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Test your basic knowledge |
USMLE Cardiovascular Physiology
Start Test
Study First
Subjects
:
health-sciences
,
usmle
Instructions:
Answer 50 questions in 15 minutes.
If you are not ready to take this test, you can
study here
.
Match each statement with the correct term.
Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.
This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. Repolarization due to inactivation of Ca channels & activation of K channels
Auscultation location of aortic area
Tricuspid Regurgitation
Phase 2 of nodal AP
Phase 3 of nodal AP
2. Slow diastolic depolarization with Na conductance that accounts for automaticity of SA/AV nodes (slope determines HR)
S2
Phase 4 of nodal AP
Patent ductus arteriosus
Phase 0 of nodal AP
3. Pushes fluid out of capillary
Capillary hydrostatic pressure (Pc)
Supplies blood to the base of the left ventricle
Fick principle
Lyme disease can cause why type of AV block?
4. Absent (no plateau in nodal tissue)
Phase 1 of ventricular AP
S1
T wave inversion on EKG
Phase 2 of nodal AP
5. Pull fluid out of capillary
Tricuspid Regurgitation
Interstitial fluid colloid osmotic pressure
Phase 1 of ventricular AP
Phase 2 of nodal AP
6. 4th rib at left sternal border
Auscultation location of pulmonic area
Phase 2 of ventricular AP
Normal S2 splitting
Auscultation location of tricuspid area
7. Plateau due to Ca influx balancing K efflux Myocyte contraction
T wave inversion on EKG
Phase 2 of ventricular AP
Tricuspid Regurgitation
Phase 2 of nodal AP
8. Continuous machine - like murmur that is loudest at S2
Lyme disease can cause why type of AV block?
Phase 0 of ventricular AP
Patent ductus arteriosus
1st degree AV block on EKG
9. Upstroke due to opening of Ca channels (Nodal tissues lack fast Na channels which allows for a prolonged AV node transmission from atria to ventricles)
U wave on EKG
Mitral Regurgitation
Patent ductus arteriosus
Phase 0 of nodal AP
10. Resting potential due to high K permeability
Phase 4 of ventricular AP
Mobitz type I (Wenckebach) 2nd degree AV block
Interstital hydrostatic pressure (Pi)
1st degree AV block on EKG
11. Indicates recent MI
Phase 4 of nodal AP
T wave inversion on EKG
Phase 1 of ventricular AP
Lyme disease can cause why type of AV block?
12. Cardiac output (CO) x total peripheral resistance (TPR) - or 2/3 diastolic pressure + 1/3 systolic pressure
T wave inversion on EKG
Net filtration pressure
Mean arterial pressure (MAP) =
Effect of inspiration on S2 splitting
13. Inspiration delays pulmonic valve closure - which increases S2 splitting
Phase 0 of ventricular AP
3rd degree complete AV block
Effect of inspiration on S2 splitting
S2
14. Pulmonic valve closure before aortic valve closure associated with aortic stenosis or LBBB (delayed left ventrical emptying)
Mobitz type I (Wenckebach) 2nd degree AV block
1st degree AV block on EKG
Paradoxical S2 splitting
Phase 4 of ventricular AP
15. Crescendo - decrescendo systolic ejection murmur following an ejection click. Associated with weak pulses & syncope.
Aortic Stenosis
Phase 1 of ventricular AP
T wave inversion on EKG
Tricuspid Regurgitation
16. Progressive lengthening of PR interval until dropped QRS complex
Phase 0 of ventricular AP
Effect of inspiration on S2 splitting
Mobitz type I (Wenckebach) 2nd degree AV block
3rd degree complete AV block
17. Pulls fluid into capillary
Phase 4 of ventricular AP
Aortic Stenosis
Normal S2 splitting
Plasma colloid osmotic pressure
18. Holosystolic 'blowing murmur' loudest at left sternal border near 4th rib - & is enhanced by inspiration (increased RA return)
Ventricular septal defect
Tricuspid Regurgitation
Supplies blood to the base of the left ventricle
Auscultation location of tricuspid area
19. = (capillary pressure - interstital fluid pressure) - (plasma oncotic pressure - interstital fluid oncotic pressure)
Net filtration pressure
Mitral Regurgitation
Phase 4 of ventricular AP
S1
20. Caused by hypokalemia or bradycardia
Mobitz type II 2nd degree AV block
U wave on EKG
Auscultation location of mitral area
Auscultation location of aortic area
21. V Tach with shifting sinusoidal waveforms on EKG - can progess to V fib - Predisposed by QT prolongation
Fick principle
Torsades de Pointes
S4
Capillary hydrostatic pressure (Pc)
22. High pitched 'blowing' diastolic murmur - can decrease intensity of murmur with vasodilation
Phase 4 of ventricular AP
Aortic regurgitation
Phase 4 of nodal AP
U wave on EKG
23. Aortic valve closure slightly before pulmonic valve closure at end of systolic ejection
Phase 0 of nodal AP
Auscultation location of mitral area
Normal S2 splitting
Phase 4 of nodal AP
24. Early diastole rapid filling sound heard with dilated ventricle (normal in children & pregnant women)
Fick principle
Torsades de Pointes
S3
S4
25. CO = rate of O2 consumption / (arterial O2 content - venous arterial O2 content)
S4
A flutter on EKG
Fick principle
Patent ductus arteriosus
26. Radius to the 4th power
Auscultation location of mitral area
Common causes of edema
Mobitz type I (Wenckebach) 2nd degree AV block
Resistance is inversely proportional to...
