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Test your basic knowledge |
USMLE Cardiovascular Physiology
Start Test
Study First
Subjects
:
health-sciences
,
usmle
Instructions:
Answer 50 questions in 15 minutes.
If you are not ready to take this test, you can
study here
.
Match each statement with the correct term.
Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.
This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. 1) increased capillary pressure - CHF 2) decreased plasma proteins - nephrotic syndrome - liver failure 3) increased capillary permeability - toxins - burns - infections 4) increased interstital osmotic pressure - lymphatic obstruction
Wide S2 splitting
Tricuspid Regurgitation
Common causes of edema
Aortic regurgitation
2. Pulls fluid into capillary
A fib on EKG
Supplies blood to the base of the left ventricle
Common causes of edema
Plasma colloid osmotic pressure
3. Associated with atrial septal defect (ASD) - allowing a left to right shunt that increases flow through pulmonic valve and delays closure
Aortic regurgitation
Phase 4 of ventricular AP
Fixed S2 splitting
Aortic Stenosis
4. = (capillary pressure - interstital fluid pressure) - (plasma oncotic pressure - interstital fluid oncotic pressure)
Phase 3 of nodal AP
Net filtration pressure
T wave inversion on EKG
Mitral Regurgitation
5. Resting potential due to high K permeability
Mitral Regurgitation
Resistance is inversely proportional to...
Phase 4 of ventricular AP
Normal S2 splitting
6. Inspiration delays pulmonic valve closure - which increases S2 splitting
Auscultation location of pulmonic area
Mobitz type I (Wenckebach) 2nd degree AV block
Phase 0 of nodal AP
Effect of inspiration on S2 splitting
7. Radius to the 4th power
Patent ductus arteriosus
Resistance is inversely proportional to...
S4
Phase 3 of nodal AP
8. Caused by hypokalemia or bradycardia
Net filtration pressure
U wave on EKG
T wave inversion on EKG
Phase 1 of ventricular AP
9. High pitched 'blowing' diastolic murmur - can decrease intensity of murmur with vasodilation
Aortic regurgitation
Lyme disease can cause why type of AV block?
Plasma colloid osmotic pressure
Fixed S2 splitting
10. Plateau due to Ca influx balancing K efflux Myocyte contraction
Phase 2 of ventricular AP
Mean arterial pressure (MAP) =
Mitral stenosis
Fixed S2 splitting
11. Holosystolic 'blowing murmur' loudest at left sternal border near 4th rib - & is enhanced by inspiration (increased RA return)
Wolff - Parkinson - White Syndrome
Tricuspid Regurgitation
T wave inversion on EKG
Interstital hydrostatic pressure (Pi)
12. Mitral & tripcuspid valve closure
Lyme disease can cause why type of AV block?
A flutter on EKG
S1
Torsades de Pointes
13. Cardiac output (CO) x total peripheral resistance (TPR) - or 2/3 diastolic pressure + 1/3 systolic pressure
Mean arterial pressure (MAP) =
Normal S2 splitting
Mobitz type II 2nd degree AV block
Mobitz type I (Wenckebach) 2nd degree AV block
14. Early diastole rapid filling sound heard with dilated ventricle (normal in children & pregnant women)
Aortic Stenosis
S3
Auscultation location of mitral area
Resistance is inversely proportional to...
15. Progressive lengthening of PR interval until dropped QRS complex
Mobitz type I (Wenckebach) 2nd degree AV block
Phase 3 of ventricular AP
Auscultation location of aortic area
3rd degree complete AV block
16. Late systolic crescendo murmur (loudest at S2) following a midsystolic click - enhanced by squatting (increased TPR)
Mitral prolapse
Auscultation location of mitral area
Fick principle
Supplies blood to the base of the left ventricle
17. 3rd degree
Mobitz type II 2nd degree AV block
Lyme disease can cause why type of AV block?
Mitral prolapse
Net filtration pressure
18. 2nd intercostal space along right sternal border
Aortic regurgitation
Normal S2 splitting
Auscultation location of aortic area
Phase 3 of ventricular AP
19. Associated with pulmonic stenosis or RBBB (delayed RV emptying)
U wave on EKG
Ventricular septal defect
Interstitial fluid colloid osmotic pressure
Wide S2 splitting
20. 5th intercostal space at left midclavicular line
Auscultation location of mitral area
Mitral stenosis
1st degree AV block on EKG
Plasma colloid osmotic pressure
21. Aortic & pulmonic valve closure
Phase 4 of ventricular AP
Wide S2 splitting
Phase 3 of ventricular AP
S2
22. 2nd intercostal space along left sternal border
Auscultation location of pulmonic area
Mobitz type I (Wenckebach) 2nd degree AV block
T wave inversion on EKG
Phase 2 of nodal AP
23. Holosystolic 'blowing murmur' loudest at left 5th intercostal space midclavicular line - & is enhanced by expiration (increased LA return) & squatting (increased TPR)
Phase 0 of nodal AP
Mitral prolapse
Aortic Stenosis
Mitral Regurgitation
24. Prolonged PR interval
Normal S2 splitting
1st degree AV block on EKG
Interstitial fluid colloid osmotic pressure
T wave inversion on EKG
25. Absent (no plateau in nodal tissue)
Phase 2 of nodal AP
Fixed S2 splitting
Net filtration pressure
S2
26. CO = rate of O2 consumption / (arterial O2 content - venous arterial O2 content)
Fick principle
Aortic Stenosis
1st degree AV block on EKG
Normal S2 splitting
27. Pull fluid out of capillary
Effect of inspiration on S2 splitting
Interstitial fluid colloid osmotic pressure
Auscultation location of aortic area
Phase 4 of nodal AP
28. Pulmonic valve closure before aortic valve closure associated with aortic stenosis or LBBB (delayed left ventrical emptying)
Paradoxical S2 splitting
Aortic regurgitation
Fixed S2 splitting
1st degree AV block on EKG
29. Pushes fluid out of capillary
S3
Capillary hydrostatic pressure (Pc)
Resistance is directly proportional to...
