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Test your basic knowledge |
USMLE Cardiovascular Physiology
Start Test
Study First
Subjects
:
health-sciences
,
usmle
Instructions:
Answer 50 questions in 15 minutes.
If you are not ready to take this test, you can
study here
.
Match each statement with the correct term.
Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.
This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. Crescendo - decrescendo systolic ejection murmur following an ejection click. Associated with weak pulses & syncope.
Wide S2 splitting
Mitral prolapse
T wave inversion on EKG
Aortic Stenosis
2. Posterior descending artery (80% from RCA - 20% from CFX via LCA)
Supplies blood to the base of the left ventricle
Effect of inspiration on S2 splitting
Aortic regurgitation
Mean arterial pressure (MAP) =
3. Late diastole 'atrial kick' sound against increased pressure from ventricular hypertrophy
Mean arterial pressure (MAP) =
Common causes of edema
S4
Mitral stenosis
4. Upstroke due to opening of Ca channels (Nodal tissues lack fast Na channels which allows for a prolonged AV node transmission from atria to ventricles)
T wave inversion on EKG
A fib on EKG
Phase 0 of nodal AP
Mitral prolapse
5. Blood viscosity (increased in polycythemia - hyperproteinemia - & hereditary spherocytosis)
Tricuspid Regurgitation
Resistance is directly proportional to...
Phase 0 of ventricular AP
Aortic Stenosis
6. 4th rib at left sternal border
S3
Interstital hydrostatic pressure (Pi)
Fixed S2 splitting
Auscultation location of tricuspid area
7. Repolarization due to inactivation of Ca channels & activation of K channels
Mitral prolapse
Torsades de Pointes
Phase 3 of nodal AP
Aortic regurgitation
8. Plateau due to Ca influx balancing K efflux Myocyte contraction
Phase 4 of ventricular AP
Phase 2 of ventricular AP
Common causes of edema
S4
9. Rapid back - to - back atrial depolarization -> 'sawtooth' appearance
Auscultation location of pulmonic area
Wide S2 splitting
S3
A flutter on EKG
10. Associated with atrial septal defect (ASD) - allowing a left to right shunt that increases flow through pulmonic valve and delays closure
Phase 4 of ventricular AP
Mobitz type I (Wenckebach) 2nd degree AV block
Plasma colloid osmotic pressure
Fixed S2 splitting
11. 1) increased capillary pressure - CHF 2) decreased plasma proteins - nephrotic syndrome - liver failure 3) increased capillary permeability - toxins - burns - infections 4) increased interstital osmotic pressure - lymphatic obstruction
Phase 0 of ventricular AP
S1
Common causes of edema
Mitral Regurgitation
12. V Tach with shifting sinusoidal waveforms on EKG - can progess to V fib - Predisposed by QT prolongation
Effect of inspiration on S2 splitting
Torsades de Pointes
Aortic Stenosis
Capillary hydrostatic pressure (Pc)
13. Slow diastolic depolarization with Na conductance that accounts for automaticity of SA/AV nodes (slope determines HR)
Mitral prolapse
S1
Phase 2 of ventricular AP
Phase 4 of nodal AP
14. Holosystolic 'blowing murmur' loudest at left 5th intercostal space midclavicular line - & is enhanced by expiration (increased LA return) & squatting (increased TPR)
Paradoxical S2 splitting
1st degree AV block on EKG
Mobitz type II 2nd degree AV block
Mitral Regurgitation
15. 5th intercostal space at left midclavicular line
Phase 4 of ventricular AP
Phase 3 of nodal AP
Normal S2 splitting
Auscultation location of mitral area
16. Atria & ventricles beat independent of each other - no relation between P waves & QRS complexes
Wide S2 splitting
3rd degree complete AV block
Mitral Regurgitation
Paradoxical S2 splitting
17. High pitched 'blowing' diastolic murmur - can decrease intensity of murmur with vasodilation
Mitral stenosis
Auscultation location of tricuspid area
Mobitz type II 2nd degree AV block
Aortic regurgitation
18. Inspiration delays pulmonic valve closure - which increases S2 splitting
Effect of inspiration on S2 splitting
T wave inversion on EKG
Patent ductus arteriosus
Aortic regurgitation
19. Pulls fluid into capillary
Phase 1 of ventricular AP
Lyme disease can cause why type of AV block?
S4
Plasma colloid osmotic pressure
20. Aortic & pulmonic valve closure
S2
Wide S2 splitting
Ventricular septal defect
Effect of inspiration on S2 splitting
21. Holosystolic 'harsh' murmur that is loudest at left sternal border near 4th rib
Ventricular septal defect
S3
Auscultation location of tricuspid area
3rd degree complete AV block
22. Early diastole rapid filling sound heard with dilated ventricle (normal in children & pregnant women)
S3
Normal S2 splitting
Common causes of edema
Auscultation location of pulmonic area
23. Ventricular pre - excitation caused by an accessory conduction pathway that bypasses the AV node. Delta wave on EKG. Can lead to SVT.
