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USMLE Step 1 Immunology

Instructions:
  • Answer 50 questions in 15 minutes.
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  • Match each statement with the correct term.
  • Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.

This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. when can graft versus host disease? What is the result?






2. Which are the only two antiinflammatory cytokines?






3. What is a factor that is a predictor for a bad transplantation?






4. What is MHC I made out of? Where is it found? What does it bind to? What type of antigens does it present?






5. Which is the most abundant antibody in blood?






6. What is the autoantibody for SLE that is nonspecific? Specific?






7. What are the autoantibodies for drug induced lupus?






8. If the alternative pathway is constitively active - how come normal cells don't get attacked with MAC?






9. What is the pathogenesis of IgG AIHA ABO hemolytic disease of a newborn? describe what happens.






10. describe the pathogenesis of delayed type IV hypersensitivity






11. What are the autoantibodies for wegeners granulomatosis?






12. What are the three types of Type III hypersensitivity ? What is the common mechanism between them?






13. ________ regulate the cell mediated response.






14. What lymph node drains the duodenum - jejunum?






15. How is the antigen loaded onto a MHC II?






16. what prevents NK cells from killing normal cells if their default is to kill?






17. To what disease do the autoantibodies to IgG (rheumatoid factor)?






18. Describe complement dependent Type II hypersensitivity. Give an example.






19. What is the pathogenesis of a hypersensitivity reaction?






20. What are the autoantibodies for myasthenia gravis?






21. What part of the lymph node specifically expands during a cellular immune response? when would this occur?






22. IgG...






23. Give an example of someone who could get hyperacute transplant rejection.






24. So antibodies are the effectors for the humoral response. List some of their functions.






25. What are the four steps in phagocytosis? What are the four disease that correspond to each step?






26. IgM can fix complement but...






27. What is the treatment of acute transplant rejection?






28. where are complements produced?






29. what will NK cells do to cells covered in IgG Ab? why?






30. Which antibody mediates immunity to worms? how?






31. What is the main function of interferons?






32. What does IgE do on the surface of the mast cell to induce inflammatory mediator release?






33. What portion of the lymph node is not well developed in DiGeorge Syndrome?






34. What is the clinical use of Muromonab?






35. What type of fenestrations are found in the red pulp of the spleen?






36. What is the presentation of Brutons agammaglobulinemia?






37. What are some sinopulmonary infections?






38. What is oprelevkin? and What is it used for?






39. Name three things that IL 1 does as a cytokine. other than the liver - who secretes IL 1






40. in which immunodef order do you see a lot of pus? no pus?






41. What is the marginal zone of the spleen? what happens there?






42. How do you test for type III hypersensitivity?






43. To what portion of the Antibody do the complements bind?






44. What is filgrastim and sargramostim? and What is it used for?






45. What are the autoantibodies for pemphigus bulgaris?






46. What does it mean if there are igM in the serum at birth?






47. What is recomb gamma interferon used for?






48. What is the common variable immunodeficiency ? How is it different from Brutons?






49. Which disease is associated with DR3?






50. other than eat and bite RBCs what else do Macrophages of spleen do>







Sorry!:) No result found.

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