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Test your basic knowledge |
MCAT Biology Circulatory System
Start Test
Study First
Subjects
:
mcat
,
health-sciences
Instructions:
Answer 50 questions in 15 minutes.
If you are not ready to take this test, you can
study here
.
Match each statement with the correct term.
Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.
This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. Confirmation of hemoglobin with no O2 bound - so it has low affinity
hemophilia
Relaxed
Arterial pressure=ventricular pressure
Tense
2. Path where impulse travels from SA to AV node
fibrinogen
Internodal tract
Fxn of circulatory system
bilirubin
3. Stretching to greater degree of heart muscle causes more forceful contraction; stretching increase occur by increasing fluid volume
pulmonary circulation
Frank - Starling Effect
Capillaries
T- tubules
4. Have single layer endothelial cells w/ spaces in between cells called intercellular cleft
Capillaries
T- tubules
Internodal tract
Portal systems
5. Why is the SA node the primary pacemaker?
It has the most Na leak channels - allowing to reach threshold potential first; all other nodes leak - but rate at as quick of a rate
Systole
Slow Ca channels
atria
6. Rh factor that follows dominant pattern (Rh+ in heterozygote)
Hemolytic disease of a newborn
Baroreceptors
B cells and T cells
Rh blood group
7. Valves between the ventricle and the atria to prevent back flow
atrioventricular valves
increased hydrostatic pressure in the capillaries - which increases the fluid that leaks out of the capillaries into the interstitum
nutrients
Waste
8. Buffer in blood. Keeps pH around 7.4
Diastole is longer
hepatic portal system and hypothalamic - hypophosial portal system
primary bicarbonate generated from CO2.
Spleen and liver
9. Open when threshold is reached causing membrane potential to increase/depolarize; operate slower than Na channels
when person that is Rh - is exposed to blood that is Rh+
nutrients - wastes - and WBC
Ca channels
basophil
10. Crosses septum and connects to Purkinje fibers to allow coordinated contraction of ventricles. Key is that is slows transmission across septum to allow ventricles to fully fill before contraction
primary bicarbonate generated from CO2.
Bundle of His
Arterial pressure=ventricular pressure
Glucose
11. Excessive bleeding that results from defective proteins
Slow Ca channels
hemophilia
macrophage
Tense
12. Pump blood out of the heart at high pressures into arteries
AV node
Peripheral resistance
Vagal Signal
ventricles
13. Osmotic pressure in capillaries due to plasma proteins
Waste
1. increase total blood volume by retaining more H2O 2. Contraction of large veins - propelling blood toward the heart
Relaxed
oncotic pressure
14. 3 factors that dictate the affinity of hemoglobin for O2
ABO blood group
CNS decreases vagal signal and sympathetic input increases
Slow Ca channels
Temperature or metabolic rate
15. 2 ways to increase venous return
1. increase total blood volume by retaining more H2O 2. Contraction of large veins - propelling blood toward the heart
coronary sinus
arteries
stroke volume
16. Neural sympathetic input by postganglionic neuron of norepinephrine innervating arterial smooth muscle
Capillaries
ABO blood group
adrenergic tone
Ischemia
17. Receives deoxygenated blood from systemic circulation (superior and inferior vena cava)
Sympathetic regulation of heart
to transport O2 to tissues and CO2 to the lungs
Hemolytic disease of a newborn
Right atrium
18. Flow from the heart to the rest of the body; pumped by the left side of the heart
Rh blood group
glycolysis. RBC have no ETC - FA oxidation - or TCA cycle
Glucose
systemic circulation
19. Pass through the capillaries in order to patrol the tissue for invading organisms; only macrophages and neutrophils can squeeze through cleft
venous return
WBC
Coronary veins
Sympathetic regulation of heart
20. Universal donor
Fxn of circulatory system
O- since there are no surface antigens for antibodies to bind to...
adipocytes
Spleen and liver
21. Is cardiac output the same or different btw the two ventricles?
Functional syncytium
Diastole
Right atrium
It is the same - otherwise it would lead to fluid backup
22. Where blood passes through 2 sets of capillaries before returning to the heart; Evolved as direct transport routes
diastolic blood pressure
serum
systemic circulation
Portal systems
23. Allow Na to leak across membrane - causing cell potential to get closer to threshold potential; allow threshold to be reached for Ca channels to open let Ca into the cell
Na leak channels
increased hydrostatic pressure in the capillaries - which increases the fluid that leaks out of the capillaries into the interstitum
serum
Blood plasma
24. Flow of blood from the heart to the lungs - pumped by the right side of the heart
valves
Immunoglobulins (antibodies)
Bundle of His
pulmonary circulation
25. At position 6 - missense mutation substitutes valine for glutamate. valine is hydrophobic - where glutamate was charged. It is an autosomal recessive disease where RBCs accumulated in small vessels - heterozygote for (blank) shows resistance to malar
fibrinogen
Sickle cell anemia
ABO blood group
Hepatic portal vein
26. Absorbed in the intestine and packaged in chylomicrons - which enter the lymphatic system - and dumped into the subclavian vein via the thoracic duct; the liver takes fats once in blood - converts them to another lipoprotein and sends them to adipocy
It has the most Na leak channels - allowing to reach threshold potential first; all other nodes leak - but rate at as quick of a rate
