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Test your basic knowledge |
USMLE Cardiovascular Physiology
Start Test
Study First
Subjects
:
health-sciences
,
usmle
Instructions:
Answer 50 questions in 15 minutes.
If you are not ready to take this test, you can
study here
.
Match each statement with the correct term.
Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.
This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. Initial repolarization due to inactivation of Na channels & opening of voltaged - gated K channels
Wide S2 splitting
Auscultation location of tricuspid area
Mitral prolapse
Phase 1 of ventricular AP
2. Pushes fluid out of capillary
Resistance is directly proportional to...
1st degree AV block on EKG
Capillary hydrostatic pressure (Pc)
Auscultation location of pulmonic area
3. Dropped QRS complexes not preceded by change in PR interval (can be 2:1 - 3:1 - etc)
Plasma colloid osmotic pressure
Mobitz type II 2nd degree AV block
Tricuspid Regurgitation
Fixed S2 splitting
4. 2nd intercostal space along left sternal border
Supplies blood to the base of the left ventricle
Wolff - Parkinson - White Syndrome
Auscultation location of pulmonic area
Fixed S2 splitting
5. CO = rate of O2 consumption / (arterial O2 content - venous arterial O2 content)
Phase 1 of ventricular AP
Fick principle
Tricuspid Regurgitation
Paradoxical S2 splitting
6. Blood viscosity (increased in polycythemia - hyperproteinemia - & hereditary spherocytosis)
Resistance is inversely proportional to...
Resistance is directly proportional to...
Phase 0 of nodal AP
Patent ductus arteriosus
7. Repolarization due to inactivation of Ca channels & activation of K channels
Phase 4 of ventricular AP
Phase 3 of nodal AP
Plasma colloid osmotic pressure
Wolff - Parkinson - White Syndrome
8. Inspiration delays pulmonic valve closure - which increases S2 splitting
Effect of inspiration on S2 splitting
Mobitz type I (Wenckebach) 2nd degree AV block
Resistance is inversely proportional to...
1st degree AV block on EKG
9. Caused by hypokalemia or bradycardia
S4
U wave on EKG
Capillary hydrostatic pressure (Pc)
Phase 0 of ventricular AP
10. Plateau due to Ca influx balancing K efflux Myocyte contraction
U wave on EKG
S3
Resistance is directly proportional to...
Phase 2 of ventricular AP
11. 2nd intercostal space along right sternal border
Auscultation location of aortic area
Auscultation location of pulmonic area
Phase 2 of nodal AP
Mitral Regurgitation
12. Atria & ventricles beat independent of each other - no relation between P waves & QRS complexes
Phase 1 of ventricular AP
3rd degree complete AV block
Patent ductus arteriosus
Wide S2 splitting
13. Pushes fluid into capillary
Phase 3 of nodal AP
Ventricular septal defect
A flutter on EKG
Interstital hydrostatic pressure (Pi)
14. Prolonged PR interval
Capillary hydrostatic pressure (Pc)
Phase 4 of ventricular AP
Torsades de Pointes
1st degree AV block on EKG
15. Cardiac output (CO) x total peripheral resistance (TPR) - or 2/3 diastolic pressure + 1/3 systolic pressure
Mean arterial pressure (MAP) =
Mitral Regurgitation
Auscultation location of pulmonic area
Phase 4 of nodal AP
16. = (capillary pressure - interstital fluid pressure) - (plasma oncotic pressure - interstital fluid oncotic pressure)
Net filtration pressure
Effect of inspiration on S2 splitting
Phase 2 of ventricular AP
Torsades de Pointes
17. Continuous machine - like murmur that is loudest at S2
Phase 0 of ventricular AP
Patent ductus arteriosus
Tricuspid Regurgitation
A fib on EKG
18. 1) increased capillary pressure - CHF 2) decreased plasma proteins - nephrotic syndrome - liver failure 3) increased capillary permeability - toxins - burns - infections 4) increased interstital osmotic pressure - lymphatic obstruction
A fib on EKG
Common causes of edema
Lyme disease can cause why type of AV block?
Ventricular septal defect
19. Pull fluid out of capillary
Mitral stenosis
Interstitial fluid colloid osmotic pressure
Plasma colloid osmotic pressure
Phase 0 of ventricular AP
20. Pulmonic valve closure before aortic valve closure associated with aortic stenosis or LBBB (delayed left ventrical emptying)
Effect of inspiration on S2 splitting
Paradoxical S2 splitting
A fib on EKG
Mobitz type II 2nd degree AV block
21. Slow diastolic depolarization with Na conductance that accounts for automaticity of SA/AV nodes (slope determines HR)
Phase 4 of nodal AP
Aortic regurgitation
Capillary hydrostatic pressure (Pc)
Phase 3 of nodal AP
22. Absent (no plateau in nodal tissue)
Mitral prolapse
Phase 2 of nodal AP
Torsades de Pointes
S2
23. V Tach with shifting sinusoidal waveforms on EKG - can progess to V fib - Predisposed by QT prolongation
Resistance is inversely proportional to...
