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Test your basic knowledge |
USMLE Cardiovascular Physiology
Start Test
Study First
Subjects
:
health-sciences
,
usmle
Instructions:
Answer 50 questions in 15 minutes.
If you are not ready to take this test, you can
study here
.
Match each statement with the correct term.
Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.
This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. Rapid repolarization due to massive K efflux & closure of Ca channels
Normal S2 splitting
Phase 3 of ventricular AP
Mitral Regurgitation
T wave inversion on EKG
2. Early diastole rapid filling sound heard with dilated ventricle (normal in children & pregnant women)
S3
Torsades de Pointes
Paradoxical S2 splitting
T wave inversion on EKG
3. Caused by hypokalemia or bradycardia
Ventricular septal defect
Phase 0 of nodal AP
U wave on EKG
Lyme disease can cause why type of AV block?
4. Crescendo - decrescendo systolic ejection murmur following an ejection click. Associated with weak pulses & syncope.
Aortic Stenosis
Phase 0 of ventricular AP
Ventricular septal defect
Phase 3 of ventricular AP
5. Associated with pulmonic stenosis or RBBB (delayed RV emptying)
1st degree AV block on EKG
Wolff - Parkinson - White Syndrome
Wide S2 splitting
Resistance is directly proportional to...
6. Initial repolarization due to inactivation of Na channels & opening of voltaged - gated K channels
Auscultation location of tricuspid area
S2
Capillary hydrostatic pressure (Pc)
Phase 1 of ventricular AP
7. Aortic valve closure slightly before pulmonic valve closure at end of systolic ejection
Normal S2 splitting
Phase 0 of nodal AP
Mobitz type I (Wenckebach) 2nd degree AV block
Mean arterial pressure (MAP) =
8. Rapid back - to - back atrial depolarization -> 'sawtooth' appearance
Phase 0 of nodal AP
A flutter on EKG
Phase 2 of ventricular AP
Paradoxical S2 splitting
9. Holosystolic 'blowing murmur' loudest at left sternal border near 4th rib - & is enhanced by inspiration (increased RA return)
Torsades de Pointes
Wide S2 splitting
Wolff - Parkinson - White Syndrome
Tricuspid Regurgitation
10. Indicates recent MI
T wave inversion on EKG
Effect of inspiration on S2 splitting
Aortic regurgitation
Resistance is directly proportional to...
11. Aortic & pulmonic valve closure
Fick principle
Auscultation location of pulmonic area
A fib on EKG
S2
12. Radius to the 4th power
Resistance is inversely proportional to...
Interstital hydrostatic pressure (Pi)
1st degree AV block on EKG
Auscultation location of pulmonic area
13. Prolonged PR interval
Net filtration pressure
Phase 3 of nodal AP
1st degree AV block on EKG
Tricuspid Regurgitation
14. Repolarization due to inactivation of Ca channels & activation of K channels
U wave on EKG
Phase 2 of nodal AP
Phase 3 of nodal AP
Paradoxical S2 splitting
15. Associated with atrial septal defect (ASD) - allowing a left to right shunt that increases flow through pulmonic valve and delays closure
Mean arterial pressure (MAP) =
3rd degree complete AV block
Fixed S2 splitting
Wide S2 splitting
16. Pushes fluid into capillary
Wolff - Parkinson - White Syndrome
T wave inversion on EKG
Capillary hydrostatic pressure (Pc)
Interstital hydrostatic pressure (Pi)
17. Blood viscosity (increased in polycythemia - hyperproteinemia - & hereditary spherocytosis)
Resistance is directly proportional to...
Fick principle
Net filtration pressure
T wave inversion on EKG
18. 'Irregularly irregular' - No discrete P waves and irregularly spaced QRS complexes
Patent ductus arteriosus
S1
Aortic regurgitation
A fib on EKG
19. Upstroke due to opening of Ca channels (Nodal tissues lack fast Na channels which allows for a prolonged AV node transmission from atria to ventricles)
Phase 0 of nodal AP
Capillary hydrostatic pressure (Pc)
S1
S3
20. Resting potential due to high K permeability
Phase 4 of ventricular AP
Torsades de Pointes
Phase 3 of nodal AP
Phase 0 of ventricular AP
21. Pulmonic valve closure before aortic valve closure associated with aortic stenosis or LBBB (delayed left ventrical emptying)
Phase 4 of nodal AP
Phase 2 of ventricular AP
Mobitz type I (Wenckebach) 2nd degree AV block
Paradoxical S2 splitting
22. Holosystolic 'blowing murmur' loudest at left 5th intercostal space midclavicular line - & is enhanced by expiration (increased LA return) & squatting (increased TPR)
Mitral Regurgitation
Wide S2 splitting
Phase 0 of ventricular AP
S2
23. High pitched 'blowing' diastolic murmur - can decrease intensity of murmur with vasodilation
Aortic regurgitation
3rd degree complete AV block
Interstitial fluid colloid osmotic pressure
Phase 1 of ventricular AP
24. Atria & ventricles beat independent of each other - no relation between P waves & QRS complexes
S2
Auscultation location of tricuspid area
3rd degree complete AV block
Phase 4 of nodal AP
25. 5th intercostal space at left midclavicular line
Mitral Regurgitation
Effect of inspiration on S2 splitting
Auscultation location of mitral area
Torsades de Pointes
26. Slow diastolic depolarization with Na conductance that accounts for automaticity of SA/AV nodes (slope determines HR)
Wolff - Parkinson - White Syndrome
Phase 3 of nodal AP
Phase 4 of nodal AP
Auscultation location of pulmonic area
27. Mitral & tripcuspid valve closure
Fick principle
S1
S2
S3
28. Late diastole 'atrial kick' sound against increased pressure from ventricular hypertrophy
Interstitial fluid colloid osmotic pressure
S4
S2
Wide S2 splitting
29. CO = rate of O2 consumption / (arterial O2 content - venous arterial O2 content)
Fick principle
Plasma colloid osmotic pressure
Torsades de Pointes
Tricuspid Regurgitation
30. 3rd degree
Auscultation location of aortic area
Lyme disease can cause why type of AV block?
