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USMLE Step 1 Biochemistry

Instructions:
  • Answer 50 questions in 15 minutes.
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  • Match each statement with the correct term.
  • Don't refresh. All questions and answers are randomly picked and ordered every time you load a test.

This is a study tool. The 3 wrong answers for each question are randomly chosen from answers to other questions. So, you might find at times the answers obvious, but you will see it re-enforces your understanding as you take the test each time.
1. What is a robertsonian translocation? What does it mean if its balanced?






2. What are the mucopolysaccharidoses? Which one is XR? AR? What accumulates in them?






3. In the fasting state - What ensures that the pyruvate doesnt get used for TCA cycle?






4. What is the fate of glucose from glycogenolysis?






5. What are five clinical features seen in Osteogenesis Imperfecta?






6. What is the difference between wet and dry beriberi?






7. What are the clinical findings in edwards syndrome?






8. What type of aa are histones made of? are they acidic or basic?






9. What is the source of ATP When you are fasting (in between meals)?






10. What is the most abundant protein in the body? From What cell type is it made? How many types are there? Where is it found (generally)?






11. What causes scurvy? What are the symptoms of scurvy?






12. Vit A is ________ - causes cardiac problems and cleft palate in newborns






13. What type of disease is MERRF? What is the clinical presentation?






14. What explains the fatty change in kwashiokor?






15. What is the pathogenesis of skurvy? name some clinical symptoms.






16. What does ultrasound show in Downs?






17. How is entry into cell cycle phases regulated?






18. Daughters of Xlinked recessive diseased fathers are...






19. 'Initiation' of protein tranlsation is activated by ATP hydrolysis. T/F?






20. What causes cataracts in a galactose metabolism deficiency?






21. How do you treat niacin deficiency do to corn based diet?






22. What are the resp. clinical symptoms of CF?






23. Which amino acid is the most basic?






24. Where does synthesis of longer chain fatty acids occur? What does it require?






25. ____ soluble vitamins are more likely to result in toxicity






26. What is given to prevent side effects When methotrexate is given? why does this work?






27. _____________ prevents fluidity of plasma membrane






28. IMP is degraded to inosine and then to hypoxanthine. What two options are next? by What enzymes?






29. All glutamate goes to _______ d ammonia is used as a buffer in urine






30. What does fructose 2 -6 bisphosphate regulate?






31. Give an example of codominance.






32. What is the treatment of hyperammonia?






33. What can cause Biotin deficiency? What are the symptoms?






34. Where is type 1 collagen found?






35. What liver enzymes are increased in alcoholism? why?






36. What is a clinical example of a missense mutation?






37. What type of polymerase is primase?






38. why does branching occur in glycogenesis?






39. _______ can not get inside cells






40. What carries electrons (as energy)?






41. What is the pyruvate dehydrogenase complex used for? What is produced in that reaction?






42. How do WBCs of CGD patients do some damage?






43. What aa (other than glutamate) is a precursor for urea?






44. Where is vit C found?






45. What toxins prevent elongation in translation in humans (by binding 40s)?






46. What dose a deficiency in Vit B12 cause? What causes it?






47. How is the nitrogen of amino acids transferred to the liver for the urea cycle? explain this process.






48. is CPSII or CPSI involved in pyrimidine synthesis?






49. What does deficiency of Vit K cause?






50. AR disease is usually...







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