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Test your basic knowledge |
USMLE Cardiovascular Physiology
Start Test
Study First
Subjects
:
health-sciences
,
usmle
Instructions:
Answer 50 questions in 15 minutes.
If you are not ready to take this test, you can
study here
.
Match each statement with the correct term.
Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.
This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. Upstroke due to opening of Ca channels (Nodal tissues lack fast Na channels which allows for a prolonged AV node transmission from atria to ventricles)
Phase 0 of ventricular AP
Phase 1 of ventricular AP
Phase 0 of nodal AP
Aortic regurgitation
2. Posterior descending artery (80% from RCA - 20% from CFX via LCA)
Capillary hydrostatic pressure (Pc)
1st degree AV block on EKG
Ventricular septal defect
Supplies blood to the base of the left ventricle
3. 4th rib at left sternal border
Auscultation location of tricuspid area
Phase 0 of nodal AP
Ventricular septal defect
Tricuspid Regurgitation
4. Pushes fluid out of capillary
Normal S2 splitting
Phase 3 of nodal AP
Tricuspid Regurgitation
Capillary hydrostatic pressure (Pc)
5. Crescendo - decrescendo systolic ejection murmur following an ejection click. Associated with weak pulses & syncope.
Aortic Stenosis
Wide S2 splitting
Phase 2 of ventricular AP
Fick principle
6. Indicates recent MI
Auscultation location of aortic area
T wave inversion on EKG
Capillary hydrostatic pressure (Pc)
Wolff - Parkinson - White Syndrome
7. Pull fluid out of capillary
Net filtration pressure
Interstitial fluid colloid osmotic pressure
Ventricular septal defect
Mitral Regurgitation
8. Rapid upstroke due to voltage - gated Na channel opening
Phase 0 of ventricular AP
Auscultation location of pulmonic area
Mobitz type II 2nd degree AV block
Mitral stenosis
9. Mitral & tripcuspid valve closure
S1
Mitral stenosis
Phase 0 of ventricular AP
Mobitz type I (Wenckebach) 2nd degree AV block
10. Associated with pulmonic stenosis or RBBB (delayed RV emptying)
Wide S2 splitting
Ventricular septal defect
Plasma colloid osmotic pressure
Phase 3 of ventricular AP
11. Aortic & pulmonic valve closure
T wave inversion on EKG
Auscultation location of mitral area
Phase 2 of nodal AP
S2
12. Pulls fluid into capillary
A fib on EKG
Plasma colloid osmotic pressure
Patent ductus arteriosus
Fixed S2 splitting
13. Aortic valve closure slightly before pulmonic valve closure at end of systolic ejection
Ventricular septal defect
T wave inversion on EKG
S1
Normal S2 splitting
14. Late diastole 'atrial kick' sound against increased pressure from ventricular hypertrophy
Net filtration pressure
Phase 0 of ventricular AP
Resistance is directly proportional to...
S4
15. Ventricular pre - excitation caused by an accessory conduction pathway that bypasses the AV node. Delta wave on EKG. Can lead to SVT.
T wave inversion on EKG
Plasma colloid osmotic pressure
Phase 3 of ventricular AP
Wolff - Parkinson - White Syndrome
16. Progressive lengthening of PR interval until dropped QRS complex
Phase 4 of ventricular AP
Mean arterial pressure (MAP) =
Mobitz type I (Wenckebach) 2nd degree AV block
Phase 4 of nodal AP
17. Pushes fluid into capillary
Patent ductus arteriosus
Interstital hydrostatic pressure (Pi)
Mitral prolapse
Phase 3 of ventricular AP
18. High pitched 'blowing' diastolic murmur - can decrease intensity of murmur with vasodilation
Mitral Regurgitation
Aortic regurgitation
Resistance is directly proportional to...
Resistance is inversely proportional to...
19. Rapid repolarization due to massive K efflux & closure of Ca channels
Fixed S2 splitting
Mitral prolapse
Phase 3 of ventricular AP
Mobitz type II 2nd degree AV block
20. Early diastole rapid filling sound heard with dilated ventricle (normal in children & pregnant women)
S2
S3
Mitral Regurgitation
Common causes of edema
21. 1) increased capillary pressure - CHF 2) decreased plasma proteins - nephrotic syndrome - liver failure 3) increased capillary permeability - toxins - burns - infections 4) increased interstital osmotic pressure - lymphatic obstruction
Common causes of edema
Auscultation location of mitral area
S2
Patent ductus arteriosus
22. 3rd degree
Phase 0 of ventricular AP
Wide S2 splitting
Lyme disease can cause why type of AV block?
