Test your basic knowledge |

USMLE Prep 2

Subjects : health-sciences, usmle
Instructions:
  • Answer 50 questions in 15 minutes.
  • If you are not ready to take this test, you can study here.
  • Match each statement with the correct term.
  • Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.

This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. where are the vegetations on the valves of a libman sacks endocarditis?






2. What is the Na/Ca exchange used for?






3. What is hypospadias caused by?






4. What causes release of myosin head from the actin filament?






5. which opponens muscle does ulnar innervate?






6. What three things can reduce the risk of non hereditary ovarian and endometrial cancer?






7. in essential fructosuria - what enzyme do patients use to metabolize fructose?






8. after triglyceride metabolism - What is the fate of the glycerol? what enzyme is involved?






9. integrin mediated adhesion of cells to ECM (and BM) involves integrin binding to what?






10. which headaches are seen mostly in men - are severe - unilateral - periorbital - episodic (around same time every day) - temporal pain - with lacrimation - nasal congestion and ptosis?






11. What are fenfluramine - phentermine?






12. at 2 years of age - What are the social - fine motor - gross motor and language developments?






13. what pathology is found around the illeo cecal valve and presents in 2 year old children with colicky abdominal pain and currant jelly stools?






14. what clinical findings help distinguish small cell carcinoma?






15. What is the triad seen in pre eclampsia?






16. the rate of blood flow of which two circulations must equal each other at all times?






17. What is capsaicin? Where does it work?






18. What is a side effect of ACE inhibitor that is more worrisome in patients with renal failure?who else is it worrisome in?






19. how will ectopic pregnancy rupture present? What is key history question for diagnosis? what would a uterine biopsy show?






20. What is contraindicated in toxic mega colon?






21. What is achalasia and how would this correlate on the esophageal mannometry?






22. Where does conjugation of bilirubin take place?






23. In What type of nephritis would you see high serum eos count?






24. What are the long term consequences of hydrocephalus?






25. a patient fearing all white coats is a phenomenon of what?






26. is Rifampin ever used as monotherapY? why either way?






27. what has the greatest effect on prognosis when treating c. diptheriae?






28. What is the most important prognostic indicator in patients with malignant melanoma?






29. which congenital hyperbilirubinemia actually presents with serious symptoms? which are less serious/






30. what dictates the resting membrane potential of most cells?






31. What is the fibrinogen level in patient with TTP- HUS? DIC?






32. What is mcc of death pre hospital phase of MI? in hospital phase?






33. there are mucus secreting cells in the bronchioles...






34. What type of mutation does aflatoxin cause? what cancer does this increase for?






35. facial pain and headache in a patient with diabetic ketoacidosis is highly suggestive of what? How do you diagnose? What is a char finding?






36. why should you not use ACE inhibitors with someone who had hereditary angioedema?






37. What is the cause of fixed splitting of S2? why?






38. SIADH patients have normal blood volume but...






39. What type of endocarditis is cytoscopy induced?






40. What does NF- KB do?






41. which trisomy is associated with endocardial cushion defects? What does thsi mean>






42. What are some of the permissive effects of cortisol?






43. in overweight individuals What is thought to contribute to insulin resistance?






44. what happens to PaO2 - % sat - and O2 content in: Anemia - polycythemia - CO poisoning?






45. Where does vasopressin act - on the medullary or cortical segment of collecting tubule?






46. What is the most common congenital adrenal hyperplasia? What does the enzyme convert What to what? and What is the presentation?






47. What is damaged in early syringomelia? later?






48. What is congestive hepatomegaly specific for?






49. what chromosome is c - myc found on?






50. what bursa is affected when on knees like a maid/gardner?