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Test your basic knowledge |
MCAT Biology Circulatory System
Start Test
Study First
Subjects
:
mcat
,
health-sciences
Instructions:
Answer 50 questions in 15 minutes.
If you are not ready to take this test, you can
study here
.
Match each statement with the correct term.
Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.
This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. Phagocytose bacteria resulting in pus; amoeboid motility and chemotaxis
when person that is Rh - is exposed to blood that is Rh+
Diastole
neutrophil
Na leak channels
2. Inadequate blood flow - resulting in tissue damage due to shortage of O2 and nutrients - and increase of metabolic waste
Ischemia
hemostasis
Functional syncytium
stroke volume
3. Allow Na to leak across membrane - causing cell potential to get closer to threshold potential; allow threshold to be reached for Ca channels to open let Ca into the cell
megakaryocytes
stroke volume
AV node
Na leak channels
4. Absorbed by the GI tract and brought to the liver via the hepatic portal vein - where they are stored in the liver and enter the blood stream when needed
amino acids and glucose
Hemolytic disease of a newborn
Blood plasma
systemic circulation
5. Highest blood pressure that occurs during ventricular contraction
amino acids and glucose
oncotic pressure
systolic blood pressure
Bundle of His
6. Return of blood to the heart by the vena cava - where increased venous return causes increased stretching of the muscle (increases stroke volume)
increase vagal signal and inhibits sympathetic input
local autoregulation
Baroreceptors
venous return
7. First branches from the aorta that provide the heart's blood supply
Coronary arteries
Platelet fxn
fibrin
oncotic pressure
8. Protein in RBC that transport O2 though the blood since O2 is too hydrophobic in plasma; protein has 4 subunits that change confirmation cooperatively depending on the concentration of O2
valves
Hemoglobin
venous blood pressure
Erythrocytes
9. Which is longer - diastole or systole?
Diastole is longer
Fast Na channels
atria
adipocytes
10. 2 lymphocytes
Ischemia
Ohm's law
hepatic portal system and hypothalamic - hypophosial portal system
B cells and T cells
11. Request by tissues to increase blood flow - where build up of metabolic waste causes arterioles to dialate
Ischemia
local autoregulation
Inflammation
2 components of antigens
12. Voltage - gated channels that open quickly; open at threshold potential
megakaryocytes
Fast Na channels
nutrients
Ca channels
13. Confirmation of hemoglobin with no O2 bound - so it has low affinity
Hemoglobin
Tense
1. increase total blood volume by retaining more H2O 2. Contraction of large veins - propelling blood toward the heart
Inflammation
14. Connected to SA node via internodal tract - and passes signal to Common bundle of His to contract ventricles
chylomicrons
Slow Ca channels
AV node
Coronary arteries
15. The difference in pressure divided blood flow; controlled by the sympathetic nervous system generating adrenergic tone
ventricles
O- since there are no surface antigens for antibodies to bind to...
Peripheral resistance
hemophilia
16. Open when threshold is reached causing membrane potential to increase/depolarize; operate slower than Na channels
Ca channels
bilirubin
Vagal Signal
macrophage
17. Rh factor that follows dominant pattern (Rh+ in heterozygote)
venous return
Rh blood group
Coronary arteries
Ohm's law
18. Path where impulse travels from SA to AV node
venous blood pressure
Internodal tract
Baroreceptors
Intercalated discs
19. Buffer in blood. Keeps pH around 7.4
Sickle cell anemia
pulse pressure
primary bicarbonate generated from CO2.
Fast Na channels
20. What is the direct cause of edema?
increased hydrostatic pressure in the capillaries - which increases the fluid that leaks out of the capillaries into the interstitum
Right atrium
High since the concentration of plasma proteins has increased due to movement of water
AB+ since no antibodies are made to any blood type
21. 2 ways to increase venous return
stroke volume
amino acids and glucose
1. increase total blood volume by retaining more H2O 2. Contraction of large veins - propelling blood toward the heart
adipocytes
22. Opposing friction force to flow - which increases with decreased radius; determined by degree of contraction of arterial smooth muscle
