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Test your basic knowledge |
MCAT Biology Circulatory System
Start Test
Study First
Subjects
:
mcat
,
health-sciences
Instructions:
Answer 50 questions in 15 minutes.
If you are not ready to take this test, you can
study here
.
Match each statement with the correct term.
Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.
This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. What is the only process RBC use to generate ATP?
pulse pressure
bicuspid (mitral) valve
hemophilia
glycolysis. RBC have no ETC - FA oxidation - or TCA cycle
2. Essentially 0 mmHg - which results b/c of branching of vessels dissipating pressure to overcome resistance
tricuspid valve
veins
Na leak channels
venous blood pressure
3. Site of exchange btw blood and tissues; smallest vessels that allow one RBC through at a time
Sickle cell anemia
Diastole
capillaries
hypoxia
4. AV valve between left atrium and left ventricle
WBC
bicuspid (mitral) valve
Ischemia
venous return
5. Excessive bleeding that results from defective proteins
Granulocytes
T- tubules
atria
hemophilia
6. Large particles consisting of fats - cholesterol - and carrier proteins; transport lipids through the blood stream
Third transportation of CO2 in the blood
Capillaries
Coronary arteries
Lipoproteins
7. Rh factor that follows dominant pattern (Rh+ in heterozygote)
Diastole is longer
AV node
Rh blood group
Thrombus
8. Stretching to greater degree of heart muscle causes more forceful contraction; stretching increase occur by increasing fluid volume
Sympathetic regulation of heart
Valves of the venous system
Frank - Starling Effect
primary bicarbonate generated from CO2.
9. Produced during cell metabolism and diffuses through the endothelial cells into the blood stream - where it is picked up by the liver and converted to forms that can be excreted (all other wastes are picked up by the kidneys)
Waste
albumin b/c it provides the bulk of oncotic pressure in blood vessels - preventing edema
ABO blood group
hypoxia
10. 20% transported stuck to hemoglobin; why increased pCO2 decreases affinity of O2
WBC
Secondary transportation of CO2 in the blood
Hemoglobin
bilirubin
11. Bone marrow cells that give rise to RBC and platelets
pulse pressure
Waste
megakaryocytes
AV node
12. Have single layer endothelial cells w/ spaces in between cells called intercellular cleft
Capillaries
neutrophil
veins
Sickle cell anemia
13. Store and release histamine and are involved in allergic rxns
T- tubules
basophil
Erythropoetin
hemostasis
14. Neural sympathetic input by postganglionic neuron of norepinephrine innervating arterial smooth muscle
Glucose
Hemolytic disease of a newborn
adrenergic tone
resistance
15. Aggregate at site of damage to a blood vessel and form a platelet plug to stop bleeding
Repolarization of nodes
Temperature or metabolic rate
Platelet fxn
local autoregulation
16. Universal donor
hemostasis
O- since there are no surface antigens for antibodies to bind to...
valves
systolic blood pressure
17. Destroy parasites and are involved in allergic rxns
Sickle cell anemia
eosinophil
fats
diastolic blood pressure
18. First branches from the aorta that provide the heart's blood supply
Waste
systolic blood pressure
Temperature or metabolic rate
Coronary arteries
19. Blood clot or scab circulating in bloodstream
Thrombus
diastolic blood pressure
pulse pressure
Temperature or metabolic rate
20. Gap junctions in the cardiac muscle - where depolarization is communicated directly btw cytoplasm of neighboring cardiac cells
heart
neutrophil
Intercalated discs
atria and ventricles
21. Capillaries dilate - increasing the cleft size - which allows more H2O to move through to tissues
varicose veins
atria and ventricles
Inflammation
Relaxed
22. 2 lymphocytes
Repolarization of nodes
B cells and T cells
Immunoglobulins (antibodies)
T- tubules
23. Connects the two capillary beds of the intestine and the liver
bilirubin
resistance
Repolarization of nodes
Hepatic portal vein
24. Occurs when increased cardiac output is needed; the postganglionic nerve directly innervates the heart - releasing norepinephrine - increasing heart rate and force of contraction
ventricles
Hemolytic disease of a newborn
Sympathetic regulation of heart
increased hydrostatic pressure in the capillaries - which increases the fluid that leaks out of the capillaries into the interstitum
25. AV valve between right atrium and right ventricle
tricuspid valve
chylomicrons
5 phases of cardiac muscle cell contraction
Fast Na channels
26. 55% of whole blood that is composed of electrolytes - lipoproteins - sugars - buffer - and metabolic waste
Hemoglobin
Cardiac muscle cells
