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Test your basic knowledge |
MCAT Biology Circulatory System
Start Test
Study First
Subjects
:
mcat
,
health-sciences
Instructions:
Answer 50 questions in 15 minutes.
If you are not ready to take this test, you can
study here
.
Match each statement with the correct term.
Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.
This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. Flow from the heart to the rest of the body; pumped by the left side of the heart
systemic circulation
hepatic portal system and hypothalamic - hypophosial portal system
nutrients - wastes - and WBC
Perfusion
2. CO2 is soluble in H2O - and thus some is dissolved and carried to lungs and tissues in plasma - O2 is not soluble in plasma at all
Third transportation of CO2 in the blood
Waste
Granulocytes
high osmolarity of tissues
3. Body's mechanism of preventing bleeding
Primary transportation fo CO2 in the blood
albumin
hemostasis
Hemolytic disease of a newborn
4. The difference in pressure divided blood flow; controlled by the sympathetic nervous system generating adrenergic tone
Peripheral resistance
adipocytes
Pulmonary and aortic semilunar valves
fibrinogen
5. Peptide hormone secreted from the kidneys to increase RBC production in bone marrow
Diastole
Ca channels
local autoregulation
Erythropoetin
6. As low as pressure gets btw heart beats in arteries
diastolic blood pressure
oncotic pressure
Tense
Erythropoetin
7. Monocyte that phagocytoses debris and microorganisms - has amoeboid motility - and displays chemotaxis
macrophage
It is the same - otherwise it would lead to fluid backup
1. increase total blood volume by retaining more H2O 2. Contraction of large veins - propelling blood toward the heart
B cells and T cells
8. Purpose of erythrocytes?
macrophage
increase vagal signal and inhibits sympathetic input
to transport O2 to tissues and CO2 to the lungs
fibrinogen
9. Voltage - gated channels that open quickly; open at threshold potential
Fast Na channels
Coronary arteries
systemic circulation
adrenergic tone
10. Connected to SA node via internodal tract - and passes signal to Common bundle of His to contract ventricles
bone marrow
Cardiac muscle cells
Blood plasma
AV node
11. Where do all components of the blood develop from?
It is the same - otherwise it would lead to fluid backup
heart
valves
bone marrow
12. Aggregate at site of damage to a blood vessel and form a platelet plug to stop bleeding
Tense
It is the same - otherwise it would lead to fluid backup
Na leak channels
Platelet fxn
13. Control of by ANS of rate of contraction through the Vagus nerve. Postganglionic release in SA node of ACH inhibits depolarization
atrioventricular valves
Erythrocytes
Vagal Signal
High since the concentration of plasma proteins has increased due to movement of water
14. Number of systole contractions per unit time
heart rate
albumin
serum
pulse pressure
15. Excessive bleeding that results from defective proteins
neutrophil
hemophilia
Coronary veins
Glucose
16. Universal donor
nutrients
O- since there are no surface antigens for antibodies to bind to...
Cardiac muscle cells
increase vagal signal and inhibits sympathetic input
17. Ensure the one - way flow through the circulatory system
Portal systems
valves
varicose veins
Erythrocytes
18. Connects the two capillary beds of the intestine and the liver
Hepatic portal vein
venous return
bone marrow
Glucose
19. What is the direct cause of edema?
hypoxia
Primary transportation fo CO2 in the blood
increased hydrostatic pressure in the capillaries - which increases the fluid that leaks out of the capillaries into the interstitum
2 components of antigens
20. Amount of blood pumped w/ each systolic contraction
Coronary veins
venous return
Granulocytes
stroke volume
21. Voltage - gated channels that stay open longer than Na channels and open later responsible for the plateau phase of cardiac muscle contraction
Secondary transportation of CO2 in the blood
capillaries
local autoregulation
Slow Ca channels
22. Stretching to greater degree of heart muscle causes more forceful contraction; stretching increase occur by increasing fluid volume
