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Test your basic knowledge |
MCAT Biology Circulatory System
Start Test
Study First
Subjects
:
mcat
,
health-sciences
Instructions:
Answer 50 questions in 15 minutes.
If you are not ready to take this test, you can
study here
.
Match each statement with the correct term.
Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.
This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. Active form of fibrinogen - protein forms a mesh that holds platelet plug together to protect wound - ibrinogen is converted to (blank) by thrombin
Capillaries
stroke volume
fibrin
primary bicarbonate generated from CO2.
2. Connected to SA node via internodal tract - and passes signal to Common bundle of His to contract ventricles
AV node
basophil
Spleen and liver
Third transportation of CO2 in the blood
3. Which is longer - diastole or systole?
Erythrocytes
hypoxia
nutrients - wastes - and WBC
Diastole is longer
4. When the valve of a vein fails and back flow occurs; blood not being moved toward the heart
varicose veins
chylomicrons
CNS decreases vagal signal and sympathetic input increases
Primary transportation fo CO2 in the blood
5. Opposing friction force to flow - which increases with decreased radius; determined by degree of contraction of arterial smooth muscle
hemostasis
Sickle cell anemia
resistance
Blood plasma
6. Essentially 0 mmHg - which results b/c of branching of vessels dissipating pressure to overcome resistance
Erythropoetin
ABO blood group
venous blood pressure
It has the most Na leak channels - allowing to reach threshold potential first; all other nodes leak - but rate at as quick of a rate
7. Breakdown product of the hemogloblin heme group
5 phases of cardiac muscle cell contraction
bilirubin
Ischemia
veins
8. Where are RBCs broken down?
hemostasis
Spleen and liver
Slow Ca channels
when person that is Rh - is exposed to blood that is Rh+
9. Ensure the one - way flow through the circulatory system
Erythrocytes
Blood plasma
fibrin
valves
10. Metabolic waste product in breakdown of amino acids
Diastole is longer
Perfusion
urea
arteries
11. Monocyte that phagocytoses debris and microorganisms - has amoeboid motility - and displays chemotaxis
macrophage
Coronary arteries
Spleen and liver
glycolysis. RBC have no ETC - FA oxidation - or TCA cycle
12. Muscular pump that forces blood through series of branching vessels
heart
B cells and T cells
urea
albumin b/c it provides the bulk of oncotic pressure in blood vessels - preventing edema
13. Neutrophil - eosinophil - and basophil
Arterial pressure=ventricular pressure
increase vagal signal and inhibits sympathetic input
WBC
Granulocytes
14. Flow of blood from the heart to the lungs - pumped by the right side of the heart
CNS decreases vagal signal and sympathetic input increases
Erythropoetin
pulmonary circulation
diastolic blood pressure
15. Bone marrow cells that give rise to RBC and platelets
fibrin
megakaryocytes
Diastole
Erythropoetin
16. Blood clot or scab circulating in bloodstream
Intercalated discs
AV node
Thrombus
arteries
17. Gap junctions in the cardiac muscle - where depolarization is communicated directly btw cytoplasm of neighboring cardiac cells
Intercalated discs
Portal systems
O- since there are no surface antigens for antibodies to bind to...
hemophilia
18. Phagocytose bacteria resulting in pus; amoeboid motility and chemotaxis
Erythropoetin
stroke volume
Sympathetic regulation of heart
neutrophil
19. Resting membrane potential of -90mV and have long duration action potentials
Thrombus
Cardiac muscle cells
Glucose
Erythrocytes
20. Why is the SA node the primary pacemaker?
It has the most Na leak channels - allowing to reach threshold potential first; all other nodes leak - but rate at as quick of a rate
Perfusion
ventricles
Inflammation
21. Path where impulse travels from SA to AV node
Na leak channels
Internodal tract
Glucose
Functional syncytium
22. Response by CNS when blood pressure is too low
chylomicrons
arteries
Capillaries
CNS decreases vagal signal and sympathetic input increases
23. Fat storage cells of the body
Coronary veins
T- tubules
adipocytes
atria
24. Caused by closure of Ca channels and opening of K channels
veins
varicose veins
Repolarization of nodes
Bundle of His
25. Occurs when increased cardiac output is needed; the postganglionic nerve directly innervates the heart - releasing norepinephrine - increasing heart rate and force of contraction
ABO blood group
basophil
Sympathetic regulation of heart
Hemolytic disease of a newborn
26. When do Rh antibodies develop?
Rh blood group
bone marrow
resistance
when person that is Rh - is exposed to blood that is Rh+
27. Vessels where deoxygenated blood from coronary sinus continue to flow into heart
increase vagal signal and inhibits sympathetic input
Coronary veins
Secondary transportation of CO2 in the blood
Third transportation of CO2 in the blood
28. Aggregate at site of damage to a blood vessel and form a platelet plug to stop bleeding
atrioventricular valves
Thrombus
Platelet fxn
fats
29. Contraction of the ventricles - where pressure increases rapidly - causing AV valves to close - Marks the beginning of the 'lub' sound
fats
Systole
primary bicarbonate generated from CO2.
