SUBJECTS
|
BROWSE
|
CAREER CENTER
|
POPULAR
|
JOIN
|
LOGIN
Business Skills
|
Soft Skills
|
Basic Literacy
|
Certifications
About
|
Help
|
Privacy
|
Terms
|
Email
Search
Test your basic knowledge |
USMLE Cardiovascular Physiology
Start Test
Study First
Subjects
:
health-sciences
,
usmle
Instructions:
Answer 50 questions in 15 minutes.
If you are not ready to take this test, you can
study here
.
Match each statement with the correct term.
Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.
This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. Atria & ventricles beat independent of each other - no relation between P waves & QRS complexes
3rd degree complete AV block
Phase 4 of ventricular AP
Mobitz type II 2nd degree AV block
Paradoxical S2 splitting
2. Rapid upstroke due to voltage - gated Na channel opening
Phase 2 of nodal AP
Phase 0 of ventricular AP
Supplies blood to the base of the left ventricle
T wave inversion on EKG
3. Absent (no plateau in nodal tissue)
Phase 2 of nodal AP
S4
Fick principle
3rd degree complete AV block
4. Rapid back - to - back atrial depolarization -> 'sawtooth' appearance
A flutter on EKG
Ventricular septal defect
Aortic regurgitation
Mitral prolapse
5. Cardiac output (CO) x total peripheral resistance (TPR) - or 2/3 diastolic pressure + 1/3 systolic pressure
Lyme disease can cause why type of AV block?
Phase 3 of ventricular AP
S3
Mean arterial pressure (MAP) =
6. Aortic valve closure slightly before pulmonic valve closure at end of systolic ejection
Resistance is inversely proportional to...
Effect of inspiration on S2 splitting
Phase 2 of ventricular AP
Normal S2 splitting
7. Pulmonic valve closure before aortic valve closure associated with aortic stenosis or LBBB (delayed left ventrical emptying)
Phase 2 of nodal AP
3rd degree complete AV block
T wave inversion on EKG
Paradoxical S2 splitting
8. V Tach with shifting sinusoidal waveforms on EKG - can progess to V fib - Predisposed by QT prolongation
Normal S2 splitting
Torsades de Pointes
Tricuspid Regurgitation
Net filtration pressure
9. Indicates recent MI
Phase 4 of nodal AP
Patent ductus arteriosus
Mitral prolapse
T wave inversion on EKG
10. Dropped QRS complexes not preceded by change in PR interval (can be 2:1 - 3:1 - etc)
Phase 4 of nodal AP
S2
Mobitz type II 2nd degree AV block
S3
11. Upstroke due to opening of Ca channels (Nodal tissues lack fast Na channels which allows for a prolonged AV node transmission from atria to ventricles)
U wave on EKG
Phase 0 of nodal AP
Phase 3 of ventricular AP
Phase 2 of ventricular AP
12. Pulls fluid into capillary
T wave inversion on EKG
Phase 2 of nodal AP
Phase 3 of ventricular AP
Plasma colloid osmotic pressure
13. Slow diastolic depolarization with Na conductance that accounts for automaticity of SA/AV nodes (slope determines HR)
Phase 4 of nodal AP
Patent ductus arteriosus
Normal S2 splitting
Phase 3 of nodal AP
14. 4th rib at left sternal border
Supplies blood to the base of the left ventricle
T wave inversion on EKG
Mobitz type I (Wenckebach) 2nd degree AV block
Auscultation location of tricuspid area
15. Late diastole 'atrial kick' sound against increased pressure from ventricular hypertrophy
Phase 3 of nodal AP
U wave on EKG
Normal S2 splitting
S4
16. 2nd intercostal space along left sternal border
Aortic regurgitation
Phase 4 of ventricular AP
Wolff - Parkinson - White Syndrome
Auscultation location of pulmonic area
17. Late systolic crescendo murmur (loudest at S2) following a midsystolic click - enhanced by squatting (increased TPR)
Phase 4 of ventricular AP
Phase 1 of ventricular AP
Mitral prolapse
Aortic regurgitation
18. Rapid repolarization due to massive K efflux & closure of Ca channels
S3
Effect of inspiration on S2 splitting
Mobitz type I (Wenckebach) 2nd degree AV block
Phase 3 of ventricular AP
19. CO = rate of O2 consumption / (arterial O2 content - venous arterial O2 content)
S2
Wolff - Parkinson - White Syndrome
Phase 0 of ventricular AP
Fick principle
20. Initial repolarization due to inactivation of Na channels & opening of voltaged - gated K channels
Auscultation location of pulmonic area
Phase 4 of ventricular AP
Capillary hydrostatic pressure (Pc)
Phase 1 of ventricular AP
21. Pushes fluid out of capillary
Wolff - Parkinson - White Syndrome
Phase 4 of ventricular AP
Capillary hydrostatic pressure (Pc)
Common causes of edema
22. High pitched 'blowing' diastolic murmur - can decrease intensity of murmur with vasodilation
Interstitial fluid colloid osmotic pressure
Net filtration pressure
Mean arterial pressure (MAP) =
Aortic regurgitation
23. Blood viscosity (increased in polycythemia - hyperproteinemia - & hereditary spherocytosis)
Aortic Stenosis
Auscultation location of tricuspid area
Interstital hydrostatic pressure (Pi)
Resistance is directly proportional to...
24. Inspiration delays pulmonic valve closure - which increases S2 splitting
Wide S2 splitting
Resistance is directly proportional to...
