Test your basic knowledge |

USMLE Prep 2

Subjects : health-sciences, usmle
Instructions:
  • Answer 50 questions in 15 minutes.
  • If you are not ready to take this test, you can study here.
  • Match each statement with the correct term.
  • Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.

This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. What can cause aortic regurg? What is the heart sound you hear?






2. What is the most common neurologic complication of VZV reactivation?






3. What is epispadias caused by?






4. which cells produce surfactant? which ones mediate gas exchange?






5. What are the acute effects of corticosteroids on the CBC?






6. which nerve in the lower leg is easily injured and causes foot drop ? What are common causes? From what nerve does it branch off or?






7. in a positively skewed distribution is the mean greater than or equal to the median or the mode?






8. How do you treat gonococcal infection? chlymadia?






9. What is the presentation of sever aortic stenosis?






10. What is 5- HETE and What does it do?






11. What is a keloid?






12. What is the difference between Acyl CoA carboxylase and Acyl CoA dehydrogenase?






13. In what form are mitochondrial DNA? What do they transcribe?






14. Where does 90% of serotonin lie? What is this NT responsible?






15. Where does terminal peptide cleavage of collagen fibrils take place?






16. what clinical findings help distinguish small cell carcinoma?






17. Where does vasopressin act - on the medullary or cortical segment of collecting tubule?






18. What is the best indicator for the severity of mitral stenosis?






19. What is the preferred treatment for DKA?






20. What type of mutation does aflatoxin cause? what cancer does this increase for?






21. where are neurons lost in huntingtons disease? What are two mc presenting symptoms?






22. What does the tuberoinfundibular pathway connect? What is it responsible for?






23. why are beta thal major patients asymptomatic at birth?






24. What are the three presentations of ataxia telangectasia? What does the mutation cause? What is the mode of inheritance?






25. how does eos release MBP to kill protozoa etc?






26. which congenital hyperbilirubinemia actually presents with serious symptoms? which are less serious/






27. what dictates the resting membrane potential of most cells?






28. What is the sole neurologic manifestation of acute rheumatic fever?






29. What is used to compare means? categorical outcomes?






30. Where does vit C act in the hydroxylation of lysine residues for collagen? What is the cofactor required?






31. other than proteinuria - What can cause foamy froathy urine?






32. which congenital adrenal hyperplasia presents with all genotypes as phenotypically female and with salt retention and hypertension?






33. which staphylococci can do mannitol fermaentation?






34. why is glucagon used in beta blocker toxicitiy?






35. what hormone is structurally similar to hCG?






36. What is the neurologic manifestation of ADPKD?






37. what pathology is found around the illeo cecal valve and presents in 2 year old children with colicky abdominal pain and currant jelly stools?






38. What can inhaled anesthetics (like halothane) cause post operatively? what virus does it immitate? What are the presenting symptoms?






39. What does the inferior gluteal nerve innervate? how does damage to this nerve manifest?






40. what happens to PaO2 - % sat - and O2 content in: Anemia - polycythemia - CO poisoning?






41. other than parvo B19 - what else is associated with red cell aplasia?






42. ___________ is liver specific






43. What causes curlings ulcers?






44. SIADH patients have normal blood volume but...






45. What three pathogens cause infectious esophagitis in HIV positive patients?






46. What are ulcers arising in the proximal duodenum in association with severe trauma or burns called?






47. what drugs causes the red man syndrome? how does it occur?






48. Where does lysyl oxidase act? What is the cofactor for that?






49. What is a major risk factor for progression ARDS? What is the pathology seen in ARDS- d/t what?






50. What is diagnostic (and possible therapeutic for intussusception)?