Test your basic knowledge |

Subjects : health-sciences, usmle
Instructions:
  • Answer 50 questions in 15 minutes.
  • If you are not ready to take this test, you can study here.
  • Match each statement with the correct term.
  • Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.

This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. What conditions are associated with budd chiari






2. What transforms conjugated bilirubin to urobilinogen






3. Is there any structural abnl with IBS - What is the course of disease and presentation






4. What are the layers of the gut wall from inside out






5. What is the path of an indirect inguinal hernia






6. What layer in the mucosa is repsonsible for motility






7. Acute gastritis is caused By what process






8. What converts inactive pepsinogen to pepsin






9. What histological findings are present in the esophagus






10. How do NSAIDs cause acute gastritis






11. What is the action of NO as a GI hormone






12. Tumor of neuroendocrine cells constituting 50% of small bowel tumors - most common sites are the appendix - ileum and rectum






13. in jaundice of obstructive etiolgy is the hyperbilirubinemia conj or unconj - what happens to urine bili - and what happens to urine urobilinogen






14. What are the complications of duodenal PUD






15. What carcinogens are associated with HCC






16. What is Trousseau's sign






17. Where is there sclerosis in alcoholic cirrohosis






18. subQ peribumbilical metastasis






19. crigler - najjar type II responds to which therapy and How does it work






20. At what spinal level does the celiac trunk exit






21. What layer in the mucosa is responsible for absorption






22. AD - mutation of APC gene on chromosome 5q - two hit hypothesis - 100% progress to CRC - 1000s of polyps - pancolonic - rectal involvement






23. What is pancreatic adenocarcinoma associated with






24. Who gets gastric ulcers






25. What is the other name for GIP (gastric inhibitory peptide)






26. How is salivary secretion stimulated






27. What is the cause of physiologic neonatal jaundice






28. How do burns cause acute gastritis and What is it called

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29. alcoholic liver disease that requires sustained - long term consumption - with swollen and necrotic hepatocytes with neutrophilic infiltration






30. What drug inhibits the H/K ATPase






31. What does bicab do in the mouth






32. How does HCC spread - What is a common tumor marker and what syndrome can it lead to...






33. What is the presenting course for appendicity

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34. gastric hypertrophy with protein loss - parietal cell atrophy and inc mucous cells






35. What are the results of hemochromatosis






36. What cell produces IF and What does it do






37. concentric onion skin bile duct fibrosis






38. Gq and inc cAMP both work to do what in parietal cells






39. trypsinogen is converted to trypsin via what enzyme






40. What is the main symptom if a VIPoma






41. What are the complications of Meckels






42. Where is IgA shuttled






43. How is the diagonsis of CRC made






44. At what spinal level does the is the bifurcation of aorta






45. occlusion of IVC or hepatic veins






46. Esophagitis can result From which 3 infectious agents - or chemical ingestion






47. rare - often fatal childhood hepatoencephalopathy

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48. congenital megacolon characterized by lack of ganglion/enteric nervous plexuses in segment on intestinal biopsy






49. a false diverticulum - herniation of mucosal tissue at junction of pharynx and esophagus - How does this present






50. Through which aspect of the inguinal canal does a direct inguinal go