Test your basic knowledge |

Subjects : health-sciences, usmle
Instructions:
  • Answer 50 questions in 15 minutes.
  • If you are not ready to take this test, you can study here.
  • Match each statement with the correct term.
  • Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.

This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. What layer in the mucosa is responsible for support






2. What serum markers increase in cholecystitis with bile duct involvement






3. What does loss of p53 cause






4. At what level of the spine does the IM exit the aorta






5. How does brain injury lead to acute gastritis and What is it called






6. What gives urine its characteristic color






7. What layer of fascia covers a direct inguinal hernia






8. Achalasia increases the risk For what complication






9. acute bowel obstruction - commonly from a recent surgery - can have well demarcated necrotic zones






10. What is the rate limiting step of carbohydrate digestion






11. What parts of the small bowel can tropical sprue effect






12. What kind of muscle is in the middle 1/3 of esophagus






13. What happens to the short gastics if the splenic artery is blocked






14. What layer in the mucosa is repsonsible for motility






15. If the abdomincal aorta is obstructed - What is the path of blood to the inferior epigastric






16. What reaction does salivary amylase catalyze






17. Where is the deep inguinal ring relative to the inferior epigastric vessels






18. Dysphagia in achalasia results from






19. What is the path of a direct inguinal hernia and Where does it travel relative to the inferior epigastric artery

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20. mildly dec UDPGT or dec bilirubin uptake - asymptomatic - elevated uncong bili without over hemolysis

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21. What are the extraintestinal manifestations of crohns






22. When do you see hypertrophy of brunners glands






23. Where does copper accumulate in Wilsons and What are ABCD






24. What are the complications of acute pancreatitis






25. What percentage of gall stones are cholesterol stones and What are the associations






26. involvement of left supraclavicular node by mets from stomach

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27. What ligament connects the liver to the abdominal wall - What is contained within it and From what structure is it derived






28. FAP + osseous and soft tissue tumors - retinal hyperplasia

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29. telescoping of 1 bowel segment into distal segment which can compromise blood supply - abdominal emergency in early childhood






30. in budd chiari syndrome - Where is the congestion and necrosis






31. Tumor of neuroendocrine cells constituting 50% of small bowel tumors - most common sites are the appendix - ileum and rectum






32. In alchoholic hepatitis which liver enzyme is higher






33. Why does volvulus occur more at cecum and sigmoid colon






34. Acute gastritis is caused By what process






35. Which kind of hemorrhoids are painful and why






36. Who gets gastric ulcers






37. What are the foregut structures and what supplies their blood and PANS innvervation






38. What is the lumen of the pancreatic duct






39. What causes nutmeg liver






40. What receptors does ACH bind on the parietal cells and What does it activate






41. What tumor cause PUD with duodenal ulcer and what glands become hypertrophied






42. AD mutation in DNA mismatch repair genes - 80% progress to CRC - proximal colon always involved






43. likely infectious form of malabsorption - responds to antibiotics






44. What receptors does gastrin bind on the parietal cell and What does it activate






45. In jaundice of hepatocellular etiology - is the hyperbilirubinemia conjugate or UN - what happens to urine bili - and urine urobilinogen






46. Where does crohns usually affect the GI tract






47. What do tumors that arise in the head of the pancreas cause






48. What is the epi for CRC






49. What structures feed into the cystic duct






50. How does loss of NO secretion affect the esophagus and what results