hemostasis
30. ABO blood group and Rh blood group
Granulocytes
Rh blood group
2 components of antigens
resistance
31. Mother has Rh - blood with Rh+ antibodies that attack the babies Rh+ blood
oncotic pressure
Vagal Signal
Hemolytic disease of a newborn
pulmonary circulation
32. 3 substances that can diffuse through intercellular cleft
systolic blood pressure
Systole
venous blood pressure
nutrients - wastes - and WBC
33. Region that initiates start of cardiac cycle - which acts as a pacemaker of the heart; has unstable resting potential due to Na leak channels
veins
diastolic blood pressure
SA node
Fast Na channels
34. Purpose of erythrocytes?
resistance
chylomicrons
to transport O2 to tissues and CO2 to the lungs
Fast Na channels
35. Glycoproteins that are coded for by 3 alleles (A - B - i)
neutrophil
ABO blood group
Immunoglobulins (antibodies)
Relaxed
36. 2 ways to increase venous return
1. increase total blood volume by retaining more H2O 2. Contraction of large veins - propelling blood toward the heart
adrenergic tone
hypoxia
AB+ since no antibodies are made to any blood type
37. Produced during cell metabolism and diffuses through the endothelial cells into the blood stream - where it is picked up by the liver and converted to forms that can be excreted (all other wastes are picked up by the kidneys)
albumin b/c it provides the bulk of oncotic pressure in blood vessels - preventing edema
diastolic blood pressure
Waste
5 phases of cardiac muscle cell contraction
38. 1. depolarization caused by fast Na channels - where action potential through intercalated discs reaches threshold potential - opening Na channels 2. initial depolarization with Na channels closing and k channels opening - but Ca channels also open 3
amino acids and glucose
Tense
Cardiac muscle cells
5 phases of cardiac muscle cell contraction
39. 55% of whole blood that is composed of electrolytes - lipoproteins - sugars - buffer - and metabolic waste
Fxn of circulatory system
Thrombus
Blood plasma
increase vagal signal and inhibits sympathetic input
40. Allow Na to leak across membrane - causing cell potential to get closer to threshold potential; allow threshold to be reached for Ca channels to open let Ca into the cell
increased hydrostatic pressure in the capillaries - which increases the fluid that leaks out of the capillaries into the interstitum
Diastole is longer
Na leak channels
5 phases of cardiac muscle cell contraction
41. Receives deoxygenated blood from systemic circulation (superior and inferior vena cava)
Right atrium
increased hydrostatic pressure in the capillaries - which increases the fluid that leaks out of the capillaries into the interstitum
oncotic pressure
pulmonary circulation
42. Valves between the ventricle and the atria to prevent back flow
Valves of the venous system
Hemoglobin
Platelet fxn
atrioventricular valves
43. Precursor to fibrin - which is necessary for blood clotting
fibrinogen
Erythrocytes
Coronary arteries
Capillaries
44. At position 6 - missense mutation substitutes valine for glutamate. valine is hydrophobic - where glutamate was charged. It is an autosomal recessive disease where RBCs accumulated in small vessels - heterozygote for (blank) shows resistance to malar
Baroreceptors
Portal systems
Relaxed
Sickle cell anemia
45. Flow from the heart to the rest of the body; pumped by the left side of the heart
systemic circulation
Right atrium
coronary sinus
albumin b/c it provides the bulk of oncotic pressure in blood vessels - preventing edema
46. At the end of the capillary - is the osmotic pressure high or low?
Perfusion
High since the concentration of plasma proteins has increased due to movement of water
high osmolarity of tissues
valves
47. Stretching to greater degree of heart muscle causes more forceful contraction; stretching increase occur by increasing fluid volume
Diastole is longer
Frank - Starling Effect
Erythropoetin
tricuspid valve
48. 2 portal systems to know
Third transportation of CO2 in the blood
urea
High since the concentration of plasma proteins has increased due to movement of water
hepatic portal system and hypothalamic - hypophosial portal system
49. Destroy parasites and are involved in allergic rxns
eosinophil
bilirubin
Hemolytic disease of a newborn
systemic arterial blood pressure
50. Maximize entry of Ca into the cell by allowing entry of Ca extracellular environment; leads to contraction of actin - myosin fibers
stroke volume
hemophilia
T- tubules
Ohm's law