Blood plasma
2 components of antigens
27. Fat storage cells of the body
Capillaries
adipocytes
Frank - Starling Effect
5 phases of cardiac muscle cell contraction
28. 1. depolarization caused by fast Na channels - where action potential through intercalated discs reaches threshold potential - opening Na channels 2. initial depolarization with Na channels closing and k channels opening - but Ca channels also open 3
fibrinogen
Primary transportation fo CO2 in the blood
5 phases of cardiac muscle cell contraction
eosinophil
29. Active form of fibrinogen - protein forms a mesh that holds platelet plug together to protect wound - ibrinogen is converted to (blank) by thrombin
CNS decreases vagal signal and sympathetic input increases
Lipoproteins
fibrin
Tense
30. Force per unit area exerted by blood on walls of arteries
Lipoproteins
Secondary transportation of CO2 in the blood
Coronary veins
systemic arterial blood pressure
31. 3 factors that dictate the affinity of hemoglobin for O2
hemophilia
Diastole
Temperature or metabolic rate
cardiac output (L/min)
32. CO2 is soluble in H2O - and thus some is dissolved and carried to lungs and tissues in plasma - O2 is not soluble in plasma at all
nutrients
coronary sinus
Third transportation of CO2 in the blood
heart rate
33. Confirmation of hemoglobin with O2 bound - where affinity is high 1. pH 2. pCO2 3.
Relaxed
Bundle of His
Valves of the venous system
Ohm's law
34. Opposing friction force to flow - which increases with decreased radius; determined by degree of contraction of arterial smooth muscle
macrophage
Hepatic portal vein
2 components of antigens
resistance
35. Hematocrit or RBC those compose 35-45% of the blood; cells are non - nucleated and have no organelles. Acquire ATP through glycolysis have biconcave shape to maximize surface area for binding O2
Erythrocytes
coronary sinus
fibrin
Cardiac muscle cells
36. Return of blood to the heart by the vena cava - where increased venous return causes increased stretching of the muscle (increases stroke volume)
glycolysis. RBC have no ETC - FA oxidation - or TCA cycle
bone marrow
CNS decreases vagal signal and sympathetic input increases
venous return
37. 2 ways to increase venous return
stroke volume
1. increase total blood volume by retaining more H2O 2. Contraction of large veins - propelling blood toward the heart
2 components of antigens
Fxn of circulatory system
38. Body's mechanism of preventing bleeding
Spleen and liver
hemostasis
2 components of antigens
Platelet fxn
39. Valves between the ventricle and the atria to prevent back flow
Blood plasma
atrioventricular valves
increase vagal signal and inhibits sympathetic input
nutrients
40. Is cardiac output the same or different btw the two ventricles?
It is the same - otherwise it would lead to fluid backup
High since the concentration of plasma proteins has increased due to movement of water
systolic blood pressure
cardiac output (L/min)
41. Because the veins have essentially 0 pressure - these valves ensure one - way flow - skeletal muscle contraction encourages flow through veins
Vagal Signal
Functional syncytium
Valves of the venous system
atrioventricular valves
42. Which is longer - diastole or systole?
Cardiac muscle cells
Diastole is longer
Capillaries
T- tubules
43. When the valve of a vein fails and back flow occurs; blood not being moved toward the heart
varicose veins
high osmolarity of tissues
heart
veins
44. The principle sugar in blood that maintains a relatively constant concentration for adequate nutrition
T- tubules
Glucose
tricuspid valve
urea
45. What causes tendency of water flow out of blood?
heart rate
high osmolarity of tissues
fats
Peripheral resistance
46. Why is the SA node the primary pacemaker?
Systole
It has the most Na leak channels - allowing to reach threshold potential first; all other nodes leak - but rate at as quick of a rate
Internodal tract
It is the same - otherwise it would lead to fluid backup
47. 73% of CO2 converted to carbonic acid by carbonic anhydrase - and carbonic acid is converted to bicarbonate - which acts a buffer
Coronary arteries
Primary transportation fo CO2 in the blood
Sickle cell anemia
oncotic pressure
48. Buffer in blood. Keeps pH around 7.4
oncotic pressure
Systole
primary bicarbonate generated from CO2.
Repolarization of nodes
49. When do Rh antibodies develop?
local autoregulation
Hemoglobin
when person that is Rh - is exposed to blood that is Rh+
macrophage
50. What is the direct cause of edema?
SA node
Repolarization of nodes
ABO blood group
increased hydrostatic pressure in the capillaries - which increases the fluid that leaks out of the capillaries into the interstitum