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USMLE Prep 2

Subjects : health-sciences, usmle
Instructions:
  • Answer 50 questions in 15 minutes.
  • If you are not ready to take this test, you can study here.
  • Match each statement with the correct term.
  • Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.

This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. why is glucagon used in beta blocker toxicitiy?






2. how will ectopic pregnancy rupture present? What is key history question for diagnosis? what would a uterine biopsy show?






3. why does liver dysfunction cause coagulation disorders?






4. what protein is increased in Crohns disease? What does it do?






5. Where does 90% of serotonin lie? What is this NT responsible?






6. What is the most common location of colonization of all s. aureus types?






7. What does prolonged PT indicated? aPTT? bleeding time?






8. What type of drug is alendronate?






9. what presents congenitally as macroglossia - generalized hypotonia - and an umbilical hernia?






10. do Class IC agents prolong the QT interval?






11. What does Rb protein do? what chrom is it on?






12. Where does glycolsylation occur of alpha procollagen chains occur? disulfide bond formation at the C terminus?






13. What is the immune deficinecy seen in ataxia telangactasia?






14. What is cataplexy and When is it seen?






15. what induces bronchial squamous metaplasia?






16. if there are keratin swirls does that mean well or poorly differentiated?






17. where are the two classical places that the ulnar nerve can be injured?

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18. what murmur is enhanced by decreased blood flow to the heart?






19. When does dysplasia become a carcinoma - in other words When does it nonreversible? What is high grade dysplasia synonymous with?






20. what drug causes aggression - nystagmus - ataxia - slurred speech - exaggerated gait and involuntary movements?






21. lipid filled plaques in which arteries does thigh claudication suggest? difficulty sustaining an erection?






22. What is the mcc of cystitis and and acute pyelonephritis? mcc of UTI in sexually active women?






23. What are the lab findings in poststreptococcal GN?






24. which RPGN is also called pauci immune GN? why?






25. What does phosphoglycerate mutase produce? In what process - instead of what? what cells used this and why?






26. eukaryotes are often polycistronic (multiple genes per mRNA) so ____________ is linked






27. which two drug types can cause orthostatic hypotension (think depression and BPH)?






28. at three years of age What are social - fine motor - gross motor and language developments?






29. non ceruloplasmin deposition - ceruloplasmin is...






30. What is a cell surface marker seen in liver angiosarcoma?






31. Which branch of the facial nerve provides taste from ant 2/3 of tongue?






32. What is a primary HSV 1 infection like?






33. What does NF- KB do?






34. When is an S4 sound normal?






35. Is the uterus enlarged in endometriosis? does it cause dyspareunia?






36. who bleed more DIC or TTP- HUS patients?






37. What are examples of action that decrease venous return to the heart?






38. What is achalasia and how would this correlate on the esophageal mannometry?






39. In What type of nephritis would you see high serum eos count?






40. What is the difference between additive and synergistic?






41. What causes curlings ulcers?






42. Where is aromatase used?






43. when do ghon complexes form - primary or secondary TB?






44. Where is high frequency sound best recepted? low frequency sound? Which is lost more in elderly?






45. What is medullary sponge kidney disease and how does it present? What does it lead to?






46. What are the two growth factors associated with angiogenesis?






47. what color pigmentations are caused by malassezia furfur? when do they become more visible?






48. What is pickwickian syndrome? What are the lab findings?






49. What is the primary histologic finding in patients with eczematous dermatitis?






50. which staphylococci can do mannitol fermaentation?







Sorry!:) No result found.

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