SUBJECTS
|
BROWSE
|
CAREER CENTER
|
POPULAR
|
JOIN
|
LOGIN
Business Skills
|
Soft Skills
|
Basic Literacy
|
Certifications
About
|
Help
|
Privacy
|
Terms
|
Email
Search
Test your basic knowledge |
USMLE Cardiovascular Physiology
Start Test
Study First
Subjects
:
health-sciences
,
usmle
Instructions:
Answer 50 questions in 15 minutes.
If you are not ready to take this test, you can
study here
.
Match each statement with the correct term.
Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.
This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. Upstroke due to opening of Ca channels (Nodal tissues lack fast Na channels which allows for a prolonged AV node transmission from atria to ventricles)
Auscultation location of pulmonic area
Paradoxical S2 splitting
Phase 0 of nodal AP
Common causes of edema
2. V Tach with shifting sinusoidal waveforms on EKG - can progess to V fib - Predisposed by QT prolongation
Mean arterial pressure (MAP) =
Torsades de Pointes
Ventricular septal defect
1st degree AV block on EKG
3. Pushes fluid into capillary
Paradoxical S2 splitting
Interstital hydrostatic pressure (Pi)
Fick principle
Auscultation location of aortic area
4. Dropped QRS complexes not preceded by change in PR interval (can be 2:1 - 3:1 - etc)
Net filtration pressure
Mobitz type II 2nd degree AV block
3rd degree complete AV block
Tricuspid Regurgitation
5. Late diastole 'atrial kick' sound against increased pressure from ventricular hypertrophy
Normal S2 splitting
S4
Wolff - Parkinson - White Syndrome
S3
6. Absent (no plateau in nodal tissue)
Mobitz type II 2nd degree AV block
Lyme disease can cause why type of AV block?
T wave inversion on EKG
Phase 2 of nodal AP
7. Initial repolarization due to inactivation of Na channels & opening of voltaged - gated K channels
Phase 1 of ventricular AP
Normal S2 splitting
Interstital hydrostatic pressure (Pi)
S3
8. Slow diastolic depolarization with Na conductance that accounts for automaticity of SA/AV nodes (slope determines HR)
Phase 0 of nodal AP
Phase 4 of nodal AP
Normal S2 splitting
Lyme disease can cause why type of AV block?
9. 1) increased capillary pressure - CHF 2) decreased plasma proteins - nephrotic syndrome - liver failure 3) increased capillary permeability - toxins - burns - infections 4) increased interstital osmotic pressure - lymphatic obstruction
Lyme disease can cause why type of AV block?
Common causes of edema
Fick principle
Aortic Stenosis
10. Aortic & pulmonic valve closure
T wave inversion on EKG
Normal S2 splitting
S2
Mitral prolapse
11. Progressive lengthening of PR interval until dropped QRS complex
Mobitz type I (Wenckebach) 2nd degree AV block
Tricuspid Regurgitation
Phase 2 of ventricular AP
1st degree AV block on EKG
12. Crescendo - decrescendo systolic ejection murmur following an ejection click. Associated with weak pulses & syncope.
Aortic Stenosis
A flutter on EKG
Mobitz type II 2nd degree AV block
Aortic regurgitation
13. Plateau due to Ca influx balancing K efflux Myocyte contraction
Mitral prolapse
Wolff - Parkinson - White Syndrome
Phase 2 of ventricular AP
Phase 1 of ventricular AP
14. 2nd intercostal space along left sternal border
Resistance is directly proportional to...
Auscultation location of pulmonic area
Plasma colloid osmotic pressure
Interstitial fluid colloid osmotic pressure
15. Ventricular pre - excitation caused by an accessory conduction pathway that bypasses the AV node. Delta wave on EKG. Can lead to SVT.
Phase 0 of ventricular AP
Paradoxical S2 splitting
Wolff - Parkinson - White Syndrome
S3
16. Aortic valve closure slightly before pulmonic valve closure at end of systolic ejection
U wave on EKG
S2
Paradoxical S2 splitting
Normal S2 splitting
17. Continuous machine - like murmur that is loudest at S2
Ventricular septal defect
Mobitz type II 2nd degree AV block
Patent ductus arteriosus
Net filtration pressure
18. Atria & ventricles beat independent of each other - no relation between P waves & QRS complexes
Wide S2 splitting
3rd degree complete AV block
U wave on EKG
Plasma colloid osmotic pressure
19. = (capillary pressure - interstital fluid pressure) - (plasma oncotic pressure - interstital fluid oncotic pressure)
Torsades de Pointes
Supplies blood to the base of the left ventricle
Interstitial fluid colloid osmotic pressure
Net filtration pressure
20. Mitral & tripcuspid valve closure
S1
Phase 0 of nodal AP
Tricuspid Regurgitation
Supplies blood to the base of the left ventricle
21. Pulmonic valve closure before aortic valve closure associated with aortic stenosis or LBBB (delayed left ventrical emptying)
A flutter on EKG
T wave inversion on EKG
Aortic Stenosis
Paradoxical S2 splitting
22. Repolarization due to inactivation of Ca channels & activation of K channels
Phase 1 of ventricular AP
Phase 4 of nodal AP
Phase 3 of nodal AP
Auscultation location of tricuspid area
23. Rapid upstroke due to voltage - gated Na channel opening
S4
Auscultation location of mitral area
Net filtration pressure
Phase 0 of ventricular AP
24. CO = rate of O2 consumption / (arterial O2 content - venous arterial O2 content)
Normal S2 splitting
Torsades de Pointes
Mean arterial pressure (MAP) =
Fick principle
25. Inspiration delays pulmonic valve closure - which increases S2 splitting
Ventricular septal defect
Effect of inspiration on S2 splitting
Interstital hydrostatic pressure (Pi)
Mitral stenosis
26. Pulls fluid into capillary
Phase 0 of nodal AP
Auscultation location of pulmonic area
Phase 1 of ventricular AP
Plasma colloid osmotic pressure
27. 'Irregularly irregular' - No discrete P waves and irregularly spaced QRS complexes
A fib on EKG
A flutter on EKG
S1
Phase 2 of ventricular AP
28. Caused by hypokalemia or bradycardia
Wide S2 splitting
Patent ductus arteriosus
U wave on EKG
S2
29. Pull fluid out of capillary
Mobitz type II 2nd degree AV block
Auscultation location of aortic area
Effect of inspiration on S2 splitting
Interstitial fluid colloid osmotic pressure
30. Holosystolic 'blowing murmur' loudest at left 5th intercostal space midclavicular line - & is enhanced by expiration (increased LA return) & squatting (increased TPR)
Torsades de Pointes
Mitral Regurgitation
3rd degree complete AV block
Normal S2 splitting
31. Indicates recent MI
T wave inversion on EKG
Lyme disease can cause why type of AV block?