Phase 1 of ventricular AP
Auscultation location of pulmonic area
Torsades de Pointes
24. Associated with pulmonic stenosis or RBBB (delayed RV emptying)
Wide S2 splitting
Capillary hydrostatic pressure (Pc)
Auscultation location of aortic area
Supplies blood to the base of the left ventricle
25. Indicates recent MI
Fick principle
Paradoxical S2 splitting
T wave inversion on EKG
Phase 3 of nodal AP
26. High pitched 'blowing' diastolic murmur - can decrease intensity of murmur with vasodilation
Auscultation location of aortic area
T wave inversion on EKG
Phase 4 of ventricular AP
Aortic regurgitation
27. Late diastole 'atrial kick' sound against increased pressure from ventricular hypertrophy
3rd degree complete AV block
S4
Net filtration pressure
Mitral Regurgitation
28. Holosystolic 'blowing murmur' loudest at left 5th intercostal space midclavicular line - & is enhanced by expiration (increased LA return) & squatting (increased TPR)
Mitral Regurgitation
U wave on EKG
Phase 2 of nodal AP
Aortic Stenosis
29. 5th intercostal space at left midclavicular line
Mobitz type I (Wenckebach) 2nd degree AV block
Auscultation location of mitral area
Wolff - Parkinson - White Syndrome
Fixed S2 splitting
30. Ventricular pre - excitation caused by an accessory conduction pathway that bypasses the AV node. Delta wave on EKG. Can lead to SVT.
T wave inversion on EKG
Ventricular septal defect
Lyme disease can cause why type of AV block?
Wolff - Parkinson - White Syndrome
31. Mitral & tripcuspid valve closure
Net filtration pressure
Phase 4 of nodal AP
S1
Phase 4 of ventricular AP
32. Upstroke due to opening of Ca channels (Nodal tissues lack fast Na channels which allows for a prolonged AV node transmission from atria to ventricles)
Auscultation location of mitral area
Mitral stenosis
Phase 2 of nodal AP
Phase 0 of nodal AP
33. Early diastole rapid filling sound heard with dilated ventricle (normal in children & pregnant women)
Mobitz type I (Wenckebach) 2nd degree AV block
Ventricular septal defect
S3
Plasma colloid osmotic pressure
34. Rapid repolarization due to massive K efflux & closure of Ca channels
Aortic Stenosis
Phase 0 of nodal AP
Phase 3 of ventricular AP
Mobitz type II 2nd degree AV block
35. Rapid upstroke due to voltage - gated Na channel opening
Phase 0 of ventricular AP
Resistance is directly proportional to...
U wave on EKG
Plasma colloid osmotic pressure
36. Radius to the 4th power
Tricuspid Regurgitation
1st degree AV block on EKG
Resistance is inversely proportional to...
Phase 3 of ventricular AP
37. Associated with atrial septal defect (ASD) - allowing a left to right shunt that increases flow through pulmonic valve and delays closure
Fixed S2 splitting
Plasma colloid osmotic pressure
Aortic regurgitation
U wave on EKG
38. Delayed 'rumbling' late diastolic murmur following an opening snap
S2
Mitral stenosis
Ventricular septal defect
Torsades de Pointes
39. 3rd degree
Phase 4 of ventricular AP
Lyme disease can cause why type of AV block?
Aortic regurgitation
S3
40. Aortic valve closure slightly before pulmonic valve closure at end of systolic ejection
S1
Normal S2 splitting
Interstital hydrostatic pressure (Pi)
Net filtration pressure
41. Progressive lengthening of PR interval until dropped QRS complex
Lyme disease can cause why type of AV block?
Phase 2 of ventricular AP
Mobitz type I (Wenckebach) 2nd degree AV block
Ventricular septal defect
42. Pulls fluid into capillary
Auscultation location of aortic area
U wave on EKG
Phase 2 of ventricular AP
Plasma colloid osmotic pressure
43. Posterior descending artery (80% from RCA - 20% from CFX via LCA)
Torsades de Pointes
S3
Supplies blood to the base of the left ventricle
Paradoxical S2 splitting
44. Rapid back - to - back atrial depolarization -> 'sawtooth' appearance
Wolff - Parkinson - White Syndrome
Supplies blood to the base of the left ventricle
A flutter on EKG
Plasma colloid osmotic pressure
45. Late systolic crescendo murmur (loudest at S2) following a midsystolic click - enhanced by squatting (increased TPR)
Wolff - Parkinson - White Syndrome
Auscultation location of aortic area
Mitral prolapse
Fick principle
46. Aortic & pulmonic valve closure
3rd degree complete AV block
Mitral stenosis
Phase 0 of ventricular AP
S2
47. Resting potential due to high K permeability
Phase 4 of ventricular AP
U wave on EKG
Torsades de Pointes
T wave inversion on EKG
48. 'Irregularly irregular' - No discrete P waves and irregularly spaced QRS complexes
Auscultation location of aortic area
Wolff - Parkinson - White Syndrome
A fib on EKG
Phase 3 of nodal AP
49. Holosystolic 'harsh' murmur that is loudest at left sternal border near 4th rib
Normal S2 splitting
Ventricular septal defect
Aortic regurgitation
Auscultation location of tricuspid area
50. Holosystolic 'blowing murmur' loudest at left sternal border near 4th rib - & is enhanced by inspiration (increased RA return)
Auscultation location of mitral area
Mitral prolapse
Tricuspid Regurgitation
Phase 0 of ventricular AP