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Test your basic knowledge |
MCAT Biology Circulatory System
Start Test
Study First
Subjects
:
mcat
,
health-sciences
Instructions:
Answer 50 questions in 15 minutes.
If you are not ready to take this test, you can
study here
.
Match each statement with the correct term.
Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.
This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. When the valve of a vein fails and back flow occurs; blood not being moved toward the heart
bilirubin
glycolysis. RBC have no ETC - FA oxidation - or TCA cycle
varicose veins
T- tubules
2. Heart rate *stroke volume= (units)
chylomicrons
cardiac output (L/min)
systemic circulation
Waste
3. Opposing friction force to flow - which increases with decreased radius; determined by degree of contraction of arterial smooth muscle
2 components of antigens
Systole
resistance
B cells and T cells
4. Pump blood out of the heart at high pressures into arteries
Pulmonary and aortic semilunar valves
basophil
Portal systems
ventricles
5. Universal donor
bone marrow
O- since there are no surface antigens for antibodies to bind to...
Immunoglobulins (antibodies)
Perfusion
6. 2 lymphocytes
arteries
B cells and T cells
Frank - Starling Effect
atrioventricular valves
7. Produced during cell metabolism and diffuses through the endothelial cells into the blood stream - where it is picked up by the liver and converted to forms that can be excreted (all other wastes are picked up by the kidneys)
oncotic pressure
Frank - Starling Effect
albumin b/c it provides the bulk of oncotic pressure in blood vessels - preventing edema
Waste
8. Region that initiates start of cardiac cycle - which acts as a pacemaker of the heart; has unstable resting potential due to Na leak channels
Pulmonary and aortic semilunar valves
SA node
Functional syncytium
Ohm's law
9. 20% transported stuck to hemoglobin; why increased pCO2 decreases affinity of O2
Secondary transportation of CO2 in the blood
It is the same - otherwise it would lead to fluid backup
Platelet fxn
atria
10. Metabolic waste product in breakdown of amino acids
Perfusion
Frank - Starling Effect
urea
eosinophil
11. CO2 is soluble in H2O - and thus some is dissolved and carried to lungs and tissues in plasma - O2 is not soluble in plasma at all
tricuspid valve
1. increase total blood volume by retaining more H2O 2. Contraction of large veins - propelling blood toward the heart
local autoregulation
Third transportation of CO2 in the blood
12. Number of systole contractions per unit time
Baroreceptors
heart rate
SA node
adipocytes
13. Glycoproteins that are coded for by 3 alleles (A - B - i)
megakaryocytes
adrenergic tone
ABO blood group
Slow Ca channels
14. 73% of CO2 converted to carbonic acid by carbonic anhydrase - and carbonic acid is converted to bicarbonate - which acts a buffer
Capillaries
Primary transportation fo CO2 in the blood
Perfusion
Bundle of His
15. Peptide hormone secreted from the kidneys to increase RBC production in bone marrow
pulse pressure
Coronary arteries
Waste
Erythropoetin
16. 3 factors that dictate the affinity of hemoglobin for O2
megakaryocytes
Baroreceptors
Temperature or metabolic rate
fibrin
17. Excessive bleeding that results from defective proteins
hemophilia
atria
CNS decreases vagal signal and sympathetic input increases
Erythropoetin
18. Transportation of blood though the body and exchange of material btw blood and tissues
Fxn of circulatory system
tricuspid valve
Vagal Signal
increase vagal signal and inhibits sympathetic input
19. Pool of deoxygenated blood at low pressure - which collects blood from coronary veins - Only deoxygenated blood to not enter the right atrium via the vena cava
coronary sinus
atria and ventricles
albumin b/c it provides the bulk of oncotic pressure in blood vessels - preventing edema
Secondary transportation of CO2 in the blood
20. Have single layer endothelial cells w/ spaces in between cells called intercellular cleft
Capillaries
oncotic pressure
Coronary veins
Right atrium
21. Vessels that carry blood away from the heart at high pressure
Fast Na channels
Coronary veins
Right atrium
arteries
22. Inadequate blood flow - resulting in tissue damage due to shortage of O2 and nutrients - and increase of metabolic waste
Ischemia
heart rate
Pulmonary and aortic semilunar valves
High since the concentration of plasma proteins has increased due to movement of water
23. Hematocrit or RBC those compose 35-45% of the blood; cells are non - nucleated and have no organelles. Acquire ATP through glycolysis have biconcave shape to maximize surface area for binding O2
Erythrocytes
Slow Ca channels
Vagal Signal
Platelet fxn
24. Control of by ANS of rate of contraction through the Vagus nerve. Postganglionic release in SA node of ACH inhibits depolarization
neutrophil
Intercalated discs
hypoxia
Vagal Signal
