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Test your basic knowledge |
MCAT Biology Circulatory System
Start Test
Study First
Subjects
:
mcat
,
health-sciences
Instructions:
Answer 50 questions in 15 minutes.
If you are not ready to take this test, you can
study here
.
Match each statement with the correct term.
Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.
This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. Precursor to fibrin - which is necessary for blood clotting
atria
fibrinogen
Temperature or metabolic rate
Granulocytes
2. Large particles consisting of fats - cholesterol - and carrier proteins; transport lipids through the blood stream
heart rate
Lipoproteins
Internodal tract
neutrophil
3. Request by tissues to increase blood flow - where build up of metabolic waste causes arterioles to dialate
local autoregulation
stroke volume
systemic arterial blood pressure
capillaries
4. Produced during cell metabolism and diffuses through the endothelial cells into the blood stream - where it is picked up by the liver and converted to forms that can be excreted (all other wastes are picked up by the kidneys)
Waste
varicose veins
O- since there are no surface antigens for antibodies to bind to...
Spleen and liver
5. Glucose - amino acids - and fats
nutrients
Primary transportation fo CO2 in the blood
Hepatic portal vein
Inflammation
6. Amount of blood pumped w/ each systolic contraction
stroke volume
atria and ventricles
primary bicarbonate generated from CO2.
increased hydrostatic pressure in the capillaries - which increases the fluid that leaks out of the capillaries into the interstitum
7. Metabolic waste product in breakdown of amino acids
urea
hepatic portal system and hypothalamic - hypophosial portal system
local autoregulation
Immunoglobulins (antibodies)
8. Opposing friction force to flow - which increases with decreased radius; determined by degree of contraction of arterial smooth muscle
venous blood pressure
resistance
capillaries
high osmolarity of tissues
9. The difference in pressure divided blood flow; controlled by the sympathetic nervous system generating adrenergic tone
systemic arterial blood pressure
Peripheral resistance
venous return
Rh blood group
10. Monocyte that phagocytoses debris and microorganisms - has amoeboid motility - and displays chemotaxis
Hepatic portal vein
Lipoproteins
Secondary transportation of CO2 in the blood
macrophage
11. Caused by closure of Ca channels and opening of K channels
increased hydrostatic pressure in the capillaries - which increases the fluid that leaks out of the capillaries into the interstitum
Repolarization of nodes
Valves of the venous system
Lipoproteins
12. Voltage - gated channels that open quickly; open at threshold potential
Fast Na channels
primary bicarbonate generated from CO2.
Third transportation of CO2 in the blood
SA node
13. Control of by ANS of rate of contraction through the Vagus nerve. Postganglionic release in SA node of ACH inhibits depolarization
CNS decreases vagal signal and sympathetic input increases
tricuspid valve
macrophage
Vagal Signal
14. Vessels that carry blood back to the heart at low pressure
veins
Ohm's law
serum
varicose veins
15. Return of blood to the heart by the vena cava - where increased venous return causes increased stretching of the muscle (increases stroke volume)
It is the same - otherwise it would lead to fluid backup
Valves of the venous system
venous return
chylomicrons
16. Open when threshold is reached causing membrane potential to increase/depolarize; operate slower than Na channels
Inflammation
Erythropoetin
Ca channels
Waste
17. As low as pressure gets btw heart beats in arteries
Erythrocytes
Waste
bilirubin
diastolic blood pressure
18. Breakdown product of the hemogloblin heme group
5 phases of cardiac muscle cell contraction
Hepatic portal vein
bilirubin
fibrinogen
19. Connected to SA node via internodal tract - and passes signal to Common bundle of His to contract ventricles
AV node
It has the most Na leak channels - allowing to reach threshold potential first; all other nodes leak - but rate at as quick of a rate
