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Test your basic knowledge |
USMLE Cardiovascular Physiology
Start Test
Study First
Subjects
:
health-sciences
,
usmle
Instructions:
Answer 50 questions in 15 minutes.
If you are not ready to take this test, you can
study here
.
Match each statement with the correct term.
Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.
This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. Dropped QRS complexes not preceded by change in PR interval (can be 2:1 - 3:1 - etc)
Mobitz type II 2nd degree AV block
Auscultation location of tricuspid area
Mitral prolapse
Torsades de Pointes
2. V Tach with shifting sinusoidal waveforms on EKG - can progess to V fib - Predisposed by QT prolongation
Supplies blood to the base of the left ventricle
Auscultation location of pulmonic area
Mobitz type II 2nd degree AV block
Torsades de Pointes
3. Holosystolic 'blowing murmur' loudest at left sternal border near 4th rib - & is enhanced by inspiration (increased RA return)
Tricuspid Regurgitation
Common causes of edema
Net filtration pressure
Phase 0 of ventricular AP
4. Atria & ventricles beat independent of each other - no relation between P waves & QRS complexes
Phase 3 of ventricular AP
Plasma colloid osmotic pressure
3rd degree complete AV block
Mobitz type II 2nd degree AV block
5. Radius to the 4th power
Normal S2 splitting
Mobitz type I (Wenckebach) 2nd degree AV block
A fib on EKG
Resistance is inversely proportional to...
6. 2nd intercostal space along left sternal border
Phase 0 of ventricular AP
Phase 4 of nodal AP
Auscultation location of pulmonic area
Phase 3 of ventricular AP
7. Ventricular pre - excitation caused by an accessory conduction pathway that bypasses the AV node. Delta wave on EKG. Can lead to SVT.
Phase 0 of nodal AP
Resistance is inversely proportional to...
Paradoxical S2 splitting
Wolff - Parkinson - White Syndrome
8. Late systolic crescendo murmur (loudest at S2) following a midsystolic click - enhanced by squatting (increased TPR)
Mitral prolapse
S1
Patent ductus arteriosus
T wave inversion on EKG
9. High pitched 'blowing' diastolic murmur - can decrease intensity of murmur with vasodilation
Aortic regurgitation
Resistance is inversely proportional to...
Common causes of edema
Phase 2 of ventricular AP
10. 2nd intercostal space along right sternal border
Mitral prolapse
Auscultation location of aortic area
Phase 4 of ventricular AP
Fixed S2 splitting
11. 4th rib at left sternal border
Phase 0 of ventricular AP
Auscultation location of tricuspid area
Interstitial fluid colloid osmotic pressure
Auscultation location of aortic area
12. Resting potential due to high K permeability
Phase 3 of ventricular AP
Phase 4 of ventricular AP
A fib on EKG
Tricuspid Regurgitation
13. Inspiration delays pulmonic valve closure - which increases S2 splitting
Effect of inspiration on S2 splitting
Aortic Stenosis
Paradoxical S2 splitting
Capillary hydrostatic pressure (Pc)
14. CO = rate of O2 consumption / (arterial O2 content - venous arterial O2 content)
Fick principle
S1
1st degree AV block on EKG
Paradoxical S2 splitting
15. Holosystolic 'harsh' murmur that is loudest at left sternal border near 4th rib
Ventricular septal defect
Supplies blood to the base of the left ventricle
Phase 3 of nodal AP
Phase 0 of ventricular AP
16. Aortic & pulmonic valve closure
S2
Net filtration pressure
Torsades de Pointes
Phase 2 of ventricular AP
17. Rapid back - to - back atrial depolarization -> 'sawtooth' appearance
Phase 2 of nodal AP
A flutter on EKG
Mean arterial pressure (MAP) =
S4
18. Posterior descending artery (80% from RCA - 20% from CFX via LCA)
S1
Resistance is inversely proportional to...
Common causes of edema
Supplies blood to the base of the left ventricle
19. Continuous machine - like murmur that is loudest at S2
Patent ductus arteriosus
Phase 2 of ventricular AP
Torsades de Pointes
Phase 1 of ventricular AP
20. Initial repolarization due to inactivation of Na channels & opening of voltaged - gated K channels
Mobitz type I (Wenckebach) 2nd degree AV block
Phase 1 of ventricular AP
A fib on EKG
Phase 3 of nodal AP
21. Associated with pulmonic stenosis or RBBB (delayed RV emptying)
Capillary hydrostatic pressure (Pc)
Wide S2 splitting
Phase 0 of ventricular AP
Aortic regurgitation
22. Crescendo - decrescendo systolic ejection murmur following an ejection click. Associated with weak pulses & syncope.
Common causes of edema
Aortic Stenosis
Paradoxical S2 splitting
Mitral Regurgitation
23. Early diastole rapid filling sound heard with dilated ventricle (normal in children & pregnant women)
Normal S2 splitting
S2
Phase 0 of ventricular AP
S3
24. Delayed 'rumbling' late diastolic murmur following an opening snap
Normal S2 splitting
Capillary hydrostatic pressure (Pc)
Phase 1 of ventricular AP
Mitral stenosis
25. 3rd degree
Lyme disease can cause why type of AV block?
