Test your basic knowledge |

Subject : health-sciences
Instructions:
  • Answer 50 questions in 15 minutes.
  • If you are not ready to take this test, you can study here.
  • Match each statement with the correct term.
  • Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.

This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. What is the algorithim for acidosis/alkalosis






2. Where does renal cell carcinoma originate and What do the cells look like






3. The fenestrated capillary endothelium constitutes what portion of the barrier






4. What is transporter in the thick ascneding loop of Henle indirectly induces the paracellular reabsorption of Mg and Ca






5. What happens to the urine in the descending limb






6. What happens to urine in the ascending limb






7. What is renal osteodystrophy






8. What are the features of membranous GN (diffuse membranous glomerulopathy) on LM - EM and IF






9. What does US show with medullary cystic disease






10. What effect does ANP have on GFR






11. What is the effect of aldosterone in principal cells






12. How is plasma volume measured






13. What is the BUN/Cr ratio in prerenal azotemia and why?






14. What is the henderson hasselbalch equation






15. What are the main causes of membranous GN






16. What is the formula for clearance of a substance per unit time






17. what happens to pH - PCO2 - and bicarb in respiratory acidosis






18. What needs to happen for postrenal obstruction to creat ARF






19. What enzyme in the proximal tubule allows the conversion of carbonic acid to water and C02






20. in TCC - What does painelss hematuria suggest






21. What substance is secreted from the kidney in response to hypoxia - and what cells do they come from






22. What happens in the early distal convoluted tubule and What does that do to the urine






23. What does LM - EM - IF show in diffuse proliferative GN






24. Where is potassium conc. Highest? Intra or extra






25. When is glucose reabsorbed and with What transporter






26. In pts with ammonium magnesium phophate stones - What can be the nidus for UTI and what worsens it






27. In miminal change disease - who gets it - What are the triggers and What is their selective loss of?






28. hypoaldosteronism or lack of collecting tubule response to aldosteron






29. What does thyroidization of the kidney result in






30. Why does Na conc nearly match Osm






31. In renal failure - what happens to potassium






32. When is TF/P = 1






33. membranous






34. What receptor responds to inc sympathetic discharge leading to renin secretion from JG cells






35. How can NSAIDs cause acute renal failure






36. What effect does inc plasma protein concentration have on RPF - GFR - and FF






37. What circumstances causes ADH secretion






38. What is the effect of AT II on the proximal tubule - and what kind of alkolosis does this allow for






39. dense deposits on EM - type and association






40. How do you interpret creatinine clearance






41. most common cause of acute renal faiure in hospital - self reversible but fatal - dz and associations - key finding






42. Where is ACE made and What are 2 of its fxns






43. What enzyme allows for conversion of 25- OH vit D to 1 -25 (OH)2 vit D






44. What does ADH do in the collecting tubule






45. What do macula densa cells sense






46. Defect in collecting ducts ability to excrete H+






47. What is the prognosis of RPGN






48. What do you see on LM and IF with rapidly progressive GN






49. Which cells sense decreases in Na delivery






50. When is TF/P <1