fats
nutrients - wastes - and WBC
tricuspid valve
27. When do Rh antibodies develop?
systemic circulation
increase vagal signal and inhibits sympathetic input
when person that is Rh - is exposed to blood that is Rh+
oncotic pressure
28. Universal acceptor
valves
Sickle cell anemia
AB+ since no antibodies are made to any blood type
Rh blood group
29. Confirmation of hemoglobin with O2 bound - where affinity is high 1. pH 2. pCO2 3.
varicose veins
hemostasis
amino acids and glucose
Relaxed
30. Receptors in the carotid arteries and aortic arch that notify CNS if blood pressure is high or low
Baroreceptors
Portal systems
atrioventricular valves
High since the concentration of plasma proteins has increased due to movement of water
31. Monocyte that phagocytoses debris and microorganisms - has amoeboid motility - and displays chemotaxis
Blood plasma
macrophage
hypoxia
fibrin
32. Protein in RBC that transport O2 though the blood since O2 is too hydrophobic in plasma; protein has 4 subunits that change confirmation cooperatively depending on the concentration of O2
Hemoglobin
5 phases of cardiac muscle cell contraction
Arterial pressure=ventricular pressure
venous return
33. Region that initiates start of cardiac cycle - which acts as a pacemaker of the heart; has unstable resting potential due to Na leak channels
SA node
hemophilia
veins
bone marrow
34. Destroy parasites and are involved in allergic rxns
Spleen and liver
eosinophil
Lipoproteins
Baroreceptors
35. Large particles consisting of fats - cholesterol - and carrier proteins; transport lipids through the blood stream
bicuspid (mitral) valve
Lipoproteins
Peripheral resistance
glycolysis. RBC have no ETC - FA oxidation - or TCA cycle
36. Glycoproteins that are coded for by 3 alleles (A - B - i)
valves
ABO blood group
Systole
fibrinogen
37. Neutrophil - eosinophil - and basophil
It is the same - otherwise it would lead to fluid backup
Bundle of His
neutrophil
Granulocytes
38. Where are RBCs broken down?
Spleen and liver
adrenergic tone
cardiac output (L/min)
T- tubules
39. Highest blood pressure that occurs during ventricular contraction
systolic blood pressure
Immunoglobulins (antibodies)
nutrients - wastes - and WBC
Diastole is longer
40. Request by tissues to increase blood flow - where build up of metabolic waste causes arterioles to dialate
Fxn of circulatory system
2 components of antigens
Secondary transportation of CO2 in the blood
local autoregulation
41. Flow of blood through a tissue
heart rate
Lipoproteins
increase vagal signal and inhibits sympathetic input
Perfusion
42. Response by CNS when blood pressure is too low
Hemoglobin
CNS decreases vagal signal and sympathetic input increases
Systole
nutrients
43. Because the veins have essentially 0 pressure - these valves ensure one - way flow - skeletal muscle contraction encourages flow through veins
1. increase total blood volume by retaining more H2O 2. Contraction of large veins - propelling blood toward the heart
Valves of the venous system
bicuspid (mitral) valve
Fast Na channels
44. When do semilunar valves close?
oncotic pressure
Arterial pressure=ventricular pressure
Fast Na channels
glycolysis. RBC have no ETC - FA oxidation - or TCA cycle
45. AV valve between right atrium and right ventricle
Third transportation of CO2 in the blood
Granulocytes
tricuspid valve
atrioventricular valves
46. Protein that maintains oncotic pressure in capillaries
albumin
Fast Na channels
glycolysis. RBC have no ETC - FA oxidation - or TCA cycle
Inflammation
47. Contraction of the ventricles - where pressure increases rapidly - causing AV valves to close - Marks the beginning of the 'lub' sound
nutrients
Systole
Glucose
stroke volume
48. Breakdown product of the hemogloblin heme group
bilirubin
High since the concentration of plasma proteins has increased due to movement of water
Valves of the venous system
Ischemia
49. Key proteins for the function of the immune system that are produced and released by B- cells
Glucose
urea
Immunoglobulins (antibodies)
eosinophil
50. Response by CNS when blood pressure is too high
serum
systemic circulation
Perfusion
increase vagal signal and inhibits sympathetic input