Test your basic knowledge |

Subject : health-sciences
Instructions:
  • Answer 50 questions in 15 minutes.
  • If you are not ready to take this test, you can study here.
  • Match each statement with the correct term.
  • Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.

This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. What 3 things does insuline def and glucagon excess lead to...






2. What are neurophysins






3. When diagnosing diabetes insipidus - what happens to the urine osm in the water deprivation test and How do you distinguish between central and nephrogenic


4. What is the effect of CRH on ACTH






5. most common thyroid cancer - ground glass nuclei - psammona bodies - nuclear grooves - prognosis and risk






6. What is the tx for diabetes inspidus - central and nephrogenic






7. What are the common causes of dec Mg






8. in the congenital bilateral adrenal hyperplasias which enzyme deficiency is characterized by dec in sex hormones - dec in cortisol - and inc in mineralcorticoids - enzyme and sx






9. What is the next layer in from ZG - What is its primary control - What does it secrete






10. Which kind of cells don't need insulin to take up glucose






11. where do you see GLUT 4 transporter






12. In which DM is there a stronger genetic association






13. The adrenal cortex derives From What embryonic tissue






14. What is the effect of GHRH on GH






15. Which ketone bodies are more common in DKA






16. What is the TX for carcinoid syndrome






17. What is the tx for gigantism/acromegaly






18. Where is low serum phosphorus sensed to maintain phosphorus homeostasis - and what substance is key for increasing serum levels of phosphorus






19. What causes primary hyperaldosteronism (Conn's syndrome) and What are the lab findings






20. nonenzymatic glycosylation leads to what 3 catageories of defects






21. What is the most common tumor of the adrenal medulla in children






22. Why do congential adrenal enzyme deficiences end up with hyperplasia and not hypoplasia






23. What is the most common tumor of the adrenal medulla in adults






24. What HLA association is with hashimotos and what kind of autoantibodies






25. How many cortical divisions are involved in primary adrenal insuff






26. How does the body respond to SIADH and What does that cause






27. What does NEG cause in small vessels






28. What is the general chronic manifestation of DM






29. What kind of ligands bind intrinsice tyrosine kinase






30. What happens in the dexamethasone suppresion test in a health individual






31. What do the three endocrine cells of the pancreas secrete






32. What is the rule of 1/3s in carcinoid syndrome






33. What are the 3 P's of MEN type 1 (Werners)






34. When is the onset of DM1 vs DM2 causing what kind of DM






35. What does the brain use for fuel in starvation






36. iodine containing hormones that control the body's metabolic rate






37. in congenital bilateral adrenal hyperplasia - what enzyme def is most common and associate with dec cortisol - dec mineralcorticoids - inc sex hormones - enzyme and sx






38. What happens to women with dec SHBG






39. Where do you see the GLUT 1 transporter






40. What is the fxn of prolactin






41. What does adrenal insufficiency cause






42. All three effects of hyperinsulinemia lead to which two clinical features






43. The adrenal medulla derives From What embryonic tissue






44. Neuroblastoma does not come with...






45. Which substances bind receptor associated tyrosine kinase via the JAK/STAT pathway






46. What drugs cause shrinkage of prolactinoma






47. What occurs to beta cells in DM1 vs DM2






48. Recurrent diarrhea - cutaneous flushing - asthatic wheezing - right side valvular disease






49. Do pts with neuroblastoma develop HTN






50. focal patches of hyperfxning follicular cells working independently of TSH