Mitral Regurgitation
S3
Auscultation location of mitral area
26. Holosystolic 'blowing murmur' loudest at left 5th intercostal space midclavicular line - & is enhanced by expiration (increased LA return) & squatting (increased TPR)
Aortic Stenosis
Plasma colloid osmotic pressure
3rd degree complete AV block
Mitral Regurgitation
27. Caused by hypokalemia or bradycardia
Resistance is directly proportional to...
Fixed S2 splitting
U wave on EKG
Phase 0 of ventricular AP
28. Pushes fluid into capillary
Interstitial fluid colloid osmotic pressure
Auscultation location of pulmonic area
Resistance is inversely proportional to...
Interstital hydrostatic pressure (Pi)
29. Plateau due to Ca influx balancing K efflux Myocyte contraction
Phase 2 of ventricular AP
Torsades de Pointes
Plasma colloid osmotic pressure
Phase 0 of nodal AP
30. = (capillary pressure - interstital fluid pressure) - (plasma oncotic pressure - interstital fluid oncotic pressure)
Aortic regurgitation
Net filtration pressure
S2
Phase 0 of ventricular AP
31. Upstroke due to opening of Ca channels (Nodal tissues lack fast Na channels which allows for a prolonged AV node transmission from atria to ventricles)
Auscultation location of mitral area
Mean arterial pressure (MAP) =
S4
Phase 0 of nodal AP
32. Prolonged PR interval
Auscultation location of mitral area
Resistance is inversely proportional to...
Effect of inspiration on S2 splitting
1st degree AV block on EKG
33. Pushes fluid out of capillary
Torsades de Pointes
Mean arterial pressure (MAP) =
Supplies blood to the base of the left ventricle
Capillary hydrostatic pressure (Pc)
34. 5th intercostal space at left midclavicular line
Ventricular septal defect
Fixed S2 splitting
Wide S2 splitting
Auscultation location of mitral area
35. Rapid repolarization due to massive K efflux & closure of Ca channels
Phase 0 of ventricular AP
Phase 3 of ventricular AP
Common causes of edema
S3
36. 'Irregularly irregular' - No discrete P waves and irregularly spaced QRS complexes
Phase 2 of nodal AP
Lyme disease can cause why type of AV block?
A fib on EKG
Phase 0 of nodal AP
37. Mitral & tripcuspid valve closure
Aortic regurgitation
Phase 0 of nodal AP
S1
Supplies blood to the base of the left ventricle
38. Aortic valve closure slightly before pulmonic valve closure at end of systolic ejection
Normal S2 splitting
Interstital hydrostatic pressure (Pi)
Auscultation location of pulmonic area
Torsades de Pointes
39. Rapid upstroke due to voltage - gated Na channel opening
Phase 2 of nodal AP
A flutter on EKG
Phase 0 of ventricular AP
Torsades de Pointes
40. Pulls fluid into capillary
Plasma colloid osmotic pressure
Phase 4 of ventricular AP
Fixed S2 splitting
Mitral prolapse
41. Indicates recent MI
Mobitz type II 2nd degree AV block
Phase 2 of ventricular AP
T wave inversion on EKG
Mitral prolapse
42. 1) increased capillary pressure - CHF 2) decreased plasma proteins - nephrotic syndrome - liver failure 3) increased capillary permeability - toxins - burns - infections 4) increased interstital osmotic pressure - lymphatic obstruction
Common causes of edema
A flutter on EKG
Plasma colloid osmotic pressure
Mean arterial pressure (MAP) =
43. Absent (no plateau in nodal tissue)
Phase 3 of nodal AP
Phase 2 of nodal AP
Auscultation location of mitral area
Mobitz type II 2nd degree AV block
44. Progressive lengthening of PR interval until dropped QRS complex
Mobitz type I (Wenckebach) 2nd degree AV block
Paradoxical S2 splitting
Phase 2 of ventricular AP
Phase 4 of nodal AP
45. Associated with atrial septal defect (ASD) - allowing a left to right shunt that increases flow through pulmonic valve and delays closure
Auscultation location of pulmonic area
Fixed S2 splitting
A flutter on EKG
Mobitz type II 2nd degree AV block
46. Pull fluid out of capillary
Interstitial fluid colloid osmotic pressure
Auscultation location of tricuspid area
Effect of inspiration on S2 splitting
1st degree AV block on EKG
47. Late diastole 'atrial kick' sound against increased pressure from ventricular hypertrophy
S4
Fixed S2 splitting
Phase 0 of nodal AP
Interstitial fluid colloid osmotic pressure
48. Slow diastolic depolarization with Na conductance that accounts for automaticity of SA/AV nodes (slope determines HR)
Tricuspid Regurgitation
1st degree AV block on EKG
Phase 4 of nodal AP
Fick principle
49. Pulmonic valve closure before aortic valve closure associated with aortic stenosis or LBBB (delayed left ventrical emptying)
Auscultation location of tricuspid area
Paradoxical S2 splitting
Capillary hydrostatic pressure (Pc)
Auscultation location of mitral area
50. Cardiac output (CO) x total peripheral resistance (TPR) - or 2/3 diastolic pressure + 1/3 systolic pressure
Mean arterial pressure (MAP) =
Phase 3 of nodal AP
Auscultation location of pulmonic area
Normal S2 splitting