Glucose
Arterial pressure=ventricular pressure
resistance
Coronary arteries
23. Valves between the large arteries and the ventricles
Pulmonary and aortic semilunar valves
ventricles
Internodal tract
to transport O2 to tissues and CO2 to the lungs
24. Where do all components of the blood develop from?
1. increase total blood volume by retaining more H2O 2. Contraction of large veins - propelling blood toward the heart
bone marrow
albumin
Cardiac muscle cells
25. At the end of the capillary - is the osmotic pressure high or low?
to transport O2 to tissues and CO2 to the lungs
varicose veins
High since the concentration of plasma proteins has increased due to movement of water
fibrin
26. Large particles consisting of fats - cholesterol - and carrier proteins; transport lipids through the blood stream
Lipoproteins
AB+ since no antibodies are made to any blood type
glycolysis. RBC have no ETC - FA oxidation - or TCA cycle
Erythropoetin
27. At position 6 - missense mutation substitutes valine for glutamate. valine is hydrophobic - where glutamate was charged. It is an autosomal recessive disease where RBCs accumulated in small vessels - heterozygote for (blank) shows resistance to malar
Tense
Capillaries
Sickle cell anemia
Slow Ca channels
28. Transportation of blood though the body and exchange of material btw blood and tissues
capillaries
Ischemia
Fxn of circulatory system
pulmonary circulation
29. Adequate circulation - but O2 supply is reduced (no build up waste products or loss of nutrients)
primary bicarbonate generated from CO2.
hypoxia
It is the same - otherwise it would lead to fluid backup
ventricles
30. What is the most important plasma protein in the body? Why?
Platelet fxn
venous return
albumin b/c it provides the bulk of oncotic pressure in blood vessels - preventing edema
Tense
31. CO2 is soluble in H2O - and thus some is dissolved and carried to lungs and tissues in plasma - O2 is not soluble in plasma at all
Third transportation of CO2 in the blood
Diastole
Slow Ca channels
Primary transportation fo CO2 in the blood
32. Region that initiates start of cardiac cycle - which acts as a pacemaker of the heart; has unstable resting potential due to Na leak channels
atria
Immunoglobulins (antibodies)
SA node
glycolysis. RBC have no ETC - FA oxidation - or TCA cycle
33. Key proteins for the function of the immune system that are produced and released by B- cells
atria and ventricles
increased hydrostatic pressure in the capillaries - which increases the fluid that leaks out of the capillaries into the interstitum
Immunoglobulins (antibodies)
AB+ since no antibodies are made to any blood type
34. 3 factors that dictate the affinity of hemoglobin for O2
Thrombus
Peripheral resistance
Temperature or metabolic rate
capillaries
35. Why is the SA node the primary pacemaker?
chylomicrons
It has the most Na leak channels - allowing to reach threshold potential first; all other nodes leak - but rate at as quick of a rate
macrophage
when person that is Rh - is exposed to blood that is Rh+
36. Purpose of erythrocytes?
to transport O2 to tissues and CO2 to the lungs
Functional syncytium
Fast Na channels
Na leak channels
37. Protein that maintains oncotic pressure in capillaries
Cardiac muscle cells
tricuspid valve
atrioventricular valves
albumin
38. Body's mechanism of preventing bleeding
increased hydrostatic pressure in the capillaries - which increases the fluid that leaks out of the capillaries into the interstitum
Capillaries
Primary transportation fo CO2 in the blood
hemostasis
39. Where blood passes through 2 sets of capillaries before returning to the heart; Evolved as direct transport routes
Portal systems
heart rate
Baroreceptors
Temperature or metabolic rate
40. Resting membrane potential of -90mV and have long duration action potentials
Sympathetic regulation of heart
chylomicrons
Cardiac muscle cells
hypoxia
41. Voltage - gated channels that stay open longer than Na channels and open later responsible for the plateau phase of cardiac muscle contraction
Sickle cell anemia
Slow Ca channels
Coronary veins
venous blood pressure
42. As low as pressure gets btw heart beats in arteries
pulmonary circulation
Waste
Hemoglobin
diastolic blood pressure
43. The principle sugar in blood that maintains a relatively constant concentration for adequate nutrition
Sickle cell anemia
amino acids and glucose
Glucose
albumin
44. Crosses septum and connects to Purkinje fibers to allow coordinated contraction of ventricles. Key is that is slows transmission across septum to allow ventricles to fully fill before contraction
T- tubules
bicuspid (mitral) valve
Bundle of His
Systole
45. Response by CNS when blood pressure is too high
veins
serum
Internodal tract
increase vagal signal and inhibits sympathetic input
46. Plasma that lacks clotting proteins
2 components of antigens
serum
bicuspid (mitral) valve
Blood plasma
47. Lipoproteins that enter lacteal vessels of lymphatic system in the intestinal wall
Slow Ca channels
Cardiac muscle cells
chylomicrons
Right atrium
48. Control of by ANS of rate of contraction through the Vagus nerve. Postganglionic release in SA node of ACH inhibits depolarization
Vagal Signal
Portal systems
Inflammation
glycolysis. RBC have no ETC - FA oxidation - or TCA cycle
49. Precursor to fibrin - which is necessary for blood clotting
fibrinogen
Lipoproteins
hepatic portal system and hypothalamic - hypophosial portal system
1. increase total blood volume by retaining more H2O 2. Contraction of large veins - propelling blood toward the heart
50. 2 portal systems to know
heart
hepatic portal system and hypothalamic - hypophosial portal system
Coronary arteries
local autoregulation