27. Holosystolic 'blowing murmur' loudest at left 5th intercostal space midclavicular line - & is enhanced by expiration (increased LA return) & squatting (increased TPR)
Mitral Regurgitation
U wave on EKG
Effect of inspiration on S2 splitting
Auscultation location of pulmonic area
28. Rapid back - to - back atrial depolarization -> 'sawtooth' appearance
A flutter on EKG
Effect of inspiration on S2 splitting
Mean arterial pressure (MAP) =
U wave on EKG
29. Delayed 'rumbling' late diastolic murmur following an opening snap
Capillary hydrostatic pressure (Pc)
Mitral stenosis
Patent ductus arteriosus
Phase 2 of ventricular AP
30. Rapid repolarization due to massive K efflux & closure of Ca channels
Torsades de Pointes
Mitral stenosis
Auscultation location of tricuspid area
Phase 3 of ventricular AP
31. 1) increased capillary pressure - CHF 2) decreased plasma proteins - nephrotic syndrome - liver failure 3) increased capillary permeability - toxins - burns - infections 4) increased interstital osmotic pressure - lymphatic obstruction
Common causes of edema
Phase 0 of nodal AP
Auscultation location of tricuspid area
Mobitz type II 2nd degree AV block
32. Pushes fluid into capillary
Tricuspid Regurgitation
Phase 0 of ventricular AP
Auscultation location of aortic area
Interstital hydrostatic pressure (Pi)
33. Holosystolic 'harsh' murmur that is loudest at left sternal border near 4th rib
Ventricular septal defect
Lyme disease can cause why type of AV block?
Aortic Stenosis
S2
34. Mitral & tripcuspid valve closure
Phase 3 of ventricular AP
Mean arterial pressure (MAP) =
Mobitz type I (Wenckebach) 2nd degree AV block
S1
35. Posterior descending artery (80% from RCA - 20% from CFX via LCA)
Fixed S2 splitting
Auscultation location of mitral area
S4
Supplies blood to the base of the left ventricle
36. Initial repolarization due to inactivation of Na channels & opening of voltaged - gated K channels
Torsades de Pointes
Phase 1 of ventricular AP
Wolff - Parkinson - White Syndrome
Mobitz type I (Wenckebach) 2nd degree AV block
37. Associated with pulmonic stenosis or RBBB (delayed RV emptying)
Plasma colloid osmotic pressure
Torsades de Pointes
Wide S2 splitting
Mitral stenosis
38. Associated with atrial septal defect (ASD) - allowing a left to right shunt that increases flow through pulmonic valve and delays closure
Fixed S2 splitting
Mobitz type I (Wenckebach) 2nd degree AV block
S3
Interstitial fluid colloid osmotic pressure
39. Rapid upstroke due to voltage - gated Na channel opening
Phase 4 of nodal AP
1st degree AV block on EKG
Aortic Stenosis
Phase 0 of ventricular AP
40. 'Irregularly irregular' - No discrete P waves and irregularly spaced QRS complexes
Normal S2 splitting
A fib on EKG
Net filtration pressure
Fick principle
41. Dropped QRS complexes not preceded by change in PR interval (can be 2:1 - 3:1 - etc)
Phase 2 of ventricular AP
Phase 3 of ventricular AP
A flutter on EKG
Mobitz type II 2nd degree AV block
42. Ventricular pre - excitation caused by an accessory conduction pathway that bypasses the AV node. Delta wave on EKG. Can lead to SVT.
3rd degree complete AV block
Auscultation location of aortic area
Wolff - Parkinson - White Syndrome
Supplies blood to the base of the left ventricle
43. Late systolic crescendo murmur (loudest at S2) following a midsystolic click - enhanced by squatting (increased TPR)
Aortic Stenosis
S2
Mitral prolapse
Net filtration pressure
44. 2nd intercostal space along left sternal border
Phase 0 of nodal AP
Capillary hydrostatic pressure (Pc)
Auscultation location of pulmonic area
Aortic Stenosis
45. 5th intercostal space at left midclavicular line
Paradoxical S2 splitting
Phase 4 of ventricular AP
Auscultation location of mitral area
Phase 3 of ventricular AP
46. Atria & ventricles beat independent of each other - no relation between P waves & QRS complexes
Fick principle
Tricuspid Regurgitation
Wolff - Parkinson - White Syndrome
3rd degree complete AV block
47. Late diastole 'atrial kick' sound against increased pressure from ventricular hypertrophy
Auscultation location of aortic area
S4
Torsades de Pointes
Normal S2 splitting
48. 3rd degree
Lyme disease can cause why type of AV block?
Supplies blood to the base of the left ventricle
Mobitz type I (Wenckebach) 2nd degree AV block
Resistance is inversely proportional to...
49. Prolonged PR interval
Tricuspid Regurgitation
Effect of inspiration on S2 splitting
1st degree AV block on EKG
Normal S2 splitting
50. Blood viscosity (increased in polycythemia - hyperproteinemia - & hereditary spherocytosis)
Resistance is directly proportional to...
Phase 1 of ventricular AP
Auscultation location of aortic area
Wolff - Parkinson - White Syndrome