Test your basic knowledge |

Subjects : health-sciences, usmle
Instructions:
  • Answer 50 questions in 15 minutes.
  • If you are not ready to take this test, you can study here.
  • Match each statement with the correct term.
  • Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.

This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. diffuse fibrosis of liver destroying nl architecture with nodular regeneration






2. What are the hindgut structures and what supplies their blood and PANS innvervation






3. What kind of gall stones are mostly radiolucent and what percentage is opaque and due to what?






4. What are the extraintestinal manifestations of crohns






5. What does GET SMASHED stand for in acute pancreatitis






6. What are additional risk factors for CRC






7. What is contained with in the hepatoduodenal ligament - What two spaces does it connect - and when would you need to compress it






8. What kind of lesions are characteristic of duodenal PUD vs cancer






9. What transforms conjugated bilirubin to urobilinogen






10. What drug blocks the H2R






11. At what level do the testicular/ovarian arteries exit the aorta






12. Failure of relaxation of lower esophageal sphincter - Name and etiology






13. What test and result confirms H pylori infxn






14. What can hemochromatosis be secondary to...






15. What portion of the bowel does sprue effect






16. Which is used more quickly - an oral glucose load - or that by IV






17. What are the tumor markers for pancreatic adenocarcinoma






18. In MSI - What is the mechanism for CRC and what syndrome is associated with this defect






19. How does gastrin increase acid secretion?






20. What is a positive murphy's sign






21. How do you DX and TX gallstones






22. What do the rugae of stomach look like in menetriers disease






23. Bile is critical for exrection of what substance






24. What do you use to diagnose meckels






25. Achalasia increases the risk For what complication






26. Why does volvulus occur more at cecum and sigmoid colon






27. Where on the stomach does the gastrohepatic ligament attach to - What does it contain - and How is used in surgery






28. What happens to the short gastics if the splenic artery is blocked






29. What is the classic triad of hemochromatosis

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30. What is the omphalomesenteric cyst






31. What causes carcinoid syndrome amd What are the symptoms






32. What are the main components of bile






33. Where is IgA shuttled






34. Pain improves with defaction - changes in stool frequency and changes in stool appearance are associated with






35. Where does crohns usually affect the GI tract






36. Which IBD has skip lesions and can hit any portion of the GI tract but sprares the rectum - and Which is mainly has continuous lesions in the colon and always has rectal involvement






37. What receptors does ACH bind on the parietal cells and What does it activate






38. What histological findings are present in the stomach






39. what kind of fistula is associated with diverticulitis






40. inflammatino of gallbadder






41. What are the two molecular pathways that lead to CRC






42. What is the TX of physiologic neonatal jaundice






43. AD - mutation of APC gene on chromosome 5q - two hit hypothesis - 100% progress to CRC - 1000s of polyps - pancolonic - rectal involvement






44. What are the histological findings of the colon






45. Where is the pectinate line






46. in jaundice of obstructive etiolgy is the hyperbilirubinemia conj or unconj - what happens to urine bili - and what happens to urine urobilinogen






47. Which IBD is autoimmune and which may be a disordered response to bacteria






48. Gallstones that reach the common channel at ampulla can block which two ducts






49. Are single polyps malignant in peutz jehgers






50. What serum enzyme is elevated inacute pancreatitis