Wolff - Parkinson - White Syndrome
Phase 3 of ventricular AP
Net filtration pressure
Phase 3 of nodal AP
24. Absent (no plateau in nodal tissue)
Fixed S2 splitting
Effect of inspiration on S2 splitting
Phase 2 of nodal AP
Interstitial fluid colloid osmotic pressure
25. Mitral & tripcuspid valve closure
Supplies blood to the base of the left ventricle
S1
Common causes of edema
Phase 2 of ventricular AP
26. Prolonged PR interval
1st degree AV block on EKG
Auscultation location of tricuspid area
Tricuspid Regurgitation
Patent ductus arteriosus
27. 'Irregularly irregular' - No discrete P waves and irregularly spaced QRS complexes
Normal S2 splitting
A fib on EKG
3rd degree complete AV block
Effect of inspiration on S2 splitting
28. Dropped QRS complexes not preceded by change in PR interval (can be 2:1 - 3:1 - etc)
1st degree AV block on EKG
Mobitz type II 2nd degree AV block
S4
Mean arterial pressure (MAP) =
29. Holosystolic 'blowing murmur' loudest at left sternal border near 4th rib - & is enhanced by inspiration (increased RA return)
Tricuspid Regurgitation
U wave on EKG
Common causes of edema
Wide S2 splitting
30. Caused by hypokalemia or bradycardia
U wave on EKG
Aortic regurgitation
Interstitial fluid colloid osmotic pressure
Phase 0 of nodal AP
31. Rapid upstroke due to voltage - gated Na channel opening
Phase 0 of ventricular AP
Capillary hydrostatic pressure (Pc)
Mitral Regurgitation
Phase 2 of ventricular AP
32. Pulmonic valve closure before aortic valve closure associated with aortic stenosis or LBBB (delayed left ventrical emptying)
S3
Paradoxical S2 splitting
Aortic regurgitation
Auscultation location of mitral area
33. Pull fluid out of capillary
Phase 1 of ventricular AP
Mitral stenosis
S1
Interstitial fluid colloid osmotic pressure
34. Resting potential due to high K permeability
Aortic Stenosis
Mobitz type I (Wenckebach) 2nd degree AV block
Phase 4 of ventricular AP
Tricuspid Regurgitation
35. Radius to the 4th power
3rd degree complete AV block
Phase 3 of ventricular AP
Resistance is inversely proportional to...
Supplies blood to the base of the left ventricle
36. Rapid repolarization due to massive K efflux & closure of Ca channels
Lyme disease can cause why type of AV block?
Interstital hydrostatic pressure (Pi)
A fib on EKG
Phase 3 of ventricular AP
37. Pushes fluid into capillary
Interstital hydrostatic pressure (Pi)
Interstitial fluid colloid osmotic pressure
Plasma colloid osmotic pressure
Fixed S2 splitting
38. 2nd intercostal space along left sternal border
Interstitial fluid colloid osmotic pressure
Mobitz type II 2nd degree AV block
Auscultation location of pulmonic area
Aortic regurgitation
39. Cardiac output (CO) x total peripheral resistance (TPR) - or 2/3 diastolic pressure + 1/3 systolic pressure
Ventricular septal defect
Phase 0 of nodal AP
Mean arterial pressure (MAP) =
Torsades de Pointes
40. Delayed 'rumbling' late diastolic murmur following an opening snap
Torsades de Pointes
Fixed S2 splitting
Mitral stenosis
S1
41. 3rd degree
Resistance is inversely proportional to...
Phase 3 of nodal AP
Lyme disease can cause why type of AV block?
1st degree AV block on EKG
42. Initial repolarization due to inactivation of Na channels & opening of voltaged - gated K channels
Auscultation location of mitral area
Net filtration pressure
Phase 1 of ventricular AP
Wide S2 splitting
43. Progressive lengthening of PR interval until dropped QRS complex
1st degree AV block on EKG
Interstitial fluid colloid osmotic pressure
Mobitz type I (Wenckebach) 2nd degree AV block
Auscultation location of aortic area
44. Continuous machine - like murmur that is loudest at S2
Patent ductus arteriosus
A flutter on EKG
Tricuspid Regurgitation
1st degree AV block on EKG
45. Associated with pulmonic stenosis or RBBB (delayed RV emptying)
A fib on EKG
Wide S2 splitting
Plasma colloid osmotic pressure
Phase 2 of nodal AP
46. 2nd intercostal space along right sternal border
S1
Auscultation location of aortic area
S3
Aortic Stenosis
47. CO = rate of O2 consumption / (arterial O2 content - venous arterial O2 content)
Normal S2 splitting
Aortic Stenosis
Fick principle
Resistance is inversely proportional to...
48. Indicates recent MI
Fixed S2 splitting
T wave inversion on EKG
Auscultation location of pulmonic area
A flutter on EKG
49. Aortic valve closure slightly before pulmonic valve closure at end of systolic ejection
Normal S2 splitting
S1
Mitral stenosis
Patent ductus arteriosus
50. Late systolic crescendo murmur (loudest at S2) following a midsystolic click - enhanced by squatting (increased TPR)
S3
Aortic regurgitation
Mitral prolapse
Normal S2 splitting