Auscultation location of aortic area
30. Initial repolarization due to inactivation of Na channels & opening of voltaged - gated K channels
Phase 4 of nodal AP
Phase 1 of ventricular AP
Wolff - Parkinson - White Syndrome
1st degree AV block on EKG
31. Indicates recent MI
Patent ductus arteriosus
Aortic Stenosis
T wave inversion on EKG
Lyme disease can cause why type of AV block?
32. Slow diastolic depolarization with Na conductance that accounts for automaticity of SA/AV nodes (slope determines HR)
Phase 4 of nodal AP
Mitral prolapse
Normal S2 splitting
Mean arterial pressure (MAP) =
33. Rapid back - to - back atrial depolarization -> 'sawtooth' appearance
Net filtration pressure
Lyme disease can cause why type of AV block?
A flutter on EKG
S4
34. Crescendo - decrescendo systolic ejection murmur following an ejection click. Associated with weak pulses & syncope.
Phase 1 of ventricular AP
Effect of inspiration on S2 splitting
Auscultation location of pulmonic area
Aortic Stenosis
35. Holosystolic 'harsh' murmur that is loudest at left sternal border near 4th rib
Ventricular septal defect
S3
Effect of inspiration on S2 splitting
Patent ductus arteriosus
36. Posterior descending artery (80% from RCA - 20% from CFX via LCA)
Phase 3 of ventricular AP
Mean arterial pressure (MAP) =
Supplies blood to the base of the left ventricle
S1
37. Continuous machine - like murmur that is loudest at S2
Mitral stenosis
Patent ductus arteriosus
Phase 0 of ventricular AP
Tricuspid Regurgitation
38. Dropped QRS complexes not preceded by change in PR interval (can be 2:1 - 3:1 - etc)
1st degree AV block on EKG
Resistance is inversely proportional to...
Mobitz type II 2nd degree AV block
Phase 4 of nodal AP
39. Aortic valve closure slightly before pulmonic valve closure at end of systolic ejection
Supplies blood to the base of the left ventricle
Patent ductus arteriosus
Normal S2 splitting
Phase 2 of nodal AP
40. Upstroke due to opening of Ca channels (Nodal tissues lack fast Na channels which allows for a prolonged AV node transmission from atria to ventricles)
Phase 3 of nodal AP
A flutter on EKG
Torsades de Pointes
Phase 0 of nodal AP
41. Repolarization due to inactivation of Ca channels & activation of K channels
Phase 3 of nodal AP
Normal S2 splitting
A fib on EKG
Effect of inspiration on S2 splitting
42. Blood viscosity (increased in polycythemia - hyperproteinemia - & hereditary spherocytosis)
Phase 0 of nodal AP
Resistance is directly proportional to...
Phase 0 of ventricular AP
S4
43. V Tach with shifting sinusoidal waveforms on EKG - can progess to V fib - Predisposed by QT prolongation
Torsades de Pointes
S4
Tricuspid Regurgitation
Auscultation location of aortic area
44. 'Irregularly irregular' - No discrete P waves and irregularly spaced QRS complexes
A fib on EKG
Capillary hydrostatic pressure (Pc)
S1
Mitral prolapse
45. Late diastole 'atrial kick' sound against increased pressure from ventricular hypertrophy
S4
Mitral stenosis
Mean arterial pressure (MAP) =
Common causes of edema
46. Atria & ventricles beat independent of each other - no relation between P waves & QRS complexes
S4
S1
Auscultation location of tricuspid area
3rd degree complete AV block
47. 4th rib at left sternal border
Auscultation location of tricuspid area
Phase 4 of ventricular AP
Aortic Stenosis
Paradoxical S2 splitting
48. Ventricular pre - excitation caused by an accessory conduction pathway that bypasses the AV node. Delta wave on EKG. Can lead to SVT.
Patent ductus arteriosus
Resistance is inversely proportional to...
Wolff - Parkinson - White Syndrome
S2
49. Rapid repolarization due to massive K efflux & closure of Ca channels
Auscultation location of tricuspid area
Mobitz type I (Wenckebach) 2nd degree AV block
Phase 3 of ventricular AP
Aortic Stenosis
50. Rapid upstroke due to voltage - gated Na channel opening
Phase 0 of ventricular AP
Plasma colloid osmotic pressure
Aortic regurgitation
1st degree AV block on EKG