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Test your basic knowledge |
USMLE Cardiovascular Physiology
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Subjects
:
health-sciences
,
usmle
Instructions:
Answer 50 questions in 15 minutes.
If you are not ready to take this test, you can
study here
.
Match each statement with the correct term.
Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.
This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. Rapid repolarization due to massive K efflux & closure of Ca channels
Aortic Stenosis
Ventricular septal defect
Resistance is inversely proportional to...
Phase 3 of ventricular AP
2. Rapid back - to - back atrial depolarization -> 'sawtooth' appearance
Interstital hydrostatic pressure (Pi)
Tricuspid Regurgitation
Auscultation location of mitral area
A flutter on EKG
3. Radius to the 4th power
Phase 1 of ventricular AP
Resistance is inversely proportional to...
Resistance is directly proportional to...
Wolff - Parkinson - White Syndrome
4. Pulmonic valve closure before aortic valve closure associated with aortic stenosis or LBBB (delayed left ventrical emptying)
Paradoxical S2 splitting
Aortic Stenosis
Interstitial fluid colloid osmotic pressure
Aortic regurgitation
5. Caused by hypokalemia or bradycardia
Effect of inspiration on S2 splitting
Fick principle
U wave on EKG
Mitral prolapse
6. High pitched 'blowing' diastolic murmur - can decrease intensity of murmur with vasodilation
Mobitz type II 2nd degree AV block
Aortic regurgitation
Common causes of edema
Normal S2 splitting
7. Dropped QRS complexes not preceded by change in PR interval (can be 2:1 - 3:1 - etc)
Mitral prolapse
Fixed S2 splitting
Mobitz type II 2nd degree AV block
3rd degree complete AV block
8. V Tach with shifting sinusoidal waveforms on EKG - can progess to V fib - Predisposed by QT prolongation
Torsades de Pointes
Wolff - Parkinson - White Syndrome
Paradoxical S2 splitting
Aortic Stenosis
9. Aortic & pulmonic valve closure
Fick principle
S2
Phase 2 of ventricular AP
Resistance is directly proportional to...
10. Pushes fluid out of capillary
S2
Capillary hydrostatic pressure (Pc)
Fixed S2 splitting
Mobitz type I (Wenckebach) 2nd degree AV block
11. Blood viscosity (increased in polycythemia - hyperproteinemia - & hereditary spherocytosis)
Phase 0 of nodal AP
Resistance is directly proportional to...
A fib on EKG
Plasma colloid osmotic pressure
12. 'Irregularly irregular' - No discrete P waves and irregularly spaced QRS complexes
Phase 2 of nodal AP
A fib on EKG
Fixed S2 splitting
Net filtration pressure
13. Plateau due to Ca influx balancing K efflux Myocyte contraction
Phase 0 of nodal AP
Phase 2 of ventricular AP
Wolff - Parkinson - White Syndrome
Torsades de Pointes
14. 5th intercostal space at left midclavicular line
Resistance is inversely proportional to...
Fixed S2 splitting
A fib on EKG
Auscultation location of mitral area
15. Rapid upstroke due to voltage - gated Na channel opening
A flutter on EKG
3rd degree complete AV block
Phase 0 of ventricular AP
Plasma colloid osmotic pressure
16. Posterior descending artery (80% from RCA - 20% from CFX via LCA)
Supplies blood to the base of the left ventricle
S3
Phase 4 of ventricular AP
Interstitial fluid colloid osmotic pressure
17. Resting potential due to high K permeability
Phase 4 of ventricular AP
Phase 3 of ventricular AP
Mitral prolapse
Phase 0 of nodal AP
18. Associated with atrial septal defect (ASD) - allowing a left to right shunt that increases flow through pulmonic valve and delays closure
Phase 0 of ventricular AP
Fixed S2 splitting
Mobitz type I (Wenckebach) 2nd degree AV block
Auscultation location of mitral area
19. Ventricular pre - excitation caused by an accessory conduction pathway that bypasses the AV node. Delta wave on EKG. Can lead to SVT.
Wolff - Parkinson - White Syndrome
1st degree AV block on EKG
S4
Phase 4 of nodal AP
20. 4th rib at left sternal border
Phase 1 of ventricular AP
Aortic Stenosis
Phase 4 of nodal AP
Auscultation location of tricuspid area
21. Early diastole rapid filling sound heard with dilated ventricle (normal in children & pregnant women)
Mobitz type I (Wenckebach) 2nd degree AV block
Resistance is inversely proportional to...
S3
U wave on EKG
22. 1) increased capillary pressure - CHF 2) decreased plasma proteins - nephrotic syndrome - liver failure 3) increased capillary permeability - toxins - burns - infections 4) increased interstital osmotic pressure - lymphatic obstruction
Mitral prolapse
T wave inversion on EKG
Common causes of edema
Auscultation location of tricuspid area
23. Late diastole 'atrial kick' sound against increased pressure from ventricular hypertrophy
Mean arterial pressure (MAP) =
S4
Lyme disease can cause why type of AV block?
Patent ductus arteriosus
24. Absent (no plateau in nodal tissue)
Phase 2 of nodal AP
Mobitz type II 2nd degree AV block
Resistance is inversely proportional to...