25. Where blood passes through 2 sets of capillaries before returning to the heart; Evolved as direct transport routes
Ischemia
Portal systems
Blood plasma
venous blood pressure
26. Because the veins have essentially 0 pressure - these valves ensure one - way flow - skeletal muscle contraction encourages flow through veins
Valves of the venous system
Perfusion
It is the same - otherwise it would lead to fluid backup
Intercalated discs
27. Gap junctions in the cardiac muscle - where depolarization is communicated directly btw cytoplasm of neighboring cardiac cells
Intercalated discs
Hepatic portal vein
oncotic pressure
serum
28. Response by CNS when blood pressure is too low
CNS decreases vagal signal and sympathetic input increases
Sickle cell anemia
Slow Ca channels
It is the same - otherwise it would lead to fluid backup
29. Body's mechanism of preventing bleeding
hemostasis
Ischemia
Primary transportation fo CO2 in the blood
Rh blood group
30. Connects the two capillary beds of the intestine and the liver
Hepatic portal vein
Secondary transportation of CO2 in the blood
Granulocytes
Right atrium
31. AV valve between right atrium and right ventricle
Third transportation of CO2 in the blood
chylomicrons
basophil
tricuspid valve
32. Vessels that carry blood back to the heart at low pressure
ventricles
veins
Functional syncytium
bicuspid (mitral) valve
33. Protein that maintains oncotic pressure in capillaries
It is the same - otherwise it would lead to fluid backup
atria and ventricles
albumin
Granulocytes
34. Open when threshold is reached causing membrane potential to increase/depolarize; operate slower than Na channels
Functional syncytium
Ca channels
coronary sinus
Right atrium
35. Glucose - amino acids - and fats
Hemolytic disease of a newborn
Glucose
nutrients
nutrients - wastes - and WBC
36. Request by tissues to increase blood flow - where build up of metabolic waste causes arterioles to dialate
fibrin
bicuspid (mitral) valve
primary bicarbonate generated from CO2.
local autoregulation
37. Amount of blood pumped w/ each systolic contraction
adipocytes
Cardiac muscle cells
Vagal Signal
stroke volume
38. Key proteins for the function of the immune system that are produced and released by B- cells
Ca channels
Fxn of circulatory system
pulse pressure
Immunoglobulins (antibodies)
39. Caused by closure of Ca channels and opening of K channels
Diastole is longer
Inflammation
Repolarization of nodes
fibrinogen
40. Allow Na to leak across membrane - causing cell potential to get closer to threshold potential; allow threshold to be reached for Ca channels to open let Ca into the cell
primary bicarbonate generated from CO2.
Na leak channels
1. increase total blood volume by retaining more H2O 2. Contraction of large veins - propelling blood toward the heart
pulmonary circulation
41. Neural sympathetic input by postganglionic neuron of norepinephrine innervating arterial smooth muscle
amino acids and glucose
systolic blood pressure
adrenergic tone
local autoregulation
42. Absorbed in the intestine and packaged in chylomicrons - which enter the lymphatic system - and dumped into the subclavian vein via the thoracic duct; the liver takes fats once in blood - converts them to another lipoprotein and sends them to adipocy
Repolarization of nodes
fats
Vagal Signal
Frank - Starling Effect
43. Is cardiac output the same or different btw the two ventricles?
increased hydrostatic pressure in the capillaries - which increases the fluid that leaks out of the capillaries into the interstitum
It is the same - otherwise it would lead to fluid backup
amino acids and glucose
systemic circulation
44. Fat storage cells of the body
adipocytes
Sickle cell anemia
oncotic pressure
bilirubin
45. Mother has Rh - blood with Rh+ antibodies that attack the babies Rh+ blood
Valves of the venous system
Third transportation of CO2 in the blood
Hemolytic disease of a newborn
High since the concentration of plasma proteins has increased due to movement of water
46. Maximize entry of Ca into the cell by allowing entry of Ca extracellular environment; leads to contraction of actin - myosin fibers
Internodal tract
2 components of antigens
Relaxed
T- tubules
47. Pass through the capillaries in order to patrol the tissue for invading organisms; only macrophages and neutrophils can squeeze through cleft
WBC
primary bicarbonate generated from CO2.
adipocytes
Immunoglobulins (antibodies)
48. Flow of blood through a tissue
CNS decreases vagal signal and sympathetic input increases
Perfusion
valves
It has the most Na leak channels - allowing to reach threshold potential first; all other nodes leak - but rate at as quick of a rate
49. AV valve between left atrium and left ventricle
bicuspid (mitral) valve
adipocytes
veins
albumin
50. Receives deoxygenated blood from systemic circulation (superior and inferior vena cava)
Lipoproteins
hemostasis
Rh blood group
Right atrium