissue
Waste
Perfusion
Immunoglobulins (antibodies)
B cells and T cells
9. Difference in pressure=blood flow (L/min)*resitance ^P=Q*R
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10. Receives deoxygenated blood from systemic circulation (superior and inferior vena cava)
chylomicrons
tricuspid valve
Right atrium
Cardiac muscle cells
11. Transportation of blood though the body and exchange of material btw blood and tissues
atrioventricular valves
Fxn of circulatory system
Immunoglobulins (antibodies)
Third transportation of CO2 in the blood
12. Produced during cell metabolism and diffuses through the endothelial cells into the blood stream - where it is picked up by the liver and converted to forms that can be excreted (all other wastes are picked up by the kidneys)
Hepatic portal vein
Waste
adipocytes
varicose veins
13. What is the only process RBC use to generate ATP?
ABO blood group
glycolysis. RBC have no ETC - FA oxidation - or TCA cycle
adrenergic tone
It has the most Na leak channels - allowing to reach threshold potential first; all other nodes leak - but rate at as quick of a rate
14. Confirmation of hemoglobin with no O2 bound - so it has low affinity
Tense
Temperature or metabolic rate
Bundle of His
albumin
15. 2 ways to increase venous return
1. increase total blood volume by retaining more H2O 2. Contraction of large veins - propelling blood toward the heart
pulmonary circulation
Arterial pressure=ventricular pressure
Slow Ca channels
16. Capillaries dilate - increasing the cleft size - which allows more H2O to move through to tissues
Blood plasma
Secondary transportation of CO2 in the blood
Tense
Inflammation
17. Osmotic pressure in capillaries due to plasma proteins
eosinophil
Slow Ca channels
Baroreceptors
oncotic pressure
18. Active form of fibrinogen - protein forms a mesh that holds platelet plug together to protect wound - ibrinogen is converted to (blank) by thrombin
Ischemia
Lipoproteins
fibrin
Vagal Signal
19. ABO blood group and Rh blood group
2 components of antigens
systolic blood pressure
high osmolarity of tissues
Immunoglobulins (antibodies)
20. At position 6 - missense mutation substitutes valine for glutamate. valine is hydrophobic - where glutamate was charged. It is an autosomal recessive disease where RBCs accumulated in small vessels - heterozygote for (blank) shows resistance to malar
Sickle cell anemia
WBC
It is the same - otherwise it would lead to fluid backup
It has the most Na leak channels - allowing to reach threshold potential first; all other nodes leak - but rate at as quick of a rate
21. Highest blood pressure that occurs during ventricular contraction
macrophage
adipocytes
systolic blood pressure
Erythropoetin
22. Neural sympathetic input by postganglionic neuron of norepinephrine innervating arterial smooth muscle
adrenergic tone
Intercalated discs
increase vagal signal and inhibits sympathetic input
nutrients - wastes - and WBC
23. Blood clot or scab circulating in bloodstream
Thrombus
Functional syncytium
Granulocytes
Erythropoetin
24. Crosses septum and connects to Purkinje fibers to allow coordinated contraction of ventricles. Key is that is slows transmission across septum to allow ventricles to fully fill before contraction
Bundle of His
Erythropoetin
neutrophil
heart
25. Body's mechanism of preventing bleeding
hemostasis
eosinophil
adrenergic tone
Valves of the venous system
26. Precursor to fibrin - which is necessary for blood clotting
Repolarization of nodes
fibrinogen
Third transportation of CO2 in the blood
Intercalated discs
27. Pump blood out of the heart at high pressures into arteries
Lipoproteins
ventricles
chylomicrons
to transport O2 to tissues and CO2 to the lungs
28. Large particles consisting of fats - cholesterol - and carrier proteins; transport lipids through the blood stream
veins
Lipoproteins
fibrinogen
AV node
29. Connects the two capillary beds of the intestine and the liver
atrioventricular valves
Third transportation of CO2 in the blood
Hepatic portal vein
Valves of the venous system
30. Return of blood to the heart by the vena cava - where increased venous return causes increased stretching of the muscle (increases stroke volume)
Ohm's law
Sickle cell anemia
1. increase total blood volume by retaining more H2O 2. Contraction of large veins - propelling blood toward the heart
venous return
31. Fat storage cells of the body
hypoxia
adipocytes
atria
Diastole
32. Confirmation of hemoglobin with O2 bound - where affinity is high 1. pH 2. pCO2 3.
Fast Na channels
Ohm's law
Relaxed
glycolysis. RBC have no ETC - FA oxidation - or TCA cycle
33. Heart rate *stroke volume= (units)
bilirubin
Right atrium
cardiac output (L/min)
venous return
34. Aggregate at site of damage to a blood vessel and form a platelet plug to stop bleeding
nutrients
Platelet fxn
adipocytes
atria and ventricles
35. Region that initiates start of cardiac cycle - which acts as a pacemaker of the heart; has unstable resting potential due to Na leak channels
hemostasis
Hemoglobin
SA node
albumin
36. CO2 is soluble in H2O - and thus some is dissolved and carried to lungs and tissues in plasma - O2 is not soluble in plasma at all
Third transportation of CO2 in the blood
ABO blood group
SA node
veins
37. 3 substances that can diffuse through intercellular cleft
nutrients - wastes - and WBC
systemic arterial blood pressure
2 components of antigens
Relaxed
38. Resting membrane potential of -90mV and have long duration action potentials
Cardiac muscle cells
hemophilia
stroke volume
Internodal tract
39. Why is the SA node the primary pacemaker?
AB+ since no antibodies are made to any blood type
It has the most Na leak channels - allowing to reach threshold potential first; all other nodes leak - but rate at as quick of a rate
1. increase total blood volume by retaining more H2O 2. Contraction of large veins - propelling blood toward the heart
valves
40. 2 lymphocytes
Coronary arteries
Sickle cell anemia
Ohm's law
B cells and T cells
41. 2 chambers of the heart
atria and ventricles
Arterial pressure=ventricular pressure
Sickle cell anemia
chylomicrons
42. 55% of whole blood that is composed of electrolytes - lipoproteins - sugars - buffer - and metabolic waste
Functional syncytium
AV node
Blood plasma
Hepatic portal vein
43. Lipoproteins that enter lacteal vessels of lymphatic system in the intestinal wall
CNS decreases vagal signal and sympathetic input increases
Cardiac muscle cells
Right atrium
chylomicrons
44. Force per unit area exerted by blood on walls of arteries
Baroreceptors
systemic arterial blood pressure
Peripheral resistance
Fxn of circulatory system
45. What causes tendency of water flow out of blood?
Slow Ca channels
Diastole is longer
high osmolarity of tissues
cardiac output (L/min)
46. Response by CNS when blood pressure is too high
nutrients
megakaryocytes
increase vagal signal and inhibits sympathetic input
albumin
47. Store and release histamine and are involved in allergic rxns
Thrombus
stroke volume
basophil
Ohm's law
48. Destroy parasites and are involved in allergic rxns
AV node
eosinophil
arteries
Spleen and liver
49. Vessels where deoxygenated blood from coronary sinus continue to flow into heart
Coronary veins
Peripheral resistance
systemic circulation
T- tubules
50. Muscular pump that forces blood through series of branching vessels
Arterial pressure=ventricular pressure
heart
Rh blood group
increase vagal signal and inhibits sympathetic input