Auscultation location of tricuspid area
25. Slow diastolic depolarization with Na conductance that accounts for automaticity of SA/AV nodes (slope determines HR)
A flutter on EKG
Mitral stenosis
Phase 4 of nodal AP
Auscultation location of mitral area
26. Initial repolarization due to inactivation of Na channels & opening of voltaged - gated K channels
Normal S2 splitting
Phase 1 of ventricular AP
Lyme disease can cause why type of AV block?
Aortic Stenosis
27. Holosystolic 'blowing murmur' loudest at left 5th intercostal space midclavicular line - & is enhanced by expiration (increased LA return) & squatting (increased TPR)
Phase 2 of nodal AP
Phase 2 of ventricular AP
Mitral Regurgitation
Interstital hydrostatic pressure (Pi)
28. Mitral & tripcuspid valve closure
Patent ductus arteriosus
Fick principle
Auscultation location of tricuspid area
S1
29. Upstroke due to opening of Ca channels (Nodal tissues lack fast Na channels which allows for a prolonged AV node transmission from atria to ventricles)
Paradoxical S2 splitting
Phase 0 of nodal AP
Lyme disease can cause why type of AV block?
Mobitz type I (Wenckebach) 2nd degree AV block
30. Indicates recent MI
T wave inversion on EKG
Lyme disease can cause why type of AV block?
Phase 2 of ventricular AP
A flutter on EKG
31. Pull fluid out of capillary
Torsades de Pointes
Auscultation location of mitral area
3rd degree complete AV block
Interstitial fluid colloid osmotic pressure
32. Pulls fluid into capillary
Patent ductus arteriosus
Mobitz type I (Wenckebach) 2nd degree AV block
Interstital hydrostatic pressure (Pi)
Plasma colloid osmotic pressure
33. Repolarization due to inactivation of Ca channels & activation of K channels
Phase 3 of nodal AP
U wave on EKG
Mobitz type II 2nd degree AV block
Capillary hydrostatic pressure (Pc)
34. 2nd intercostal space along left sternal border
Supplies blood to the base of the left ventricle
S4
Phase 2 of ventricular AP
Auscultation location of pulmonic area
35. 3rd degree
Lyme disease can cause why type of AV block?
Phase 1 of ventricular AP
Mobitz type II 2nd degree AV block
A fib on EKG
36. Atria & ventricles beat independent of each other - no relation between P waves & QRS complexes
3rd degree complete AV block
U wave on EKG
Phase 3 of nodal AP
Auscultation location of pulmonic area
37. Delayed 'rumbling' late diastolic murmur following an opening snap
S4
Auscultation location of aortic area
Mitral stenosis
S3
38. Inspiration delays pulmonic valve closure - which increases S2 splitting
T wave inversion on EKG
Effect of inspiration on S2 splitting
Patent ductus arteriosus
Capillary hydrostatic pressure (Pc)
39. Prolonged PR interval
Resistance is directly proportional to...
1st degree AV block on EKG
S4
Auscultation location of tricuspid area
40. Continuous machine - like murmur that is loudest at S2
A flutter on EKG
Patent ductus arteriosus
Phase 1 of ventricular AP
Interstital hydrostatic pressure (Pi)
41. CO = rate of O2 consumption / (arterial O2 content - venous arterial O2 content)
Fick principle
Normal S2 splitting
S1
Wide S2 splitting
42. 2nd intercostal space along right sternal border
Aortic regurgitation
Mobitz type I (Wenckebach) 2nd degree AV block
Fixed S2 splitting
Auscultation location of aortic area
43. Aortic valve closure slightly before pulmonic valve closure at end of systolic ejection
Normal S2 splitting
Auscultation location of pulmonic area
Phase 4 of ventricular AP
A fib on EKG
44. Cardiac output (CO) x total peripheral resistance (TPR) - or 2/3 diastolic pressure + 1/3 systolic pressure
U wave on EKG
Interstital hydrostatic pressure (Pi)
Supplies blood to the base of the left ventricle
Mean arterial pressure (MAP) =
45. Holosystolic 'blowing murmur' loudest at left sternal border near 4th rib - & is enhanced by inspiration (increased RA return)
Phase 2 of ventricular AP
Wide S2 splitting
S4
Tricuspid Regurgitation
46. Associated with pulmonic stenosis or RBBB (delayed RV emptying)
Wide S2 splitting
Phase 2 of nodal AP
Plasma colloid osmotic pressure
Capillary hydrostatic pressure (Pc)
47. Crescendo - decrescendo systolic ejection murmur following an ejection click. Associated with weak pulses & syncope.
Aortic Stenosis
S3
Phase 4 of ventricular AP
Auscultation location of mitral area
48. Holosystolic 'harsh' murmur that is loudest at left sternal border near 4th rib
Ventricular septal defect
Lyme disease can cause why type of AV block?
1st degree AV block on EKG
Mean arterial pressure (MAP) =
49. = (capillary pressure - interstital fluid pressure) - (plasma oncotic pressure - interstital fluid oncotic pressure)
Interstitial fluid colloid osmotic pressure
Phase 4 of nodal AP
Net filtration pressure
Tricuspid Regurgitation
50. Late systolic crescendo murmur (loudest at S2) following a midsystolic click - enhanced by squatting (increased TPR)
Auscultation location of mitral area
Paradoxical S2 splitting
Effect of inspiration on S2 splitting
Mitral prolapse
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