Effect of inspiration on S2 splitting
Phase 3 of nodal AP
25. Holosystolic 'blowing murmur' loudest at left sternal border near 4th rib - & is enhanced by inspiration (increased RA return)
Tricuspid Regurgitation
Patent ductus arteriosus
Phase 3 of ventricular AP
Mitral stenosis
26. Holosystolic 'harsh' murmur that is loudest at left sternal border near 4th rib
T wave inversion on EKG
Normal S2 splitting
Ventricular septal defect
Mobitz type I (Wenckebach) 2nd degree AV block
27. 5th intercostal space at left midclavicular line
Auscultation location of mitral area
Phase 4 of nodal AP
Lyme disease can cause why type of AV block?
Normal S2 splitting
28. Pull fluid out of capillary
T wave inversion on EKG
Torsades de Pointes
Interstitial fluid colloid osmotic pressure
Tricuspid Regurgitation
29. Continuous machine - like murmur that is loudest at S2
Mitral Regurgitation
Phase 2 of ventricular AP
Patent ductus arteriosus
Interstital hydrostatic pressure (Pi)
30. Prolonged PR interval
S3
1st degree AV block on EKG
Phase 2 of ventricular AP
T wave inversion on EKG
31. Progressive lengthening of PR interval until dropped QRS complex
Mobitz type I (Wenckebach) 2nd degree AV block
Interstital hydrostatic pressure (Pi)
Mitral stenosis
Normal S2 splitting
32. Posterior descending artery (80% from RCA - 20% from CFX via LCA)
Paradoxical S2 splitting
Supplies blood to the base of the left ventricle
Phase 2 of ventricular AP
Wolff - Parkinson - White Syndrome
33. Resting potential due to high K permeability
Phase 4 of ventricular AP
U wave on EKG
Phase 2 of nodal AP
Mitral Regurgitation
34. Radius to the 4th power
Phase 0 of ventricular AP
Mobitz type I (Wenckebach) 2nd degree AV block
Resistance is inversely proportional to...
Mitral stenosis
35. Mitral & tripcuspid valve closure
S2
Plasma colloid osmotic pressure
S1
Phase 0 of ventricular AP
36. Repolarization due to inactivation of Ca channels & activation of K channels
Phase 3 of nodal AP
Mobitz type II 2nd degree AV block
Net filtration pressure
Tricuspid Regurgitation
37. Plateau due to Ca influx balancing K efflux Myocyte contraction
Phase 4 of nodal AP
Phase 0 of nodal AP
Phase 2 of nodal AP
Phase 2 of ventricular AP
38. 3rd degree
Supplies blood to the base of the left ventricle
Lyme disease can cause why type of AV block?
Wide S2 splitting
Patent ductus arteriosus
39. Holosystolic 'blowing murmur' loudest at left 5th intercostal space midclavicular line - & is enhanced by expiration (increased LA return) & squatting (increased TPR)
Mobitz type I (Wenckebach) 2nd degree AV block
U wave on EKG
Mitral Regurgitation
Resistance is inversely proportional to...
40. 2nd intercostal space along right sternal border
U wave on EKG
S2
Paradoxical S2 splitting
Auscultation location of aortic area
41. 1) increased capillary pressure - CHF 2) decreased plasma proteins - nephrotic syndrome - liver failure 3) increased capillary permeability - toxins - burns - infections 4) increased interstital osmotic pressure - lymphatic obstruction
Common causes of edema
Phase 3 of ventricular AP
Interstitial fluid colloid osmotic pressure
Resistance is inversely proportional to...
42. Associated with pulmonic stenosis or RBBB (delayed RV emptying)
Fixed S2 splitting
Phase 0 of ventricular AP
Wide S2 splitting
Phase 2 of nodal AP
43. Early diastole rapid filling sound heard with dilated ventricle (normal in children & pregnant women)
Mobitz type I (Wenckebach) 2nd degree AV block
Phase 1 of ventricular AP
S3
T wave inversion on EKG
44. Ventricular pre - excitation caused by an accessory conduction pathway that bypasses the AV node. Delta wave on EKG. Can lead to SVT.
Auscultation location of tricuspid area
Wolff - Parkinson - White Syndrome
Phase 0 of ventricular AP
Phase 1 of ventricular AP
45. = (capillary pressure - interstital fluid pressure) - (plasma oncotic pressure - interstital fluid oncotic pressure)
Auscultation location of pulmonic area
Net filtration pressure
Phase 4 of ventricular AP
Interstital hydrostatic pressure (Pi)
46. Pushes fluid into capillary
Supplies blood to the base of the left ventricle
Phase 4 of ventricular AP
Interstital hydrostatic pressure (Pi)
Normal S2 splitting
47. Caused by hypokalemia or bradycardia
Auscultation location of pulmonic area
Phase 1 of ventricular AP
Phase 2 of ventricular AP
U wave on EKG
48. Associated with atrial septal defect (ASD) - allowing a left to right shunt that increases flow through pulmonic valve and delays closure
Wide S2 splitting
Capillary hydrostatic pressure (Pc)
Plasma colloid osmotic pressure
Fixed S2 splitting
49. 'Irregularly irregular' - No discrete P waves and irregularly spaced QRS complexes
Phase 4 of ventricular AP
Phase 3 of ventricular AP
A fib on EKG
Phase 0 of nodal AP
50. Delayed 'rumbling' late diastolic murmur following an opening snap
Mitral stenosis
Net filtration pressure
Aortic Stenosis
S4