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USMLE Cardiovascular Physiology
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Subjects
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health-sciences
,
usmle
Instructions:
Answer 50 questions in 15 minutes.
If you are not ready to take this test, you can
study here
.
Match each statement with the correct term.
Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.
This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. V Tach with shifting sinusoidal waveforms on EKG - can progess to V fib - Predisposed by QT prolongation
Phase 0 of nodal AP
S2
Torsades de Pointes
A fib on EKG
2. Repolarization due to inactivation of Ca channels & activation of K channels
Phase 3 of nodal AP
S4
Normal S2 splitting
Effect of inspiration on S2 splitting
3. 1) increased capillary pressure - CHF 2) decreased plasma proteins - nephrotic syndrome - liver failure 3) increased capillary permeability - toxins - burns - infections 4) increased interstital osmotic pressure - lymphatic obstruction
Phase 1 of ventricular AP
Ventricular septal defect
Phase 4 of nodal AP
Common causes of edema
4. Crescendo - decrescendo systolic ejection murmur following an ejection click. Associated with weak pulses & syncope.
Aortic regurgitation
S1
Aortic Stenosis
Phase 2 of nodal AP
5. Associated with pulmonic stenosis or RBBB (delayed RV emptying)
Mitral Regurgitation
Paradoxical S2 splitting
Auscultation location of pulmonic area
Wide S2 splitting
6. Progressive lengthening of PR interval until dropped QRS complex
Mobitz type I (Wenckebach) 2nd degree AV block
Auscultation location of tricuspid area
Supplies blood to the base of the left ventricle
Interstital hydrostatic pressure (Pi)
7. Mitral & tripcuspid valve closure
Interstital hydrostatic pressure (Pi)
Paradoxical S2 splitting
Capillary hydrostatic pressure (Pc)
S1
8. Holosystolic 'blowing murmur' loudest at left sternal border near 4th rib - & is enhanced by inspiration (increased RA return)
Auscultation location of tricuspid area
Tricuspid Regurgitation
S2
Wide S2 splitting
9. Resting potential due to high K permeability
Tricuspid Regurgitation
Mean arterial pressure (MAP) =
Mitral Regurgitation
Phase 4 of ventricular AP
10. Plateau due to Ca influx balancing K efflux Myocyte contraction
Supplies blood to the base of the left ventricle
S2
Phase 2 of ventricular AP
Auscultation location of tricuspid area
11. Slow diastolic depolarization with Na conductance that accounts for automaticity of SA/AV nodes (slope determines HR)
Aortic regurgitation
Fick principle
Phase 4 of nodal AP
Plasma colloid osmotic pressure
12. Indicates recent MI
T wave inversion on EKG
Paradoxical S2 splitting
S4
Wolff - Parkinson - White Syndrome
13. Cardiac output (CO) x total peripheral resistance (TPR) - or 2/3 diastolic pressure + 1/3 systolic pressure
Resistance is directly proportional to...
Fick principle
Mean arterial pressure (MAP) =
Phase 2 of nodal AP
14. Late diastole 'atrial kick' sound against increased pressure from ventricular hypertrophy
Phase 3 of ventricular AP
Phase 3 of nodal AP
Phase 0 of nodal AP
S4
15. Radius to the 4th power
Resistance is inversely proportional to...
Supplies blood to the base of the left ventricle
S1
Phase 0 of ventricular AP
16. Rapid back - to - back atrial depolarization -> 'sawtooth' appearance
3rd degree complete AV block
A flutter on EKG
Plasma colloid osmotic pressure
Mitral Regurgitation
17. Inspiration delays pulmonic valve closure - which increases S2 splitting
Effect of inspiration on S2 splitting
Auscultation location of pulmonic area
S1
Phase 0 of ventricular AP
18. Ventricular pre - excitation caused by an accessory conduction pathway that bypasses the AV node. Delta wave on EKG. Can lead to SVT.
Phase 3 of ventricular AP
Mean arterial pressure (MAP) =
Wide S2 splitting
Wolff - Parkinson - White Syndrome
19. Rapid repolarization due to massive K efflux & closure of Ca channels
Resistance is inversely proportional to...
Phase 3 of ventricular AP
S2
Interstital hydrostatic pressure (Pi)
20. 2nd intercostal space along left sternal border
Mitral stenosis
A flutter on EKG
S2
Auscultation location of pulmonic area
21. Blood viscosity (increased in polycythemia - hyperproteinemia - & hereditary spherocytosis)
Phase 3 of nodal AP
Resistance is directly proportional to...