Portal systems
fibrinogen
20. What is the direct cause of edema?
urea
when person that is Rh - is exposed to blood that is Rh+
Hemolytic disease of a newborn
increased hydrostatic pressure in the capillaries - which increases the fluid that leaks out of the capillaries into the interstitum
21. Store and release histamine and are involved in allergic rxns
arteries
hemophilia
macrophage
basophil
22. Maximize entry of Ca into the cell by allowing entry of Ca extracellular environment; leads to contraction of actin - myosin fibers
T- tubules
Systole
resistance
1. increase total blood volume by retaining more H2O 2. Contraction of large veins - propelling blood toward the heart
23. Heart rate *stroke volume= (units)
cardiac output (L/min)
heart rate
Diastole is longer
It has the most Na leak channels - allowing to reach threshold potential first; all other nodes leak - but rate at as quick of a rate
24. Absorbed in the intestine and packaged in chylomicrons - which enter the lymphatic system - and dumped into the subclavian vein via the thoracic duct; the liver takes fats once in blood - converts them to another lipoprotein and sends them to adipocy
systemic arterial blood pressure
heart
hemophilia
fats
25. Bone marrow cells that give rise to RBC and platelets
oncotic pressure
Inflammation
high osmolarity of tissues
megakaryocytes
26. Response by CNS when blood pressure is too low
T- tubules
CNS decreases vagal signal and sympathetic input increases
Tense
bicuspid (mitral) valve
27. Occurs when increased cardiac output is needed; the postganglionic nerve directly innervates the heart - releasing norepinephrine - increasing heart rate and force of contraction
bicuspid (mitral) valve
Thrombus
Sympathetic regulation of heart
venous blood pressure
28. Hematocrit or RBC those compose 35-45% of the blood; cells are non - nucleated and have no organelles. Acquire ATP through glycolysis have biconcave shape to maximize surface area for binding O2
heart rate
nutrients - wastes - and WBC
eosinophil
Erythrocytes
29. Lipoproteins that enter lacteal vessels of lymphatic system in the intestinal wall
fats
chylomicrons
Slow Ca channels
eosinophil
30. Essentially 0 mmHg - which results b/c of branching of vessels dissipating pressure to overcome resistance
venous blood pressure
Baroreceptors
Hepatic portal vein
albumin b/c it provides the bulk of oncotic pressure in blood vessels - preventing edema
31. Vessels where deoxygenated blood from coronary sinus continue to flow into heart
Inflammation
Vagal Signal
venous return
Coronary veins
32. Stretching to greater degree of heart muscle causes more forceful contraction; stretching increase occur by increasing fluid volume
Repolarization of nodes
Right atrium
Frank - Starling Effect
local autoregulation
33. Transportation of blood though the body and exchange of material btw blood and tissues
Fxn of circulatory system
ventricles
hepatic portal system and hypothalamic - hypophosial portal system
Inflammation
34. Contraction of the ventricles - where pressure increases rapidly - causing AV valves to close - Marks the beginning of the 'lub' sound
Ohm's law
Systole
high osmolarity of tissues
megakaryocytes
35. What is the only process RBC use to generate ATP?
Right atrium
Baroreceptors
glycolysis. RBC have no ETC - FA oxidation - or TCA cycle
AB+ since no antibodies are made to any blood type
36. Highest blood pressure that occurs during ventricular contraction
Arterial pressure=ventricular pressure
systolic blood pressure
2 components of antigens
high osmolarity of tissues
37. Active form of fibrinogen - protein forms a mesh that holds platelet plug together to protect wound - ibrinogen is converted to (blank) by thrombin
pulmonary circulation
bone marrow
fibrin
atria
38. AV valve between left atrium and left ventricle
hemostasis
valves
bicuspid (mitral) valve
Na leak channels
39. Response by CNS when blood pressure is too high
increase vagal signal and inhibits sympathetic input
coronary sinus
Secondary transportation of CO2 in the blood
macrophage
40. Inadequate blood flow - resulting in tissue damage due to shortage of O2 and nutrients - and increase of metabolic waste
neutrophil
serum
Ischemia
Platelet fxn
41. Neural sympathetic input by postganglionic neuron of norepinephrine innervating arterial smooth muscle
Portal systems
resistance
adrenergic tone
Hemolytic disease of a newborn
42. AV valve between right atrium and right ventricle
tricuspid valve
It has the most Na leak channels - allowing to reach threshold potential first; all other nodes leak - but rate at as quick of a rate
adrenergic tone
Fxn of circulatory system
43. 2 chambers of the heart
CNS decreases vagal signal and sympathetic input increases
Valves of the venous system
atria and ventricles
Tense
44. 55% of whole blood that is composed of electrolytes - lipoproteins - sugars - buffer - and metabolic waste
increased hydrostatic pressure in the capillaries - which increases the fluid that leaks out of the capillaries into the interstitum
pulse pressure
Blood plasma
High since the concentration of plasma proteins has increased due to movement of water
45. 20% transported stuck to hemoglobin; why increased pCO2 decreases affinity of O2
nutrients
systolic blood pressure
heart
Secondary transportation of CO2 in the blood
46. Valves between the ventricle and the atria to prevent back flow
atrioventricular valves
venous return
Ischemia
bone marrow
47. Confirmation of hemoglobin with no O2 bound - so it has low affinity
diastolic blood pressure
megakaryocytes
eosinophil
Tense
48. What causes tendency of water flow out of blood?
macrophage
high osmolarity of tissues
hypoxia
WBC
49. Aggregate at site of damage to a blood vessel and form a platelet plug to stop bleeding
stroke volume
WBC
Baroreceptors
Platelet fxn
50. Confirmation of hemoglobin with O2 bound - where affinity is high 1. pH 2. pCO2 3.
Relaxed
resistance
5 phases of cardiac muscle cell contraction
atria and ventricles