Fixed S2 splitting
23. Inspiration delays pulmonic valve closure - which increases S2 splitting
S3
Paradoxical S2 splitting
Effect of inspiration on S2 splitting
Auscultation location of pulmonic area
24. Resting potential due to high K permeability
Ventricular septal defect
Phase 4 of ventricular AP
Phase 0 of nodal AP
Interstitial fluid colloid osmotic pressure
25. CO = rate of O2 consumption / (arterial O2 content - venous arterial O2 content)
Patent ductus arteriosus
Fick principle
T wave inversion on EKG
Ventricular septal defect
26. Initial repolarization due to inactivation of Na channels & opening of voltaged - gated K channels
S2
Phase 0 of nodal AP
Phase 1 of ventricular AP
A flutter on EKG
27. Pulmonic valve closure before aortic valve closure associated with aortic stenosis or LBBB (delayed left ventrical emptying)
Interstital hydrostatic pressure (Pi)
Mitral Regurgitation
Paradoxical S2 splitting
Phase 4 of nodal AP
28. Holosystolic 'blowing murmur' loudest at left 5th intercostal space midclavicular line - & is enhanced by expiration (increased LA return) & squatting (increased TPR)
Aortic Stenosis
Mitral Regurgitation
Phase 4 of nodal AP
Mean arterial pressure (MAP) =
29. Blood viscosity (increased in polycythemia - hyperproteinemia - & hereditary spherocytosis)
Interstitial fluid colloid osmotic pressure
Resistance is directly proportional to...
Phase 4 of nodal AP
Capillary hydrostatic pressure (Pc)
30. Dropped QRS complexes not preceded by change in PR interval (can be 2:1 - 3:1 - etc)
Auscultation location of pulmonic area
Mobitz type II 2nd degree AV block
Aortic Stenosis
Phase 1 of ventricular AP
31. Late systolic crescendo murmur (loudest at S2) following a midsystolic click - enhanced by squatting (increased TPR)
Mean arterial pressure (MAP) =
Mitral prolapse
Normal S2 splitting
Phase 2 of nodal AP
32. Rapid back - to - back atrial depolarization -> 'sawtooth' appearance
S1
A flutter on EKG
Aortic regurgitation
Torsades de Pointes
33. 5th intercostal space at left midclavicular line
Aortic Stenosis
Auscultation location of mitral area
Capillary hydrostatic pressure (Pc)
Lyme disease can cause why type of AV block?
34. Repolarization due to inactivation of Ca channels & activation of K channels
Auscultation location of aortic area
Phase 3 of nodal AP
Resistance is inversely proportional to...
3rd degree complete AV block
35. Atria & ventricles beat independent of each other - no relation between P waves & QRS complexes
Common causes of edema
3rd degree complete AV block
Auscultation location of aortic area
Mobitz type II 2nd degree AV block
36. Associated with atrial septal defect (ASD) - allowing a left to right shunt that increases flow through pulmonic valve and delays closure
Fixed S2 splitting
Net filtration pressure
Fick principle
Phase 2 of ventricular AP
37. Radius to the 4th power
Phase 4 of ventricular AP
Fick principle
1st degree AV block on EKG
Resistance is inversely proportional to...
38. 2nd intercostal space along left sternal border
Wide S2 splitting
Mitral stenosis
Auscultation location of pulmonic area
1st degree AV block on EKG
39. Caused by hypokalemia or bradycardia
S1
Common causes of edema
U wave on EKG
A fib on EKG
40. Cardiac output (CO) x total peripheral resistance (TPR) - or 2/3 diastolic pressure + 1/3 systolic pressure
Plasma colloid osmotic pressure
Patent ductus arteriosus
Auscultation location of aortic area
Mean arterial pressure (MAP) =
41. Plateau due to Ca influx balancing K efflux Myocyte contraction
Net filtration pressure
3rd degree complete AV block
Lyme disease can cause why type of AV block?
Phase 2 of ventricular AP
42. Holosystolic 'harsh' murmur that is loudest at left sternal border near 4th rib
Ventricular septal defect
Phase 2 of nodal AP
Capillary hydrostatic pressure (Pc)
Mobitz type I (Wenckebach) 2nd degree AV block
43. Continuous machine - like murmur that is loudest at S2
Wide S2 splitting
A flutter on EKG
Mitral prolapse
Patent ductus arteriosus
44. V Tach with shifting sinusoidal waveforms on EKG - can progess to V fib - Predisposed by QT prolongation
Aortic Stenosis
S3
Phase 0 of nodal AP
Torsades de Pointes
45. 'Irregularly irregular' - No discrete P waves and irregularly spaced QRS complexes
Phase 0 of ventricular AP
Common causes of edema
Net filtration pressure
A fib on EKG
46. Holosystolic 'blowing murmur' loudest at left sternal border near 4th rib - & is enhanced by inspiration (increased RA return)
Tricuspid Regurgitation
Mobitz type I (Wenckebach) 2nd degree AV block
Torsades de Pointes
S3
47. Absent (no plateau in nodal tissue)
Phase 2 of nodal AP
Ventricular septal defect
Capillary hydrostatic pressure (Pc)
Resistance is directly proportional to...
48. Delayed 'rumbling' late diastolic murmur following an opening snap
Mitral stenosis
Fick principle
Phase 0 of nodal AP
Paradoxical S2 splitting
49. = (capillary pressure - interstital fluid pressure) - (plasma oncotic pressure - interstital fluid oncotic pressure)
A flutter on EKG
Auscultation location of tricuspid area
Net filtration pressure
Phase 2 of nodal AP
50. Slow diastolic depolarization with Na conductance that accounts for automaticity of SA/AV nodes (slope determines HR)
Phase 4 of nodal AP
Effect of inspiration on S2 splitting
Interstitial fluid colloid osmotic pressure
Phase 1 of ventricular AP