Test your basic knowledge |

USMLE Prep 2

Subjects : health-sciences, usmle
Instructions:
  • Answer 50 questions in 15 minutes.
  • If you are not ready to take this test, you can study here.
  • Match each statement with the correct term.
  • Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.

This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. What are three symptoms in s.typhi?






2. what protein is increased in Crohns disease? What does it do?






3. What is a major risk factor for progression ARDS? What is the pathology seen in ARDS- d/t what?






4. after a thrombus extraction - what serum enzyme shoots up and why?






5. What are the acute effects of corticosteroids on the CBC?






6. What type of vision is myopia? In What type of patients does it improve?






7. name three pathological states that present with large tongues.






8. What are the lab findings in poststreptococcal GN?






9. What is the neurologic manifestation of ADPKD?






10. what composes the superior and inferior borders of the right side of the cardiac silouhette in a CXR? Where is the pulm arter?






11. why does hypothyroidism cause increased CPK levels?






12. What are the two pharmacologic antagonists that offer clear benefit in allergic asthma?






13. IL4 is used for isotypye switching to what?






14. why is there only minor blood pressure increase during exercise if sympathetic activity is high (to increase CO and HR)?






15. What is a cardiac cause of head pounding with exertion and nocturnal palpitations? What can cause this?






16. what increases turbulence and thus causes bruits? (specifically in terms of viscosity and velocity)






17. Where does conjugation of bilirubin take place?






18. what happens to capacitance with age?






19. what receptors do first generation anti histamines block?






20. in the fluid running along the PT - what happens to PAH - inulin - urea - creatinine? how about glucose - aa - bicarb?






21. What is the most important prognostic indicator in patients with malignant melanoma?






22. how long is substance P? What does it do?






23. is Rifampin ever used as monotherapY? why either way?






24. What are the two growth factors associated with angiogenesis?






25. What is acanthosis nigricans associated with?






26. What type of cancer is keratin swirls indicative of ? What is the prognosis of this cancer in the esophagus? What is it associated with?






27. which cells produce surfactant? which ones mediate gas exchange?






28. What is the mc manifestation of CMV in HIV patient? immunocompetent?






29. why does neutrophila occur with corticosteroids?






30. SIADH patients have normal blood volume but...






31. erythema nodosum - elevated ACE - scattered granulomas - arthralgias - hilar lymphadenoapthy are indicative of what?






32. What is congestive hepatomegaly specific for?






33. how can HAV be inactivated?






34. h1 receptor anatagonists are not effective in treatment of asthma only for...






35. in overweight individuals What is thought to contribute to insulin resistance?






36. What are the two mcc of focal brain lesions in HIV positive patients?






37. What is the fibrinogen level in patient with TTP- HUS? DIC?






38. What is a keloid?






39. What are the first line agents used in acute gouty arthritis? why not use colchicine? when would you use glucocorticoids?






40. Which branch of the facial nerve provides taste from ant 2/3 of tongue?






41. Where is aromatase used?






42. What type of endocarditis is cytoscopy induced?






43. How do bradykinin - C3a and C5a cause edema?






44. What are the three presentations of ataxia telangectasia? What does the mutation cause? What is the mode of inheritance?






45. What are fenfluramine - phentermine?






46. What are the three dopaminergic systems and What are they responsible for? disease?






47. what clinical findings help distinguish small cell carcinoma?






48. What is the mc location of brain germinomas?What are the classic symptoms?






49. Which is faster atrial muscle or ventricular muscle?






50. PDAs are often asymptomatic. How do you treat?