Test your basic knowledge |

Subjects : health-sciences, usmle
Instructions:
  • Answer 50 questions in 15 minutes.
  • If you are not ready to take this test, you can study here.
  • Match each statement with the correct term.
  • Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.

This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. Where does type B chronic gastritis occur and What causes it






2. What carcinogens are associated with HCC






3. Progressive dyshphage beginning with solids and moving to liquids and weight loss






4. What is the ddx associated with appendicitis






5. What are the two molecular pathways that lead to CRC






6. Which kind of hemorrhoids are painful and why






7. What does the splenorenal ligament connect - and What does it contain






8. What are additional risk factors for CRC






9. What source of salivary secretion is the most serous and What is the most mucinous






10. Where is the deep inguinal ring relative to the inferior epigastric vessels






11. secretin - source - action - regulation






12. What does K- ras mutation cause






13. What do you treat Wilsons disease with and What is the inheritance






14. occlusion of IVC or hepatic veins






15. What serum markers increase in cholecystitis with bile duct involvement






16. What are the complications of duodenal PUD






17. Where does type A chronic gastritis occur and What causes it






18. What are the tumor markers for pancreatic adenocarcinoma






19. If the abdominal aorta is blocked - How does blood get to the inferior pancreaticduodenal arter






20. What is pancreatic adenocarcinoma associated with






21. How does CRC present in the distal and proximal colon






22. What is the risk with peutz jehgers






23. What is the cause of physiologic neonatal jaundice






24. What kind of digestion is bile needed for






25. Which IBD may or may not have blood diarrhea - and which will always have bloody diarrhea






26. What causes carcinoid syndrome amd What are the symptoms






27. What are causes of extrahepatic biliary obstruction






28. What are esophageal strictures associated with






29. What cells make pepsin - What does it do - and what regulates it






30. GIP - source - action regulation






31. What serum enzyme is elevated in acute pancreatitis and mumps






32. glandular metaplasia - replacement of nonkeratinized (stratified) squamous epithelium with intestinal (columnar) epithelium in the distal esophagus

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33. What cells secrete bicarb - What does it do - and what regulates it






34. What structures feed into the common bile duct






35. HCC is associated with what other conditions






36. People of what decent are associated with celiac sprue and what findings/antibodies are present






37. What is the most common diaphragmatic hernia and What are the two types






38. Which monosaccharides are absorbed by the enterocytes and which transporters carry each






39. Autodigestion of pancreas by pancreatic enzymes






40. What is the most common indication of emergent abdominal surgery in children






41. If trypsin activates more trypsinogen - what kind of feedback loop is established






42. What receptors does gastrin bind on the parietal cell and What does it activate






43. Which IBD is autoimmune and which may be a disordered response to bacteria






44. What do you use to diagnose meckels






45. What does histo show for alpha1 antitrypsin def






46. What does the gastrocolic ligament connect - What is contained - and what larger structure is it a part of...






47. If the hemochromatosis is primary - What is the pattern of inheritance






48. What are the hindgut structures and what supplies their blood and PANS innvervation






49. Where is B12 absorbed






50. What is the classic triad of hemochromatosis

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