Patent ductus arteriosus
Ventricular septal defect
32. Associated with pulmonic stenosis or RBBB (delayed RV emptying)
Wide S2 splitting
Resistance is directly proportional to...
Phase 4 of ventricular AP
Resistance is inversely proportional to...
33. Late systolic crescendo murmur (loudest at S2) following a midsystolic click - enhanced by squatting (increased TPR)
Mitral prolapse
Phase 0 of ventricular AP
Lyme disease can cause why type of AV block?
Tricuspid Regurgitation
34. Radius to the 4th power
Ventricular septal defect
Resistance is inversely proportional to...
Mobitz type II 2nd degree AV block
Common causes of edema
35. Prolonged PR interval
Mitral Regurgitation
Aortic regurgitation
Auscultation location of aortic area
1st degree AV block on EKG
36. 3rd degree
3rd degree complete AV block
Lyme disease can cause why type of AV block?
Phase 0 of nodal AP
A fib on EKG
37. High pitched 'blowing' diastolic murmur - can decrease intensity of murmur with vasodilation
Mitral stenosis
Aortic regurgitation
Phase 3 of ventricular AP
S3
38. Posterior descending artery (80% from RCA - 20% from CFX via LCA)
Ventricular septal defect
Plasma colloid osmotic pressure
Mobitz type I (Wenckebach) 2nd degree AV block
Supplies blood to the base of the left ventricle
39. Holosystolic 'blowing murmur' loudest at left sternal border near 4th rib - & is enhanced by inspiration (increased RA return)
Phase 3 of nodal AP
3rd degree complete AV block
Tricuspid Regurgitation
Phase 0 of nodal AP
40. 2nd intercostal space along right sternal border
Mobitz type II 2nd degree AV block
Interstitial fluid colloid osmotic pressure
Auscultation location of aortic area
Phase 2 of ventricular AP
41. Pushes fluid out of capillary
S4
Plasma colloid osmotic pressure
Tricuspid Regurgitation
Capillary hydrostatic pressure (Pc)
42. Rapid back - to - back atrial depolarization -> 'sawtooth' appearance
A flutter on EKG
S3
U wave on EKG
Aortic regurgitation
43. Holosystolic 'harsh' murmur that is loudest at left sternal border near 4th rib
Auscultation location of pulmonic area
Torsades de Pointes
Mean arterial pressure (MAP) =
Ventricular septal defect
44. Associated with atrial septal defect (ASD) - allowing a left to right shunt that increases flow through pulmonic valve and delays closure
Paradoxical S2 splitting
Common causes of edema
Lyme disease can cause why type of AV block?
Fixed S2 splitting
45. Blood viscosity (increased in polycythemia - hyperproteinemia - & hereditary spherocytosis)
Phase 3 of ventricular AP
Resistance is directly proportional to...
Phase 3 of nodal AP
Tricuspid Regurgitation
46. Cardiac output (CO) x total peripheral resistance (TPR) - or 2/3 diastolic pressure + 1/3 systolic pressure
Phase 4 of ventricular AP
Mean arterial pressure (MAP) =
S1
Net filtration pressure
47. Delayed 'rumbling' late diastolic murmur following an opening snap
Mitral stenosis
Phase 0 of nodal AP
A flutter on EKG
Mean arterial pressure (MAP) =
48. Early diastole rapid filling sound heard with dilated ventricle (normal in children & pregnant women)
Fixed S2 splitting
A fib on EKG
S3
Auscultation location of tricuspid area
49. Rapid repolarization due to massive K efflux & closure of Ca channels
Supplies blood to the base of the left ventricle
S1
Plasma colloid osmotic pressure
Phase 3 of ventricular AP
50. 5th intercostal space at left midclavicular line
Auscultation location of mitral area
Aortic regurgitation
Torsades de Pointes
Supplies blood to the base of the left ventricle