veins
capillaries
Frank - Starling Effect
high osmolarity of tissues
23. Metabolic waste product in breakdown of amino acids
urea
Spleen and liver
Intercalated discs
varicose veins
24. Flow of blood from the heart to the lungs - pumped by the right side of the heart
when person that is Rh - is exposed to blood that is Rh+
Hemolytic disease of a newborn
Peripheral resistance
pulmonary circulation
25. Why is the SA node the primary pacemaker?
chylomicrons
Arterial pressure=ventricular pressure
Relaxed
It has the most Na leak channels - allowing to reach threshold potential first; all other nodes leak - but rate at as quick of a rate
26. Have single layer endothelial cells w/ spaces in between cells called intercellular cleft
albumin b/c it provides the bulk of oncotic pressure in blood vessels - preventing edema
heart rate
Capillaries
basophil
27. Vessels that carry blood back to the heart at low pressure
diastolic blood pressure
Inflammation
Fast Na channels
veins
28. Receives deoxygenated blood from systemic circulation (superior and inferior vena cava)
pulse pressure
increased hydrostatic pressure in the capillaries - which increases the fluid that leaks out of the capillaries into the interstitum
Lipoproteins
Right atrium
29. Filling of the ventricles by squeezing of the atria - marks the beginning of the 'dub' sound
amino acids and glucose
Ischemia
Diastole
Baroreceptors
30. Difference in pressure=blood flow (L/min)*resitance ^P=Q*R
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31. First branches from the aorta that provide the heart's blood supply
basophil
Repolarization of nodes
Coronary arteries
Rh blood group
32. Neutrophil - eosinophil - and basophil
Frank - Starling Effect
Systole
Granulocytes
megakaryocytes
33. Hematocrit or RBC those compose 35-45% of the blood; cells are non - nucleated and have no organelles. Acquire ATP through glycolysis have biconcave shape to maximize surface area for binding O2
arteries
Erythrocytes
systolic blood pressure
Granulocytes
34. Large particles consisting of fats - cholesterol - and carrier proteins; transport lipids through the blood stream
local autoregulation
Lipoproteins
macrophage
atria
35. 20% transported stuck to hemoglobin; why increased pCO2 decreases affinity of O2
atria and ventricles
Peripheral resistance
Secondary transportation of CO2 in the blood
neutrophil
36. Opposing friction force to flow - which increases with decreased radius; determined by degree of contraction of arterial smooth muscle
resistance
Diastole is longer
local autoregulation
macrophage
37. Occurs when increased cardiac output is needed; the postganglionic nerve directly innervates the heart - releasing norepinephrine - increasing heart rate and force of contraction
Hepatic portal vein
Valves of the venous system
Sympathetic regulation of heart
coronary sinus
38. At position 6 - missense mutation substitutes valine for glutamate. valine is hydrophobic - where glutamate was charged. It is an autosomal recessive disease where RBCs accumulated in small vessels - heterozygote for (blank) shows resistance to malar
Internodal tract
ventricles
cardiac output (L/min)
Sickle cell anemia
39. Blood clot or scab circulating in bloodstream
Slow Ca channels
atrioventricular valves
Thrombus
capillaries
40. Absorbed by the GI tract and brought to the liver via the hepatic portal vein - where they are stored in the liver and enter the blood stream when needed
amino acids and glucose
High since the concentration of plasma proteins has increased due to movement of water
chylomicrons
hemophilia
41. Crosses septum and connects to Purkinje fibers to allow coordinated contraction of ventricles. Key is that is slows transmission across septum to allow ventricles to fully fill before contraction
bilirubin
ventricles
Bundle of His
5 phases of cardiac muscle cell contraction
42. Inadequate blood flow - resulting in tissue damage due to shortage of O2 and nutrients - and increase of metabolic waste
Sympathetic regulation of heart
Ischemia
Repolarization of nodes
Ohm's law
43. When do Rh antibodies develop?
when person that is Rh - is exposed to blood that is Rh+
Peripheral resistance
Diastole
Ca channels
44. Gap junctions in the cardiac muscle - where depolarization is communicated directly btw cytoplasm of neighboring cardiac cells
Erythrocytes
stroke volume
Internodal tract
Intercalated discs
45. Heart rate *stroke volume= (units)
cardiac output (L/min)
Arterial pressure=ventricular pressure
Fxn of circulatory system
increased hydrostatic pressure in the capillaries - which increases the fluid that leaks out of the capillaries into the interstitum
46. Response by CNS when blood pressure is too high
adipocytes
adrenergic tone
increase vagal signal and inhibits sympathetic input
bicuspid (mitral) valve
47. Mother has Rh - blood with Rh+ antibodies that attack the babies Rh+ blood
Hemolytic disease of a newborn
Erythrocytes
fibrinogen
megakaryocytes
48. Caused by closure of Ca channels and opening of K channels
hepatic portal system and hypothalamic - hypophosial portal system
oncotic pressure
Valves of the venous system
Repolarization of nodes
49. Return of blood to the heart by the vena cava - where increased venous return causes increased stretching of the muscle (increases stroke volume)
Capillaries
WBC
venous return
Erythrocytes
50. Protein that maintains oncotic pressure in capillaries
albumin
Fxn of circulatory system
Slow Ca channels
fats