Test your basic knowledge |

Subjects : health-sciences, usmle
Instructions:
  • Answer 50 questions in 15 minutes.
  • If you are not ready to take this test, you can study here.
  • Match each statement with the correct term.
  • Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.

This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. What intervention will intervention will relieve portal HTN






2. What kind of salivary gland tumor is painless - moveable mass - bening with high rate of recurrence - most common salivary gland tumor






3. How does brain injury lead to acute gastritis and What is it called






4. Which monosaccharides are absorbed by the enterocytes and which transporters carry each






5. FAP + malignant CNS tumor






6. What is the frequency of basal electric rhythm in the duodenum






7. What is the prognosis of adenocarcinoma






8. What congenital birth defect is associated with Hirschsprung






9. mildly dec UDPGT or dec bilirubin uptake - asymptomatic - elevated uncong bili without over hemolysis


10. What is the ddx associated with appendicitis






11. Where is the pancreatic enzyme concentration highest and what reaction does it catalyze






12. What serum marker increases with primary biliary cirrhosis and what other autoimmune conditions are it associated with






13. In what syndrome is gastric very elevatad - and amino acids are potent gastrin stimulators






14. Which kind of hemorrhoids are painful and why






15. Through which aspect of the inguinal canal does a direct inguinal go






16. Diaphragmatic hernias occur in infants because of defective development of which membrane






17. Transmural esophageal rupture due to violent retching


18. What structures feed into the cystic duct






19. What kind of muscle is in the lower 1/3 of the esophagus






20. What does the gastrocolic ligament connect - What is contained - and what larger structure is it a part of...






21. What is the most important mechanism in gastric acid secretion






22. Scleroderma is associated with what kind of esophageal dysmotility






23. Abuse of what substance leads to acute gastritis






24. What does high flow rate mean






25. What are the histological findings in the duodenum


26. What are esophageal strictures associated with






27. What is the rule of 2s for meckels






28. What causes pancreatic insuff and What does it cause






29. Where does type A chronic gastritis occur and What causes it






30. alcoholic liver disease that requires sustained - long term consumption - with swollen and necrotic hepatocytes with neutrophilic infiltration






31. What is the main symptom if a VIPoma






32. What happens to the short gastics if the splenic artery is blocked






33. What retroperitoneal structure flanks both sides of the pancreas on CT






34. What kind of muscle is in the middle 1/3 of esophagus






35. What does K- ras mutation cause






36. What receptor does histamine bind on the parietal cell and What does it activate






37. In viral hepatitis - which liver enzyme is higher






38. What source of salivary secretion is the most serous and What is the most mucinous






39. Malabsorption syndromes have what common clinical presentation






40. Which glands secrete alkaline mucus to neutralize acid contents entering the duodenum from the stomach and are located in the duodenal submucosa






41. What can fistula between the gallbladder and small intestine create and how can you tell






42. Between what structures do strong anastamoses exist






43. necrosis of intestinal mucosa and possible perforation - usual colon involvement - more common in preemies






44. People of what decent are associated with celiac sprue and what findings/antibodies are present






45. What is the clinical presentation of acute pancreatitis






46. What can hemochromatosis be secondary to...






47. Why are most diverticula considered false






48. What kind of pathways do CCK act on to cause pancreatic secretion






49. What pancreatic enzymes are responsible for fat digestion






50. What are the effects of atropine on parietal cells and G cells