Test your basic knowledge |

Subjects : health-sciences, usmle
Instructions:
  • Answer 50 questions in 15 minutes.
  • If you are not ready to take this test, you can study here.
  • Match each statement with the correct term.
  • Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.

This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. in carcinoid tumors - What is seen on EM






2. Progressive dyshphage beginning with solids and moving to liquids and weight loss






3. subQ peribumbilical metastasis






4. How do burns cause acute gastritis and What is it called

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5. What receptor does histamine bind on the parietal cell and What does it activate






6. bilateral mets to ovaries with abundant mucus - signet ring cells






7. What does autoimmune destruction of parietal cells lead to...






8. mostly sporadic lesions in children < 5 - 80% in rectum - When is there no risk of malignant potential






9. involvement of left supraclavicular node by mets from stomach

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10. In viral hepatitis - which liver enzyme is higher






11. What is contained with in the hepatoduodenal ligament - What two spaces does it connect - and when would you need to compress it






12. In MSI - What is the mechanism for CRC and what syndrome is associated with this defect






13. If the abdomincal aorta is obstructed - What is the path of blood to the inferior epigastric






14. What kind of pathways do CCK act on to cause pancreatic secretion






15. What are the complications of chronic pancreatitis






16. What is the presentation of pancreatic adenocarcinoma






17. gastric hypertrophy with protein loss - parietal cell atrophy and inc mucous cells






18. What layer in the mucosa is responsible for absorption






19. If the hemochromatosis is primary - What is the pattern of inheritance






20. What histological findings are present in the stomach






21. What histological findings are present in the esophagus






22. What is the triad of Plummer - Vinson syndrome






23. What are the four Fs of gallstones






24. What is the frequency of basal electric rhythm of the ilieum






25. Autoantibodies to gluten (gliadin) in wheat and other grains






26. What are motilin receptor agonists used for clinically






27. How many layers outpouch in a false diverticula and they occur At what point of weakness and what?






28. secretin - source - action - regulation






29. What are the extraintestinal manifestations of crohns






30. What is the action of NO as a GI hormone






31. At what spinal level does the SMA exit






32. What are the histological findings in the duodenum

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33. alcoholic liver disease with short term change and moderate EtOH intake - macrovesicular fatty change that may be reversible






34. diffuse fibrosis of liver destroying nl architecture with nodular regeneration






35. What drug inhibits the H/K ATPase






36. Achalasia increases the risk For what complication






37. Where does copper accumulate in Wilsons and What are ABCD






38. in budd chiari syndrome - Where is the congestion and necrosis






39. Through which aspect of the inguinal canal does a direct inguinal go






40. What are the ABCDEF of esophageal cancer






41. Mucosal lacerations at the gastroesophageal junction due to severe vomiting - presentation - and risk groups






42. How does hirschsprung present and appear on imaging






43. Where on the stomach does the gastrohepatic ligament attach to - What does it contain - and How is used in surgery






44. What are the histological findings in the ileum

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45. What causes pancreatic insuff and What does it cause






46. inc cholesterol and/or bilirubin - dec bile salts and gallbladder stasis






47. What are the complications of duodenal PUD






48. What portion of the bowel does sprue effect






49. How does gastrin increase acid secretion?






50. What is one potential precipitating factor for intussusception