Effect of inspiration on S2 splitting
Wolff - Parkinson - White Syndrome
22. 4th rib at left sternal border
Aortic regurgitation
Mitral Regurgitation
1st degree AV block on EKG
Auscultation location of tricuspid area
23. Posterior descending artery (80% from RCA - 20% from CFX via LCA)
Auscultation location of aortic area
Normal S2 splitting
Mitral prolapse
Supplies blood to the base of the left ventricle
24. CO = rate of O2 consumption / (arterial O2 content - venous arterial O2 content)
Fick principle
S2
Net filtration pressure
Aortic regurgitation
25. Early diastole rapid filling sound heard with dilated ventricle (normal in children & pregnant women)
U wave on EKG
Phase 4 of nodal AP
1st degree AV block on EKG
S3
26. 2nd intercostal space along right sternal border
Auscultation location of mitral area
Mobitz type II 2nd degree AV block
T wave inversion on EKG
Auscultation location of aortic area
27. Caused by hypokalemia or bradycardia
Fick principle
Mitral Regurgitation
Phase 2 of nodal AP
U wave on EKG
28. Late systolic crescendo murmur (loudest at S2) following a midsystolic click - enhanced by squatting (increased TPR)
Phase 4 of nodal AP
Wolff - Parkinson - White Syndrome
Mitral prolapse
Phase 3 of ventricular AP
29. Associated with atrial septal defect (ASD) - allowing a left to right shunt that increases flow through pulmonic valve and delays closure
Aortic regurgitation
Auscultation location of aortic area
Fixed S2 splitting
U wave on EKG
30. Prolonged PR interval
Interstitial fluid colloid osmotic pressure
1st degree AV block on EKG
Phase 0 of nodal AP
Mean arterial pressure (MAP) =
31. Pull fluid out of capillary
Tricuspid Regurgitation
Interstitial fluid colloid osmotic pressure
Lyme disease can cause why type of AV block?
Mitral Regurgitation
32. Holosystolic 'blowing murmur' loudest at left 5th intercostal space midclavicular line - & is enhanced by expiration (increased LA return) & squatting (increased TPR)
Capillary hydrostatic pressure (Pc)
Phase 2 of ventricular AP
Lyme disease can cause why type of AV block?
Mitral Regurgitation
33. 'Irregularly irregular' - No discrete P waves and irregularly spaced QRS complexes
A fib on EKG
3rd degree complete AV block
Phase 2 of ventricular AP
Phase 1 of ventricular AP
34. Pushes fluid into capillary
S4
Interstital hydrostatic pressure (Pi)
A fib on EKG
1st degree AV block on EKG
35. Pulmonic valve closure before aortic valve closure associated with aortic stenosis or LBBB (delayed left ventrical emptying)
Paradoxical S2 splitting
Fixed S2 splitting
Mean arterial pressure (MAP) =
Fick principle
36. High pitched 'blowing' diastolic murmur - can decrease intensity of murmur with vasodilation
Paradoxical S2 splitting
Phase 3 of ventricular AP
Aortic regurgitation
Wolff - Parkinson - White Syndrome
37. Pulls fluid into capillary
Aortic regurgitation
Wide S2 splitting
Plasma colloid osmotic pressure
Tricuspid Regurgitation
38. Holosystolic 'harsh' murmur that is loudest at left sternal border near 4th rib
Mitral stenosis
Resistance is directly proportional to...
Ventricular septal defect
Mitral prolapse
39. Continuous machine - like murmur that is loudest at S2
S4
Phase 3 of nodal AP
Phase 1 of ventricular AP
Patent ductus arteriosus
40. Delayed 'rumbling' late diastolic murmur following an opening snap
Mitral stenosis
Aortic regurgitation
Phase 2 of nodal AP
S4
41. Aortic & pulmonic valve closure
Tricuspid Regurgitation
Auscultation location of tricuspid area
Mitral stenosis
S2
42. Aortic valve closure slightly before pulmonic valve closure at end of systolic ejection
Normal S2 splitting
T wave inversion on EKG
Capillary hydrostatic pressure (Pc)
Common causes of edema
43. Rapid upstroke due to voltage - gated Na channel opening
Phase 3 of nodal AP
Phase 2 of ventricular AP
Resistance is directly proportional to...
Phase 0 of ventricular AP
44. Dropped QRS complexes not preceded by change in PR interval (can be 2:1 - 3:1 - etc)
Paradoxical S2 splitting
Mobitz type II 2nd degree AV block
Lyme disease can cause why type of AV block?
Phase 0 of ventricular AP
45. Pushes fluid out of capillary
Capillary hydrostatic pressure (Pc)
Normal S2 splitting
Phase 2 of nodal AP
Phase 0 of ventricular AP
46. Atria & ventricles beat independent of each other - no relation between P waves & QRS complexes
Effect of inspiration on S2 splitting
Plasma colloid osmotic pressure
Ventricular septal defect
3rd degree complete AV block
47. 3rd degree
S4
Lyme disease can cause why type of AV block?
Effect of inspiration on S2 splitting
Mobitz type II 2nd degree AV block
48. Upstroke due to opening of Ca channels (Nodal tissues lack fast Na channels which allows for a prolonged AV node transmission from atria to ventricles)
Phase 4 of ventricular AP
Phase 0 of nodal AP
Torsades de Pointes
Mobitz type II 2nd degree AV block
49. 5th intercostal space at left midclavicular line
Wide S2 splitting
S4
Auscultation location of mitral area
Paradoxical S2 splitting
50. Absent (no plateau in nodal tissue)
Auscultation location of mitral area
Phase 2 of nodal AP
Phase 3 of nodal AP
A flutter on EKG
Sorry, Topic not found.:)
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