Test your basic knowledge |

Subjects : health-sciences, usmle
Instructions:
  • Answer 50 questions in 15 minutes.
  • If you are not ready to take this test, you can study here.
  • Match each statement with the correct term.
  • Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.

This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. How are all 3 monosaccharides transported to the blood






2. What are the histological findings in the ileum

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3. What is the most common diaphragmatic hernia and What are the two types






4. Which is used more quickly - an oral glucose load - or that by IV






5. Which monosaccharides are absorbed by the enterocytes and which transporters carry each






6. in budd chiari syndrome - Where is the congestion and necrosis






7. Autodigestion of pancreas by pancreatic enzymes






8. How does gastrin increase acid secretion?






9. absent UDPGT - presents early in life - early mortality






10. What arteries exit just below the SMA






11. What is indirect bilirubin






12. alcoholic liver disease with short term change and moderate EtOH intake - macrovesicular fatty change that may be reversible






13. What kind of gall stones are mostly radiolucent and what percentage is opaque and due to what?






14. What infection causes Whipple disease and What can you see on LM






15. What structures feed into the cystic duct






16. Gastrin - source - action - regulation






17. How do you DX and TX gallstones






18. What does a gastrinoma cause






19. signet ring cells - acanthosis nigracans - dz - character/association - spread






20. What is the most important mechanism in gastric acid secretion






21. What receptors does ACH bind on the parietal cells and What does it activate






22. rare - often fatal childhood hepatoencephalopathy

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23. Unencapsulated lymphoid tissue found in lamina proporia and submucosa of small intestine containing specialized M cells that take up antigen






24. Where is IgA shuttled






25. what kind of muscle is in the upper 1/3 of esophagus






26. in jaundice of obstructive etiolgy is the hyperbilirubinemia conj or unconj - what happens to urine bili - and what happens to urine urobilinogen






27. What is the classic triad of hemochromatosis

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28. What is the presentation of pancreatic adenocarcinoma






29. How do secondary biliary cirrhosis - primary biliary cirrhosis and primary sclerosing cholangitis present






30. What layer in the mucosa is responsible for support






31. a false diverticulum - herniation of mucosal tissue at junction of pharynx and esophagus - How does this present






32. persistence of viteline duct or yolk stalk possibly containing ectopic acid secreting gastric mucosa and/or pancreatic tissue






33. gastric hypertrophy with protein loss - parietal cell atrophy and inc mucous cells






34. Achalasia can be secondary to what infectious disease common in South America






35. Bilirubin is the product of what?






36. What layer in the mucosa is responsible for absorption






37. When do you see hypertrophy of brunners glands






38. subQ peribumbilical metastasis






39. Progressive dyshphage beginning with solids and moving to liquids and weight loss






40. How does brain injury lead to acute gastritis and What is it called






41. What layer in the mucosa is repsonsible for motility






42. What serum marker increases with primary biliary cirrhosis and what other autoimmune conditions are it associated with






43. involvement of left supraclavicular node by mets from stomach

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44. Is there any structural abnl with IBS - What is the course of disease and presentation






45. How is bilirubin carried in the blood






46. Which serum enzyme increases with heavy EtOH consumption






47. What are causes of extrahepatic biliary obstruction






48. How does loss of NO secretion affect the esophagus and what results






49. What are the two molecular pathways that lead to CRC






50. What does GET SMASHED stand for in acute pancreatitis