Test your basic knowledge |

Subjects : health-sciences, usmle
Instructions:
  • Answer 50 questions in 15 minutes.
  • If you are not ready to take this test, you can study here.
  • Match each statement with the correct term.
  • Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.

This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. How does brain injury lead to acute gastritis and What is it called






2. What happens to the short gastics if the splenic artery is blocked






3. Where does type B chronic gastritis occur and What causes it






4. What is the lumen of the pancreatic duct






5. What does a gastrinoma cause






6. what percentage of colonic polyps are non - neoplastic






7. What are the signs and symptoms of budd chiari






8. What cell produces IF and What does it do






9. What layer in the mucosa is responsible for absorption






10. Which is used more quickly - an oral glucose load - or that by IV






11. If the abdominal aorta is blocked - How does blood get to the left colic artery






12. alcoholic liver disease that requires sustained - long term consumption - with swollen and necrotic hepatocytes with neutrophilic infiltration






13. What are the foregut structures and what supplies their blood and PANS innvervation






14. in jaundice of obstructive etiolgy is the hyperbilirubinemia conj or unconj - what happens to urine bili - and what happens to urine urobilinogen






15. What is the frequency of basal electric rhythm of the ilieum






16. trypsinogen is converted to trypsin via what enzyme






17. What serum markers increase in cholecystitis with bile duct involvement






18. What is indirect bilirubin






19. Why are most diverticula considered false






20. What source of salivary secretion is the most serous and What is the most mucinous






21. Where does copper accumulate in Wilsons and What are ABCD






22. Where is B12 absorbed






23. What layer in the mucosa is repsonsible for motility






24. How does gastrin increase acid secretion?






25. alcoholic liver disease with short term change and moderate EtOH intake - macrovesicular fatty change that may be reversible






26. How is salivary secretion stimulated






27. People of what decent are associated with celiac sprue and what findings/antibodies are present






28. Where are oligosaccharide hydrolases and What do they do






29. What is the action of NO as a GI hormone






30. telescoping of 1 bowel segment into distal segment which can compromise blood supply - abdominal emergency in early childhood






31. How do NSAIDs cause acute gastritis






32. In MSI - What is the mechanism for CRC and what syndrome is associated with this defect






33. early bilious vomiting - with proximal stomach distention - double bubble - due to failure of recanalization of small bowel - dz and association






34. How do you DX and TX gallstones






35. What is contained within the muscularis externa






36. What is the frequency of basal electric rhythm in the duodenum






37. What portion of the bowel does sprue effect






38. Malabsorption syndromes have what common clinical presentation






39. In PUD with a duodenal ulcer does pain inc or dec with meals






40. What serum enzyme is decreased in wilsons disease






41. What intervention will intervention will relieve portal HTN






42. What conditions are associated with budd chiari






43. What is the mechanism for reyes syndrome






44. What findings are associated with reyes






45. What are the complications of duodenal PUD






46. What factors increase risk of malignancy of adenomatous polyps






47. multiple juvenil polyps in GI tract - risk






48. What infection causes Whipple disease and What can you see on LM






49. What is charcot triad of cholangitis






50. mostly sporadic lesions in children < 5 - 80% in rectum - When is there no risk of malignant potential