T wave inversion on EKG
Mobitz type I (Wenckebach) 2nd degree AV block
31. Progressive lengthening of PR interval until dropped QRS complex
Aortic regurgitation
Phase 2 of ventricular AP
Resistance is inversely proportional to...
Mobitz type I (Wenckebach) 2nd degree AV block
32. Pushes fluid out of capillary
Wide S2 splitting
Patent ductus arteriosus
Interstital hydrostatic pressure (Pi)
Capillary hydrostatic pressure (Pc)
33. Delayed 'rumbling' late diastolic murmur following an opening snap
Mitral stenosis
Wolff - Parkinson - White Syndrome
S2
Mitral Regurgitation
34. Inspiration delays pulmonic valve closure - which increases S2 splitting
Effect of inspiration on S2 splitting
S4
U wave on EKG
Torsades de Pointes
35. Late systolic crescendo murmur (loudest at S2) following a midsystolic click - enhanced by squatting (increased TPR)
Ventricular septal defect
U wave on EKG
Patent ductus arteriosus
Mitral prolapse
36. Dropped QRS complexes not preceded by change in PR interval (can be 2:1 - 3:1 - etc)
Mitral prolapse
Mobitz type II 2nd degree AV block
Auscultation location of pulmonic area
Torsades de Pointes
37. Ventricular pre - excitation caused by an accessory conduction pathway that bypasses the AV node. Delta wave on EKG. Can lead to SVT.
Wolff - Parkinson - White Syndrome
Ventricular septal defect
S4
Capillary hydrostatic pressure (Pc)
38. Posterior descending artery (80% from RCA - 20% from CFX via LCA)
Mitral Regurgitation
U wave on EKG
Mobitz type I (Wenckebach) 2nd degree AV block
Supplies blood to the base of the left ventricle
39. Continuous machine - like murmur that is loudest at S2
Aortic regurgitation
Effect of inspiration on S2 splitting
Patent ductus arteriosus
Paradoxical S2 splitting
40. Pulls fluid into capillary
Phase 2 of nodal AP
Mobitz type II 2nd degree AV block
Tricuspid Regurgitation
Plasma colloid osmotic pressure
41. V Tach with shifting sinusoidal waveforms on EKG - can progess to V fib - Predisposed by QT prolongation
Torsades de Pointes
Resistance is inversely proportional to...
Normal S2 splitting
Auscultation location of pulmonic area
42. 4th rib at left sternal border
Phase 2 of ventricular AP
Auscultation location of aortic area
S1
Auscultation location of tricuspid area
43. 2nd intercostal space along right sternal border
A flutter on EKG
Torsades de Pointes
Auscultation location of aortic area
Mobitz type I (Wenckebach) 2nd degree AV block
44. Pull fluid out of capillary
Plasma colloid osmotic pressure
S2
Interstitial fluid colloid osmotic pressure
Effect of inspiration on S2 splitting
45. Cardiac output (CO) x total peripheral resistance (TPR) - or 2/3 diastolic pressure + 1/3 systolic pressure
Resistance is directly proportional to...
Mobitz type II 2nd degree AV block
T wave inversion on EKG
Mean arterial pressure (MAP) =
46. Plateau due to Ca influx balancing K efflux Myocyte contraction
U wave on EKG
Effect of inspiration on S2 splitting
Phase 0 of nodal AP
Phase 2 of ventricular AP
47. 2nd intercostal space along left sternal border
Phase 0 of nodal AP
Resistance is directly proportional to...
Auscultation location of pulmonic area
Common causes of edema
48. Rapid upstroke due to voltage - gated Na channel opening
Torsades de Pointes
Aortic Stenosis
Aortic regurgitation
Phase 0 of ventricular AP
49. = (capillary pressure - interstital fluid pressure) - (plasma oncotic pressure - interstital fluid oncotic pressure)
Wolff - Parkinson - White Syndrome
Auscultation location of aortic area
Net filtration pressure
Phase 1 of ventricular AP
50. 1) increased capillary pressure - CHF 2) decreased plasma proteins - nephrotic syndrome - liver failure 3) increased capillary permeability - toxins - burns - infections 4) increased interstital osmotic pressure - lymphatic obstruction
Normal S2 splitting
Ventricular septal defect
Phase 0 of nodal AP
Common causes of edema